Neurodegeneration in a transgenic mouse model of multiple system atrophy is associated with altered expression of oligodendroglial-derived neurotrophic factors.
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Review: Multiple system atrophy: emerging targets for interventional therapiesMultiple system atrophy: a clinical and neuropathological perspectiveGlia and alpha-synuclein in neurodegeneration: A complex interactionExploring myelin dysfunction in multiple system atrophyGlia: an emerging target for neurological disease therapyAnimal models of multiple system atrophyThe contribution of alpha synuclein to neuronal survival and function - Implications for Parkinson's diseaseAnimal modeling an oligodendrogliopathy--multiple system atrophyAntidepressants reduce neuroinflammatory responses and astroglial alpha-synuclein accumulation in a transgenic mouse model of multiple system atrophySerine 421 regulates mutant huntingtin toxicity and clearance in mice.Time course and progression of wild type α-synuclein accumulation in a transgenic mouse model.Next-generation active immunization approach for synucleinopathies: implications for Parkinson's disease clinical trials.PGC-1α rescues Huntington's disease proteotoxicity by preventing oxidative stress and promoting TFEB function.Widespread microRNA dysregulation in multiple system atrophy - disease-related alteration in miR-96.Towards translational therapies for multiple system atrophyNovel oligodendroglial alpha synuclein viral vector models of multiple system atrophy: studies in rodents and nonhuman primates.α-Synuclein impairs oligodendrocyte progenitor maturation in multiple system atrophyReducing C-terminal-truncated alpha-synuclein by immunotherapy attenuates neurodegeneration and propagation in Parkinson's disease-like models.Cerebrolysin™ efficacy in a transgenic model of tauopathy: role in regulation of mitochondrial structure.Impaired eukaryotic translation initiation factor 2B activity specifically in oligodendrocytes reproduces the pathology of vanishing white matter disease in mice.FAS-dependent cell death in α-synuclein transgenic oligodendrocyte models of multiple system atrophyPreserved functional autonomic phenotype in adult mice overexpressing moderate levels of human alpha-synuclein in oligodendrocytes.Novel therapeutic approaches in multiple system atrophyIntact olfaction in a mouse model of multiple system atrophy.Environmental neurotoxic challenge of conditional alpha-synuclein transgenic mice predicts a dopaminergic olfactory-striatal interplay in early PD.A novel triple repeat mutant tau transgenic model that mimics aspects of pick's disease and fronto-temporal tauopathies.Active immunization against alpha-synuclein ameliorates the degenerative pathology and prevents demyelination in a model of multiple system atrophyLenalidomide reduces microglial activation and behavioral deficits in a transgenic model of Parkinson's disease.Filamentous aggregations of phosphorylated α-synuclein in Schwann cells (Schwann cell cytoplasmic inclusions) in multiple system atrophyNeuronal to oligodendroglial α-synuclein redistribution in a double transgenic model of multiple system atrophy.Overexpression of α-synuclein in oligodendrocytes does not increase susceptibility to focal striatal excitotoxicity.A brain-targeted, modified neurosin (kallikrein-6) reduces α-synuclein accumulation in a mouse model of multiple system atrophy.Systemic Central Nervous System (CNS)-targeted Delivery of Neuropeptide Y (NPY) Reduces Neurodegeneration and Increases Neural Precursor Cell Proliferation in a Mouse Model of Alzheimer Disease.Models of multiple system atrophyInflammatory pre-conditioning restricts the seeded induction of α-synuclein pathology in wild type mice.Glial dysfunction in the pathogenesis of α-synucleinopathies: emerging concepts.The role of glia in α-synucleinopathies.Immunotherapy for neurodegenerative diseases: focus on α-synucleinopathies.Oligodendroglia and neurotrophic factors in neurodegeneration.Multiple system atrophy of the cerebellar type: clinical state of the art.
P2860
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P2860
Neurodegeneration in a transgenic mouse model of multiple system atrophy is associated with altered expression of oligodendroglial-derived neurotrophic factors.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@ast
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@en
type
label
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@ast
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@en
prefLabel
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@ast
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@en
P2093
P2860
P50
P1476
Neurodegeneration in a transge ...... -derived neurotrophic factors.
@en
P2093
Anthony Adame
Chandra Inglis
Christina Patrick
Kristen Whitney
Michael Mante
P2860
P304
P356
10.1523/JNEUROSCI.0567-10.2010
P407
P577
2010-05-01T00:00:00Z