Diabetes-accelerated memory dysfunction via cerebrovascular inflammation and Abeta deposition in an Alzheimer mouse model with diabetes - Test2 - elhamkhani - TriplyDB

Diabetes-accelerated memory dysfunction via cerebrovascular inflammation and Abeta deposition in an Alzheimer mouse model with diabetes

Diabetes-accelerated memory dysfunction via cerebrovascular inflammation and Abeta deposition in an Alzheimer mouse model with diabetes

http://www.wikidata.org/entity/Q33859600

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Menopause, obesity and inflammation: interactive risk factors for Alzheimer's disease Diabetes Mellitus Induces Alzheimer's Disease Pathology: Histopathological Evidence from Animal Models The roles of lipid and glucose metabolism in modulation of β-amyloid, tau, and neurodegeneration in the pathogenesis of Alzheimer disease Shared Neuropathological Characteristics of Obesity, Type 2 Diabetes and Alzheimer's Disease: Impacts on Cognitive Decline Parkinson's disease, insulin resistance and novel agents of neuroprotection The role of mammalian sirtuins in the regulation of metabolism, aging, and longevity Alzheimer's disease and insulin resistance: translating basic science into clinical applications Food for thought: the role of appetitive peptides in age-related cognitive decline Roles of vascular and metabolic components in cognitive dysfunction of Alzheimer disease: short- and long-term modification by non-genetic risk factors Alzheimer's disease and type 2 diabetes via chronic inflammatory mechanisms Neuroinflammation in Alzheimer's disease Immunohistochemical study of semicarbazide-sensitive amine oxidase/vascular adhesion protein-1 in the hippocampal vasculature: pathological synergy of Alzheimer's disease and diabetes mellitus A pilot study to examine the correlation between cognition and blood biomarkers in a Singapore Chinese male cohort with type 2 diabetes mellitus Diabetes mellitus accelerates Aβ pathology in brain accompanied by enhanced GAβ generation in nonhuman primates Energy intake and exercise as determinants of brain health and vulnerability to injury and disease Protection against the synaptic targeting and toxicity of Alzheimer's-associated Aβ oligomers by insulin mimetic chiro-inositols.A ketone ester diet exhibits anxiolytic and cognition-sparing properties, and lessens amyloid and tau pathologies in a mouse model of Alzheimer's disease Endoplasmic reticulum stress-induced neuronal inflammatory response and apoptosis likely plays a key role in the development of diabetic encephalopathy.High-fat diet-induced memory impairment in triple-transgenic Alzheimer's disease (3xTgAD) mice is independent of changes in amyloid and tau pathology.Folic Acid Reduces Tau Phosphorylation by Regulating PP2A Methylation in Streptozotocin-Induced Diabetic Mice.Combined immunotherapy with "anti-insulin resistance" therapy as a novel therapeutic strategy against neurodegenerative diseases.Short-term environmental enrichment enhances adult neurogenesis, vascular network and dendritic complexity in the hippocampus of type 1 diabetic mice.Repurposing diabetes drugs for brain insulin resistance in Alzheimer disease.Transgenic mice overexpressing amyloid precursor protein exhibit early metabolic deficits and a pathologically low leptin state associated with hypothalamic dysfunction in arcuate neuropeptide Y neurons Melatonin Modulates Neuronal Cell Death Induced by Endoplasmic Reticulum Stress under Insulin Resistance Condition.Linking type 2 diabetes and Alzheimer's disease.Diabetes and cardiovascular disease in older adults: current status and future directions.Altered expression of diabetes-related genes in Alzheimer's disease brains: the Hisayama study.Linking cardiometabolic disorders to sporadic Alzheimer's disease: a perspective on potential mechanisms and mediators.The procognitive effects of leptin in the brain and their clinical implications Obesity and diabetes cause cognitive dysfunction in the absence of accelerated β-amyloid deposition in a novel murine model of mixed or vascular dementia.Disrupted energy metabolism and neuronal circuit dysfunction in cognitive impairment and Alzheimer's disease.Hyperleucinemia causes hippocampal retromer deficiency linking diabetes to Alzheimer's disease.Adiponectin is protective against oxidative stress induced cytotoxicity in amyloid-beta neurotoxicity Diabetes mellitus and Alzheimer's disease: shared pathology and treatment?Insulin resistance in Alzheimer's disease.Insulin-resistant brain state: the culprit in sporadic Alzheimer's disease?A common pathogenic mechanism linking type-2 diabetes and Alzheimer's disease: evidence from animal models.Bidirectional metabolic regulation of neurocognitive function.Molecular targets of the multifunctional iron-chelating drug, M30, in the brains of mouse models of type 2 diabetes mellitus

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Diabetes-accelerated memory dysfunction via cerebrovascular inflammation and Abeta deposition in an Alzheimer mouse model with diabetes

http://www.wikidata.org/entity/Q33859600

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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Diabetes-accelerated memory dy ...... imer mouse model with diabetes

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Proceedings of the National Academy of Sciences of the United States of America

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Diabetes-accelerated memory dy ...... imer mouse model with diabetes

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Daisuke Takeuchi

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Hitomi Kurinami

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Kozue Uchio-Yamada

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Kyoko Sawada

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Mitsuru Shinohara

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Naoyuki Sato

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Ryuichi Morishita

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Shuko Takeda

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Takanori Kunieda

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P2860

The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics

GSK-3alpha regulates production of Alzheimer's disease amyloid-beta peptides

Obesity wars: molecular progress confronts an expanding epidemic

Neurovascular regulation in the normal brain and in Alzheimer's disease

Diabetes and atherosclerosis: epidemiology, pathophysiology, and management.

Insulin receptor signaling regulates synapse number, dendritic plasticity, and circuit function in vivo.

Does insulin dysfunction play a role in Alzheimer's disease?

Insulin resistance syndrome and Alzheimer's disease: age- and obesity-related effects on memory, amyloid, and inflammation.

Insulin, insulin-degrading enzyme and amyloid-beta peptide in Alzheimer's disease: review and hypothesis.

Inflammation in Alzheimer disease: driving force, bystander or beneficial response?

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7036-7041

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20231468

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Alzheimer's disease

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2872449

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