Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage - Test2 - elhamkhani - TriplyDB

Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

http://www.wikidata.org/entity/Q33864438

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Epigenetic control of HIV-1 post integration latency: implications for therapy The macrophage: a therapeutic target in HIV-1 infection Recent developments in human immunodeficiency virus-1 latency research Bioinformatic analysis of HIV-1 entry and pathogenesis.Targeting the Brain Reservoirs: Toward an HIV Cure HIV-1 infection and first line ART induced differential responses in mitochondria from blood lymphocytes and monocytes: the ANRS EP45 "Aging" study HIV-1 infection of T cells and macrophages are differentially modulated by virion-associated Hck: a Nef-dependent phenomenon Mechanisms Underlying HIV Associated Non-infectious Lung Disease.Macrophage internal HIV-1 is protected from neutralizing antibodies.Proteomic analysis of HIV-infected macrophages Macrophages and their relevance in Human Immunodeficiency Virus Type I infection.A role for microRNA-155 modulation in the anti-HIV-1 effects of Toll-like receptor 3 stimulation in macrophages.Analyses of nanoformulated antiretroviral drug charge, size, shape and content for uptake, drug release and antiviral activities in human monocyte-derived macrophages.HIV DNA reservoir increases risk for cognitive disorders in cART-naïve patients.Candida albicans delays HIV-1 replication in macrophages.Caveolin 1 inhibits HIV replication by transcriptional repression mediated through NF-κB.Pulsed stable isotope labeling of amino acids in cell culture uncovers the dynamic interactions between HIV-1 and the monocyte-derived macrophage.Monocytes mediate HIV neuropathogenesis: mechanisms that contribute to HIV associated neurocognitive disorders Nuclear receptor signaling inhibits HIV-1 replication in macrophages through multiple trans-repression mechanisms Proteomic biosignatures for monocyte-macrophage differentiation.Eradication of HIV-1 from the macrophage reservoir: an uncertain goal?The role of monocyte-lineage cells in human immunodeficiency virus persistence: mechanisms and progress.Role of the macrophage in HIV-associated neurocognitive disorders and other comorbidities in patients on effective antiretroviral treatment.LSD1 cooperates with CTIP2 to promote HIV-1 transcriptional silencing.Morphine and galectin-1 modulate HIV-1 infection of human monocyte-derived macrophages.Uracil DNA glycosylase interacts with the p32 subunit of the replication protein A complex to modulate HIV-1 reverse transcription for optimal virus dissemination.Alterations in P-Glycoprotein Expression and Function Between Macrophage Subsets.Preparation, characterization, and safety evaluation of poly(lactide-co-glycolide) nanoparticles for protein delivery into macrophages.Nanoparticle based galectin-1 gene silencing, implications in methamphetamine regulation of HIV-1 infection in monocyte derived macrophages Virus-producing cells determine the host protein profiles of HIV-1 virion cores.HIV-1 induces telomerase activity in monocyte-derived macrophages, possibly safeguarding one of its reservoirs.Inhibition of HIV Expression and Integration in Macrophages by Methylglyoxal-Bis-Guanylhydrazone.Human immunodeficiency virus (HIV) latency: the major hurdle in HIV eradication Accurate predictions of population-level changes in sequence and structural properties of HIV-1 Env using a volatility-controlled diffusion model.Pathways of cell-cell transmission of HTLV-1 Caveolin-1 reduces HIV-1 infectivity by restoration of HIV Nef mediated impairment of cholesterol efflux by apoA-I.Impact of cellular autophagy on viruses: Insights from hepatitis B virus and human retroviruses.Presence of high-risk human papillomavirus genotype and human immunodeficiency virus DNA in anal high-grade and low-grade squamous intraepithelial lesions.In vivo platforms for analysis of HIV persistence and eradication.HIV-1 Nef in macrophage-mediated disease pathogenesis.

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

http://www.wikidata.org/entity/Q33864438

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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type

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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Molecular mechanisms of HIV-1 persistence in the monocyte-macrophage lineage

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Christian Schwartz

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Georges Herbein

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Olivier Rohr

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Valentin Le Douce

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Creative Commons Attribution 2.0 Generic

P2860

HIV-1 TAR miRNA protects against apoptosis by altering cellular gene expression

Isolation of a T-lymphotropic retrovirus from a patient at risk for acquired immune deficiency syndrome (AIDS)

HIV-1 Nef inhibits ASK1-dependent death signalling providing a potential mechanism for protecting the infected host cell

Human immunodeficiency virus type 1 Nef binds to tumor suppressor p53 and protects cells against p53-mediated apoptosis.

Recruitment of chromatin-modifying enzymes by CTIP2 promotes HIV-1 transcriptional silencing

Residual human immunodeficiency virus type 1 viremia in some patients on antiretroviral therapy is dominated by a small number of invariant clones rarely found in circulating CD4+ T cells

Repression of the HIV-1 5' LTR promoter and inhibition of HIV-1 replication by using engineered zinc-finger transcription factors

Active nuclear import of human immunodeficiency virus type 1 preintegration complexes

HIV-1 entry inhibitors: an overview

YY1 and FoxD3 regulate antiretroviral zinc finger protein OTK18 promoter activation induced by HIV-1 infection

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10.1186/1742-4690-7-32

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7

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Georges Herbein

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bone marrow cells

hemic and immune systems

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