Interruption of the cydB locus in Brucella abortus attenuates intracellular survival and virulence in the mouse model of infection.
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Whole-Genome Analyses of Speciation Events in Pathogenic BrucellaeThe genome of M. acetivorans reveals extensive metabolic and physiological diversityThe genome sequence of the facultative intracellular pathogen Brucella melitensis.Molecular host-pathogen interaction in brucellosis: current understanding and future approaches to vaccine development for mice and humansHost cell-free growth of the Q fever bacterium Coxiella burnetiiContrasting Lifestyles Within the Host CellMariner mutagenesis of Brucella melitensis reveals genes with previously uncharacterized roles in virulence and survival.The airborne metagenome in an indoor urban environment.Genome sequence of Brucella abortus vaccine strain S19 compared to virulent strains yields candidate virulence genesThe time course of the transcriptomic response of Sinorhizobium meliloti 1021 following a shift to acidic pH.RegA Plays a Key Role in Oxygen-Dependent Establishment of Persistence and in Isocitrate Lyase Activity, a Critical Determinant of In vivo Brucella suis Pathogenicity.Opsonized virulent Brucella abortus replicates within nonacidic, endoplasmic reticulum-negative, LAMP-1-positive phagosomes in human monocytesTwo coregulated efflux transporters modulate intracellular heme and protoporphyrin IX availability in Streptococcus agalactiae.Differential use of the two high-oxygen-affinity terminal oxidases of Brucella suis for in vitro and intramacrophagic multiplication.Induction of dnaK through its native heat shock promoter is necessary for intramacrophagic replication of Brucella suis.Virulence criteria for Brucella abortus strains as determined by interferon regulatory factor 1-deficient mice.The analysis of the intramacrophagic virulome of Brucella suis deciphers the environment encountered by the pathogen inside the macrophage host cellDelayed accumulation of intestinal coliform bacteria enhances life span and stress resistance in Caenorhabditis elegans fed respiratory deficient E. coli.Characterization and protective property of Brucella abortus cydC and looP mutants.Evaluation of protection afforded by Brucella abortus and Brucella melitensis unmarked deletion mutants exhibiting different rates of clearance in BALB/c mice.Isolation and characterization of mini-Tn5Km2 insertion mutants of Brucella abortus deficient in internalization and intracellular growth in HeLa cells.Conservation analysis of the CydX protein yields insights into small protein identification and evolution.The cytochrome bd respiratory oxygen reductasesThe cytochrome bd-type quinol oxidase is important for survival of Mycobacterium smegmatis under peroxide and antibiotic-induced stress.Different roles of the two high-oxygen-affinity terminal oxidases of Brucella suis: Cytochrome c oxidase, but not ubiquinol oxidase, is required for persistence in miceThe small protein CydX is required for function of cytochrome bd oxidase in Brucella abortusBrucella abortus requires the heme transporter BhuA for maintenance of chronic infection in BALB/c mice.Identification of novel Listeria monocytogenes secreted virulence factors following mutational activation of the central virulence regulator, PrfA.SodA is a major metabolic antioxidant in Brucella abortus 2308 that plays a significant, but limited, role in the virulence of this strain in the mouse model.The Brucella abortus general stress response system regulates chronic mammalian infection and is controlled by phosphorylation and proteolysis.Cytochrome bd Displays Significant Quinol Peroxidase Activity.Survival of the fittest: how Brucella strains adapt to their intracellular niche in the host.Cytochrome bd oxidase and nitric oxide: from reaction mechanisms to bacterial physiology.Internal affairs: investigating the Brucella intracellular lifestyle.Cysteine is exported from the Escherichia coli cytoplasm by CydDC, an ATP-binding cassette-type transporter required for cytochrome assembly.Heme-heme and heme-ligand interactions in the di-heme oxygen-reducing site of cytochrome bd from Escherichia coli revealed by nanosecond absorption spectroscopy.FixL-like sensor FlbS of Brucella abortus binds haem and is necessary for survival within eukaryotic cells.A fluorescence-based reporter for monitoring expression of mycobacterial cytochrome bd in response to antibacterials and during infection.Brucella abortus ΔrpoE1 confers protective immunity against wild type challenge in a mouse model of brucellosis.Brucella abortus rough mutants are cytopathic for macrophages in culture.
P2860
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P2860
Interruption of the cydB locus in Brucella abortus attenuates intracellular survival and virulence in the mouse model of infection.
description
2001 nî lūn-bûn
@nan
2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Interruption of the cydB locus ...... the mouse model of infection.
@ast
Interruption of the cydB locus ...... the mouse model of infection.
@en
type
label
Interruption of the cydB locus ...... the mouse model of infection.
@ast
Interruption of the cydB locus ...... the mouse model of infection.
@en
prefLabel
Interruption of the cydB locus ...... the mouse model of infection.
@ast
Interruption of the cydB locus ...... the mouse model of infection.
@en
P2093
P2860
P1476
Interruption of the cydB locus ...... the mouse model of infection.
@en
P2093
P2860
P304
P356
10.1128/JB.183.8.2454-2462.2001
P407
P577
2001-04-01T00:00:00Z