IL-23 suppresses innate immune response independently of IL-17A during carcinogenesis and metastasis. - Test2 - elhamkhani - TriplyDB

IL-23 suppresses innate immune response independently of IL-17A during carcinogenesis and metastasis.

IL-23 suppresses innate immune response independently of IL-17A during carcinogenesis and metastasis.

http://www.wikidata.org/entity/Q33929197

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Dendritic Cells in Oncolytic Virus-Based Anti-Cancer Therapy Glucocorticoid-induced reversal of interleukin-1β-stimulated inflammatory gene expression in human oviductal cells.Homeostatic regulation of Salmonella-induced mucosal inflammation and injury by IL-23 New insights into IL-12-mediated tumor suppression.Interleukin 23 promotes hepatocellular carcinoma metastasis via NF-kappa B induced matrix metalloproteinase 9 expression Interleukin-27 signaling promotes immunity against endogenously arising murine tumors.Preclinical development of AMG 139, a human antibody specifically targeting IL-23 Transmission of endoplasmic reticulum stress and pro-inflammation from tumor cells to myeloid cells.A distal enhancer in Il12b is the target of transcriptional repression by the STAT3 pathway and requires the basic leucine zipper (B-ZIP) protein NFIL3.TREM-1 is induced in tumor associated macrophages by cyclo-oxygenase pathway in human non-small cell lung cancer.Opposing roles for IL-23 and IL-12 in maintaining occult cancer in an equilibrium state.NK cells require IL-28R for optimal in vivo activity IL-23 in infections, inflammation, autoimmunity and cancer: possible role in HIV-1 and AIDS Expression of Th17 cell population regulatory cytokines in laryngeal carcinoma - Preliminary study Host immunity contributes to the anti-melanoma activity of BRAF inhibitors.Systemic delivery of chTNT-3/CpG immunoconjugates for immunotherapy in murine solid tumor models Increased Th17 cells in the tumor microenvironment is mediated by IL-23 via tumor-secreted prostaglandin E2.Cross-talk between myeloid-derived suppressor cells (MDSC), macrophages, and dendritic cells enhances tumor-induced immune suppression The inflammatory cytokine interleukin-23 is elevated in lung cancer, particularly small cell type.Baseline tumor growth and immune control in laboratory mice are significantly influenced by subthermoneutral housing temperature Tristetraprolin down-regulates IL-23 expression in colon cancer cells.Regulation of Interleukin-23 Expression in Health and Disease.Immune chaperone gp96 drives the contributions of macrophages to inflammatory colon tumorigenesis.3-Hydroxy-4,7-megastigmadien-9-one, isolated from Ulva pertusa, attenuates TLR9-mediated inflammatory response by down-regulating mitogen-activated protein kinase and NF-κB pathways.Interleukin 23 regulates proliferation of lung cancer cells in a concentration-dependent way in association with the interleukin-23 receptor.Alternatively spliced NKp30 isoforms affect the prognosis of gastrointestinal stromal tumors.Immune-checkpoint protein VISTA critically regulates the IL-23/IL-17 inflammatory axis.Toll-like receptor 3 regulates NK cell responses to cytokines and controls experimental metastasis.Hmgb1-IL-23-IL-17-IL-6-Stat3 axis promotes tumor growth in murine models of melanoma.Defective IL-23/IL-17 Axis Protects p47phox-/- Mice from Colon Cancer Targeting the IL-12/IL-23 axis: An alternative approach to removing tumor induced immune suppression.Myc Cooperates with Ras by Programming Inflammation and Immune Suppression.Interleukin (IL)-23 Stimulates IFN-γ Secretion by CD56bright Natural Killer Cells and Enhances IL-18-Driven Dendritic Cells Activation.Genetic variability in IL23R and risk of colorectal adenoma and colorectal cancer.IL-23 enhances the malignant properties of hepatoma cells by attenuation of HNF4α.

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IL-23 suppresses innate immune response independently of IL-17A during carcinogenesis and metastasis.

http://www.wikidata.org/entity/Q33929197

description

2010 nî lūn-bûn

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2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2010 թվականի ապրիլին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

@ast

IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

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type

label

IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

@ast

IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

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prefLabel

IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

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IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

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PNAS.1003251107

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Proceedings of the National Academy of Sciences of the United States of America

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IL-23 suppresses innate immune ...... carcinogenesis and metastasis.

@en

P2093

Andreas Möller

http://www.w3.org/2001/XMLSchema#string

Bianca von Scheidt

http://www.w3.org/2001/XMLSchema#string

Daniel M Andrews

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Martin Oft

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Michele W L Teng

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Nicole McLaughlin

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Shin Foong Ngiow

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P2860

Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12

A receptor for the heterodimeric cytokine IL-23 is composed of IL-12Rbeta1 and a novel cytokine receptor subunit, IL-23R

A functional interleukin 12 receptor complex is composed of two beta-type cytokine receptor subunits

A human colonic commensal promotes colon tumorigenesis via activation of T helper type 17 T cell responses

T helper 17 cells promote cytotoxic T cell activation in tumor immunity

Regulation of cutaneous malignancy by gammadelta T cells

IL-23 promotes tumour incidence and growth

Interleukin-17 family members and inflammation

A human natural killer cell subset provides an innate source of IL-22 for mucosal immunity

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

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10.1073/PNAS.1003251107

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18

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107

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Geoffrey R Hill

Yoichiro Iwakura

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2010-04-19T00:00:00Z

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43201512

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20404142

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2889517

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