Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.
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Human Primary Epithelial Cells Acquire an Epithelial-Mesenchymal-Transition Phenotype during Long-Term Infection by the Oral Opportunistic Pathogen, Porphyromonas gingivalis.A Novel Kinase Function of a Nucleoside-diphosphate-kinase Homolog in P. gingivalis is Critical in Subversion of Host Cell Apoptosis by Targeting Heat-Shock Protein 27.Unfolding Role of a Danger Molecule Adenosine Signaling in Modulation of Microbial Infection and Host Cell Response.Porphyromonas gingivalis Traffics into Endoplasmic Reticulum-Rich-Autophagosomes for Successful Survival in Human Gingival Epithelial Cells.Stimulation of TLR3 triggers release of lysosomal ATP in astrocytes and epithelial cells that requires TRPML1 channels.
P2860
Opportunistic Pathogen Porphyromonas gingivalis Modulates Danger Signal ATP-Mediated Antibacterial NOX2 Pathways in Primary Epithelial Cells.
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name
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
@ast
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
@en
type
label
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
@ast
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
@en
prefLabel
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
@ast
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
@en
P2093
P2860
P356
P1476
Opportunistic Pathogen Porphyr ...... s in Primary Epithelial Cells.
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P2093
Chul Hee Choi
Gill Diamond
Jeff Deguzman
JoAnn S Roberts
Jungnam Lee
P2860
P356
10.3389/FCIMB.2017.00291
P577
2017-07-04T00:00:00Z