p21(WAF1/CIP1) upregulation through the stress granule-associated protein CUGBP1 confers resistance to bortezomib-mediated apoptosis. - Test2 - elhamkhani - TriplyDB

p21(WAF1/CIP1) upregulation through the stress granule-associated protein CUGBP1 confers resistance to bortezomib-mediated apoptosis.

p21(WAF1/CIP1) upregulation through the stress granule-associated protein CUGBP1 confers resistance to bortezomib-mediated apoptosis.

http://www.wikidata.org/entity/Q33921377

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Feedback Regulation of Kinase Signaling Pathways by AREs and GREs Role of HRI in apoptosis resistance Characterization of fragile X mental retardation protein recruitment and dynamics in Drosophila stress granules Diverse Strategies Used by Picornaviruses to Escape Host RNA Decay Pathways The RNA-binding protein CUG-BP1 increases survivin expression in oesophageal cancer cells through enhanced mRNA stability.Development of peptide-based reversing agents for p-glycoprotein-mediated resistance to carfilzomib Tudor-SN, a component of stress granules, regulates growth under salt stress by modulating GA20ox3 mRNA levels in Arabidopsis.HDAC inhibitor L-carnitine and proteasome inhibitor bortezomib synergistically exert anti-tumor activity in vitro and in vivo Modulation of TNF-α mRNA stability by human antigen R and miR181s in sepsis-induced immunoparalysis.Stress granules, P-bodies and cancer.Arginylated calreticulin at plasma membrane increases susceptibility of cells to apoptosis.Altered CELF1 binding to target transcripts in malignant T cells.Lysine acetyltransferase NuA4 and acetyl-CoA regulate glucose-deprived stress granule formation in Saccharomyces cerevisiae.Can we observe changes in mRNA "state"? Overview of methods to study mRNA interactions with regulatory proteins relevant in cancer related processes.Overexpression of miR-214-3p in esophageal squamous cancer cells enhances sensitivity to cisplatin by targeting survivin directly and indirectly through CUG-BP1.Overexpression of RNA-binding protein CELF1 prevents apoptosis and destabilizes pro-apoptotic mRNAs in oral cancer cells.RNA-binding protein CELF1 promotes tumor growth and alters gene expression in oral squamous cell carcinoma.CELFish ways to modulate mRNA decay.Lithium down-regulates histone deacetylase 1 (HDAC1) and induces degradation of mutant huntingtin.Exocytosis of polyubiquitinated proteins in bortezomib-resistant leukemia cells: a role for MARCKS in acquired resistance to proteasome inhibitors.The role of the 3' untranslated region in post-transcriptional regulation of protein expression in mammalian cells.Bortezomib for the treatment of previously untreated multiple myeloma.Nucleoli and stress granules: connecting distant relatives.mRNPs meet stress granules.Sorafenib, a multikinase inhibitor, induces formation of stress granules in hepatocarcinoma cells.Role of cyclooxygenase-2 in Trypanosoma cruzi survival in the early stages of parasite host-cell interaction CUGBP1 promotes cell proliferation and suppresses apoptosis via down-regulating C/EBPα in human non-small cell lung cancers.Overexpression of CUGBP1 is associated with the progression of non-small cell lung cancer.Cellular uptake kinetics of bortezomib in relation to efficacy in myeloma cells and the influence of drug transporters.Knockdown of CUG-binding protein 1 induces apoptosis of human laryngeal cancer cells.5-Fluorouracil affects assembly of stress granules based on RNA incorporation.Wig-1 regulates cell cycle arrest and cell death through the p53 targets FAS and 14-3-3σ.The proteasome inhibitor bortezomib is a potent inducer of zinc finger AN1-type domain 2a gene expression: role of heat shock factor 1 (HSF1)-heat shock factor 2 (HSF2) heterocomplexes.Reactive oxygen species-mediated activation of the Akt/ASK1/p38 signaling cascade and p21(Cip1) downregulation are required for shikonin-induced apoptosis.The translation inhibitor pateamine A prevents cachexia-induced muscle wasting in mice.MG132 plus apoptosis antigen-1 (APO-1) antibody cooperate to restore p53 activity inducing autophagy and p53-dependent apoptosis in HPV16 E6-expressing keratinocytes.Inactivation of the mTORC1-eukaryotic translation initiation factor 4E pathway alters stress granule formation.Characterization of Fragile X Mental Retardation Protein granules formation and dynamics in Drosophila.RNA Granules and Diseases: A Case Study of Stress Granules in ALS and FTLD DDX3 regulates endoplasmic reticulum stress-induced ATF4 expression.

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p21(WAF1/CIP1) upregulation through the stress granule-associated protein CUGBP1 confers resistance to bortezomib-mediated apoptosis.

http://www.wikidata.org/entity/Q33921377

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2011 nî lūn-bûn

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2011 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2011 թվականի մայիսին հրատարակված գիտական հոդված

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2011年の論文

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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p21(WAF1/CIP1) upregulation th ...... bortezomib-mediated apoptosis.

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Cristina Gareau

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Laetitia Coudert

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P2860

Competition of CUGBP1 and calreticulin for the regulation of p21 translation determines cell fate.

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

EDEN and EDEN-BP, a cis element and an associated factor that mediate sequence-specific mRNA deadenylation in Xenopus embryos.

CUG-BP binds to RNA substrates and recruits PARN deadenylase

Molecular basis for impaired muscle differentiation in myotonic dystrophy

HuR regulates p21 mRNA stabilization by UV light

Conserved GU-rich elements mediate mRNA decay by binding to CUG-binding protein 1

Inhibition of ribosome recruitment induces stress granule formation independently of eukaryotic initiation factor 2alpha phosphorylation

The deacetylase HDAC6 is a novel critical component of stress granules involved in the stress response

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