Inhibition of CHOP translation by a peptide encoded by an open reading frame localized in the chop 5'UTR - Test2 - elhamkhani - TriplyDB

Inhibition of CHOP translation by a peptide encoded by an open reading frame localized in the chop 5'UTR

Inhibition of CHOP translation by a peptide encoded by an open reading frame localized in the chop 5'UTR

http://www.wikidata.org/entity/Q33948307

about

Distinct cytoplasmic and nuclear functions of the stress induced protein DDIT3/CHOP/GADD153 Disease-associated mutations that alter the RNA structural ensemble A pharmacoproteomic approach implicates eukaryotic elongation factor 2 kinase in ER stress-induced cell death Translation of 5' leaders is pervasive in genes resistant to eIF2 repression Discovery and characterization of smORF-encoded bioactive polypeptides Unified translation repression mechanism for microRNAs and upstream AUGs Differential regulation of CHOP translation by phosphorylated eIF4E under stress conditions.A mitochondrial specific stress response in mammalian cells Functional RNA interference (RNAi) screen identifies system A neutral amino acid transporter 2 (SNAT2) as a mediator of arsenic-induced endoplasmic reticulum stress uORFdb--a comprehensive literature database on eukaryotic uORF biology.The myxoid/round cell liposarcoma fusion oncogene FUS-DDIT3 and the normal DDIT3 induce a liposarcoma phenotype in transfected human fibrosarcoma cells.Phosphorylation of eIF2 facilitates ribosomal bypass of an inhibitory upstream ORF to enhance CHOP translation.Stress-induced translation of ATF5 mRNA is regulated by the 5'-untranslated region.Interleukin-1 receptor-associated kinase-2 (IRAK2) is a critical mediator of endoplasmic reticulum (ER) stress signaling Zebrafish transgenic line huORFZ is an effective living bioindicator for detecting environmental toxicants.Transgenic zebrafish model to study translational control mediated by upstream open reading frame of human chop gene.Translational repression of mouse mu opioid receptor expression via leaky scanning.A Recurrent Germline Mutation in the 5'UTR of the Androgen Receptor Causes Complete Androgen Insensitivity by Activating Aberrant uORF Translation.Clinical significance of GADD153 expression in stage I non-small cell lung cancer.Translation reinitiation at alternative open reading frames regulates gene expression in an integrated stress response.Genome-wide search for novel human uORFs and N-terminal protein extensions using ribosomal footprinting Cellular response to endoplasmic reticulum stress: a matter of life or death.Upstream AUGs and upstream ORFs can regulate the downstream ORF in Plasmodium falciparum.Ribosome Elongation Stall Directs Gene-specific Translation in the Integrated Stress Response.Eukaryotic initiation factor 2 phosphorylation and translational control in metabolism.The role of endoplasmic reticulum stress in maintaining and targeting multiple myeloma: a double-edged sword of adaptation and apoptosis.Overexpression of CHOP in Myelinating Cells Does Not Confer a Significant Phenotype under Normal or Metabolic Stress Conditions.Global cellular response to chemotherapy-induced apoptosis.The A2A adenosine receptor is a dual coding gene: a novel mechanism of gene usage and signal transduction.Translational regulation of BACE-1 expression in neuronal and non-neuronal cells.DDIT3 Expression in Liposarcoma Development.The regulatory potential of upstream open reading frames in eukaryotic gene expression.The African swine fever virus DP71L protein recruits the protein phosphatase 1 catalytic subunit to dephosphorylate eIF2alpha and inhibits CHOP induction but is dispensable for these activities during virus infection.Regulatory mechanisms, expression levels and proliferation effects of the FUS-DDIT3 fusion oncogene in liposarcoma.Upstream Open Reading Frames Differentially Regulate Gene-specific Translation in the Integrated Stress Response IRE1α induces thioredoxin-interacting protein to activate the NLRP3 inflammasome and promote programmed cell death under irremediable ER stress.Mitochondrial dysfunction in cancer: Potential roles of ATF5 and the mitochondrial UPR.Expression of human Hemojuvelin (HJV) is tightly regulated by two upstream open reading frames in HJV mRNA that respond to iron overload in hepatic cells.Identification of novel Arabidopsis thaliana upstream open reading frames that control expression of the main coding sequences in a peptide sequence-dependent manner.Leaky scanning and reinitiation regulate BACE1 gene expression.

P2860

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P2860

Inhibition of CHOP translation by a peptide encoded by an open reading frame localized in the chop 5'UTR

http://www.wikidata.org/entity/Q33948307

description

2001 nî lūn-bûn

@nan

2001 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2001 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2001年の論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年論文

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2001年论文

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name

Inhibition of CHOP translation ...... me localized in the chop 5'UTR

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Inhibition of CHOP translation ...... me localized in the chop 5'UTR

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type

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Inhibition of CHOP translation ...... me localized in the chop 5'UTR

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Inhibition of CHOP translation ...... me localized in the chop 5'UTR

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prefLabel

Inhibition of CHOP translation ...... me localized in the chop 5'UTR

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Inhibition of CHOP translation ...... me localized in the chop 5'UTR

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P1433

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P1433

Nucleic Acids Research

P1476

Inhibition of CHOP translation ...... me localized in the chop 5'UTR

@en

P2093

Bruhat A

http://www.w3.org/2001/XMLSchema#string

Carraro V

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Fafournoux P

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Ferrara M

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Jousse C

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Urano F

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P2860

CHOP enhancement of gene transcription by interactions with Jun/Fos AP-1 complex proteins

Fusion of the dominant negative transcription regulator CHOP with a novel gene FUS by translocation t(12;16) in malignant liposarcoma

Fusion of CHOP to a novel RNA-binding protein in human myxoid liposarcoma

Identification of novel stress-induced genes downstream of chop

Point mutations define a sequence flanking the AUG initiator codon that modulates translation by eukaryotic ribosomes

Rearrangement of the transcription factor gene CHOP in myxoid liposarcomas with t(12;16)(q13;p11)

A novel effector domain from the RNA-binding protein TLS or EWS is required for oncogenic transformation by CHOP

Regulated expression of three C/EBP isoforms during adipose conversion of 3T3-L1 cells

Fusion of the EWS and CHOP genes in myxoid liposarcoma

Physical and functional association between GADD153 and CCAAT/enhancer-binding protein beta during cellular stress

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4341-4351

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scholarly article

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10.1093/NAR/29.21.4341

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21

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29

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2001-11-01T00:00:00Z

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279243334

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11691921

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60176

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