Bile acids induce cholera toxin expression in Vibrio cholerae in a ToxT-independent manner.
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Biochemical basis for activation of virulence genes by bile salts in Vibrio parahaemolyticus.Bile salt receptor complex activates a pathogenic type III secretion systemPhenotypic characterization of pore mutants of the Vibrio cholerae porin OmpUCholera toxin production during anaerobic trimethylamine N-oxide respiration is mediated by stringent response in Vibrio cholerae.The Vibrio cholerae VprA-VprB two-component system controls virulence through endotoxin modification.Bile Salts Modulate the Mucin-Activated Type VI Secretion System of Pandemic Vibrio cholerae.ToxR Antagonizes H-NS Regulation of Horizontally Acquired Genes to Drive Host ColonizationTwo regulators of Vibrio parahaemolyticus play important roles in enterotoxicity by controlling the expression of genes in the Vp-PAI region.Bile Sensing: The Activation of Vibrio parahaemolyticus VirulenceBile acid-induced virulence gene expression of Vibrio parahaemolyticus reveals a novel therapeutic potential for bile acid sequestrants.Efficient responses to host and bacterial signals during Vibrio cholerae colonizationMolecular keys of the tropism of integration of the cholera toxin phage.vttRA and vttRB Encode ToxR family proteins that mediate bile-induced expression of type three secretion system genes in a non-O1/non-O139 Vibrio cholerae strain.Bile acids and bicarbonate inversely regulate intracellular cyclic di-GMP in Vibrio cholerae.VirA and VirG activate the Ti plasmid repABC operon, elevating plasmid copy number in response to wound-released chemical signals.Expression of colonization factor CS5 of enterotoxigenic Escherichia coli (ETEC) is enhanced in vivo and by the bile component Na glycocholate hydrate.Effects of polyamines on Vibrio cholerae virulence properties.Effects of amino acid supplementation on porin expression and ToxR levels in Vibrio choleraeVibrio cholerae CsrA Regulates ToxR Levels in Response to Amino Acids and Is Essential for Virulence.Intestinal GPS: bile and bicarbonate control cyclic di-GMP to provide Vibrio cholerae spatial cues within the small intestine.Vibrio cholerae leuO Transcription Is Positively Regulated by ToxR and Contributes to Bile ResistanceRegulatory networks controlling Vibrio cholerae virulence gene expressionDisulfide bond formation and ToxR activity in Vibrio cholerae.Bile salt-induced intermolecular disulfide bond formation activates Vibrio cholerae virulence.Regulation of virulence in Vibrio cholerae: the ToxR regulon.Small-molecule inhibitors of toxT expression in Vibrio cholerae.Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.Bile salts induce resistance to polymyxin in enterohemorrhagic Escherichia coli O157:H7.Repair of DNA damage induced by bile salts in Salmonella enterica.Characterization of V. cholerae T3SS-dependent cytotoxicity in cultured intestinal epithelial cells.Bile salts and alkaline pH reciprocally modulate the interaction between the periplasmic domains of Vibrio cholerae ToxR and ToxS.Survival of the Fittest: How Bacterial Pathogens Utilize Bile To Enhance Infection.Redox pathway sensing bile salts activates virulence gene expression in Vibrio cholerae.Mechanism for inhibition of Vibrio cholerae ToxT activity by the unsaturated fatty acid components of bile.Vibrio cholerae RND family efflux systems are required for antimicrobial resistance, optimal virulence factor production, and colonization of the infant mouse small intestine.Identification of a bile-induced exopolysaccharide required for Salmonella biofilm formation on gallstone surfaces.Identification and characterization of the functional toxboxes in the Vibrio cholerae cholera toxin promoter.Low concentrations of bile salts induce stress responses and reduce motility in Bacillus cereus ATCC 14579 [corrected]Regulatory actions of ToxR and CalR on their own genes and type III secretion system 1 in Vibrio parahaemolyticusRole of the histone-like nucleoid structuring protein in colonization, motility, and bile-dependent repression of virulence gene expression in Vibrio cholerae.
P2860
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P2860
Bile acids induce cholera toxin expression in Vibrio cholerae in a ToxT-independent manner.
description
2005 nî lūn-bûn
@nan
2005 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@ast
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@en
type
label
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@ast
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@en
prefLabel
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@ast
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@en
P2860
P356
P1476
Bile acids induce cholera toxi ...... in a ToxT-independent manner.
@en
P2093
Deborah T Hung
John J Mekalanos
P2860
P304
P356
10.1073/PNAS.0409559102
P407
P577
2005-02-07T00:00:00Z