Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
about
Characterization of human exonuclease 1 in complex with mismatch repair proteins, subcellular localization and association with PCNAIntegrating phenotypic and expression profiles to map arsenic-response networksFunctional residues on the surface of the N-terminal domain of yeast Pms1Differential ATP binding and intrinsic ATP hydrolysis by amino-terminal domains of the yeast Mlh1 and Pms1 proteins.New insights into the mechanism of DNA mismatch repairAssessing pathogenicity of MLH1 variants by co-expression of human MLH1 and PMS2 genes in yeast.Spontaneous hepatocellular carcinoma is reduced in transgenic mice overexpressing human O6- methylguanine-DNA methyltransferase.Systematic mutagenesis of the Saccharomyces cerevisiae MLH1 gene reveals distinct roles for Mlh1p in meiotic crossing over and in vegetative and meiotic mismatch repairDNA binding by yeast Mlh1 and Pms1: implications for DNA mismatch repair.Methylation-induced G(2)/M arrest requires a full complement of the mismatch repair protein hMLH1Single molecule studies of DNA mismatch repair.Modulation of microRNA processing by mismatch repair protein MutLα.Mismatch repair hMSH2, hMLH1, hMSH6 and hPMS2 mRNA expression profiles in precancerous and cancerous urotheliumEvolving views of DNA replication (in)fidelity.Characterization of a highly conserved binding site of Mlh1 required for exonuclease I-dependent mismatch repair.Novel Implications in Molecular Diagnosis of Lynch Syndrome.Ntg2p, a Saccharomyces cerevisiae DNA N-glycosylase/apurinic or apyrimidinic lyase involved in base excision repair of oxidative DNA damage, interacts with the DNA mismatch repair protein Mlh1p. Identification of a Mlh1p binding motif.Over-Expression of YLR162W in Saccharomyces cerevisiae Inhibits Cell Proliferation and Renders Cells Susceptible to the Hypoxic Conditions Induced by Cobalt ChlorideImplication of protein kinase C in the regulation of DNA mismatch repair protein expression and function.The elevated expression of a mismatch repair protein is a predictor for biochemical recurrence after radical prostatectomy.Overexpression of MutL homolog 1 and MutS homolog 2 proteins have reversed prognostic implications for stage I-II colon cancer patients.Yeast mutator phenotype enforced by Arabidopsis PMS1 expression.Genomic Instability Promoted by Overexpression of Mismatch Repair Factors in Yeast: A Model for Understanding Cancer Progression.PMS1 from Arabidopsis thaliana: optimization of protein overexpression in Escherichia coli.
P2860
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P2860
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
description
2001 nî lūn-bûn
@nan
2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@ast
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@en
type
label
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@ast
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@en
prefLabel
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@ast
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@en
P2093
P2860
P1476
Inactivation of DNA mismatch repair by increased expression of yeast MLH1.
@en
P2093
C A Afshari
K J Martin
P R Bushel
P V Shcherbakova
S E Bennett
T A Kunkel
P2860
P304
P356
10.1128/MCB.21.3.940-951.2001
P407
P577
2001-02-01T00:00:00Z