p53 is a potential mediator of pregnancy and hormone-induced resistance to mammary carcinogenesis
about
Skp2B attenuates p53 function by inhibiting prohibitinCarcinogen inducibility in vivo and down-regulation of DMBT1 during breast carcinogenesisPhysical interaction and mutual transrepression between CCAAT/enhancer-binding protein beta and the p53 tumor suppressorProgesterone receptor knockout mice have an improved glucose homeostasis secondary to beta -cell proliferationA noncoding RNA is a potential marker of cell fate during mammary gland development.Hormonal control of p53 and chemopreventionProgesterone's role in mammary gland development and tumorigenesis as disclosed by experimental mouse geneticsPregnancy-induced changes in cell-fate in the mammary glandActivation of p53, inhibition of telomerase activity and induction of estrogen receptor beta are associated with the anti-growth effects of combination of ovarian hormones and retinoids in immortalized human mammary epithelial cellsThe protective role of pregnancy in breast cancerThe p53-estrogen receptor loop in cancerThe p53 tumor suppressor gene is regulated in vivo by nuclear factor 1-C2 in the mouse mammary gland during pregnancyInduction of a novel histone deacetylase 1/c-Myc/Mnt/Max complex formation is implicated in parity-induced refractoriness to mammary carcinogenesisLack of effects of postnatal exposure to a mixture of aryl hydrocarbon-receptor agonists on the development of methylnitrosourea-induced mammary tumors in sprague-dawley rats.Parity-induced decrease in systemic growth hormone alters mammary gland signaling: a potential role in pregnancy protection from breast cancer.Prognostic interaction between expression of p53 and estrogen receptor in patients with node-negative breast cancer: results from IBCSG Trials VIII and IXEffect of the p53-tristetraprolin-stathmin-1 pathway on trophoblasts at maternal-fetal interface.Deregulated estrogen receptor alpha and p53 heterozygosity collaborate in the development of mammary hyperplasia.Stress signaling and the shaping of the mammary tissue in development and cancer.Effects of parity and serum prolactin levels on the incidence and regression of DMBA-induced tumors in OFA hr/hr rats.Mitochondrial Hep27 is a c-Myb target gene that inhibits Mdm2 and stabilizes p53.Mechanisms of estrogen receptor antagonism toward p53 and its implications in breast cancer therapeutic response and stem cell regulation.The complex relationship between pregnancy and breast cancer.Transcriptional responses to estrogen and progesterone in mammary gland identify networks regulating p53 activity.Transcriptional regulation of estrogen receptor-alpha by p53 in human breast cancer cells.TP53 and breast cancer.Proliferation of estrogen receptor-alpha-positive mammary epithelial cells is restrained by transforming growth factor-beta1 in adult mice.P53 genotype as a determinant of ER expression and tamoxifen response in the MMTV-Wnt-1 model of mammary carcinogenesisMammary epithelial stem cells: transplantation and self-renewal analysis.p53 codon 72 polymorphism and recurrent pregnancy loss: a meta-analysis.Hormones and progeny of breast tumor cells.Estrogen and progesterone induce persistent increases in p53-dependent apoptosis and suppress mammary tumors in BALB/c-Trp53+/- miceParity induces differentiation and reduces Wnt/Notch signaling ratio and proliferation potential of basal stem/progenitor cells isolated from mouse mammary epitheliumEvidence that an early pregnancy causes a persistent decrease in the number of functional mammary epithelial stem cells--implications for pregnancy-induced protection against breast cancer.The in vivo Therapeutic Effect of Free Wanderer Powder ( xiāo yáo sǎn, Xiaoyaosan) on Mice with 4T1 Cell Induced Breast Cancer Model.New biological insights on the link between radiation exposure and breast cancer risk.ICRP Publication 131: Stem Cell Biology with Respect to Carcinogenesis Aspects of Radiological Protection.Functional significance of Aurora kinase A regulatory interactions with p53-ERα complex in human breast cancer cells.Estrogen receptor-alpha binds p53 tumor suppressor protein directly and represses its function.Antiestrogens are pro-apoptotic in normal human breast epithelial cells.
P2860
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P2860
p53 is a potential mediator of pregnancy and hormone-induced resistance to mammary carcinogenesis
description
2001 nî lūn-bûn
@nan
2001 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@ast
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@en
type
label
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@ast
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@en
prefLabel
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@ast
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@en
P2093
P2860
P356
P1476
p53 is a potential mediator of ...... ance to mammary carcinogenesis
@en
P2093
B W O'Malley
L Sivaraman
O M Conneely
P2860
P304
12379-12384
P356
10.1073/PNAS.221459098
P407
P577
2001-10-16T00:00:00Z