Senescent mouse cells fail to overtly regulate the HIRA histone chaperone and do not form robust Senescence Associated Heterochromatin Foci. - Test2 - elhamkhani - TriplyDB

Senescent mouse cells fail to overtly regulate the HIRA histone chaperone and do not form robust Senescence Associated Heterochromatin Foci.

Senescent mouse cells fail to overtly regulate the HIRA histone chaperone and do not form robust Senescence Associated Heterochromatin Foci.

http://www.wikidata.org/entity/Q33997986

about

Markers of cellular senescence. Telomere shortening as a marker of cellular senescence Reorganization of chromosome architecture in replicative cellular senescence Drosophila Yemanuclein and HIRA cooperate for de novo assembly of H3.3-containing nucleosomes in the male pronucleus Higher-order unfolding of satellite heterochromatin is a consistent and early event in cell senescence Zinc finger protein 637 protects cells against oxidative stress-induced premature senescence by mTERT-mediated telomerase activity and telomere maintenance.Ring-like distribution of constitutive heterochromatin in bovine senescent cells.Aging, cellular senescence, and cancer Minichromosome maintenance 2 bound with retroviral Gp70 is localized to cytoplasm and enhances DNA-damage-induced apoptosis.Age-associated increase in heterochromatic marks in murine and primate tissues.Distinct p53 transcriptional programs dictate acute DNA-damage responses and tumor suppression.Unfolding the story of chromatin organization in senescent cells.Detection of senescence-associated heterochromatin foci (SAHF).The Mre11 complex suppresses oncogene-driven breast tumorigenesis and metastasis.GSK3β activity is essential for senescence-associated heterochromatin foci (SAHF) formation induced by HMGA2 in WI38 cells.Regulation of oncogene-induced cell cycle exit and senescence by chromatin modifiers Tumor-produced, active interleukin-1β regulates gene expression in carcinoma-associated fibroblasts.Fibroblasts produce brain-derived neurotrophic factor and induce mesenchymal transition of oral tumor cells.Techniques to Induce and Quantify Cellular Senescence.Temporally distinct roles for tumor suppressor pathways in cell cycle arrest and cellular senescence in Cyclin D1-driven tumor Contrasting behavior of the p18INK4c and p16INK4a tumor suppressors in both replicative and oncogene-induced senescence.Postmitotic neurons develop a p21-dependent senescence-like phenotype driven by a DNA damage response Epigenetic Mechanisms Impacting Aging: A Focus on Histone Levels and Telomeres.[Cellular senescence and the myth of Janus].

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P2860

Senescent mouse cells fail to overtly regulate the HIRA histone chaperone and do not form robust Senescence Associated Heterochromatin Foci.

http://www.wikidata.org/entity/Q33997986

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

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name

Senescent mouse cells fail to ...... sociated Heterochromatin Foci.

@ast

Senescent mouse cells fail to ...... sociated Heterochromatin Foci.

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type

label

Senescent mouse cells fail to ...... sociated Heterochromatin Foci.

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Senescent mouse cells fail to ...... sociated Heterochromatin Foci.

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prefLabel

Senescent mouse cells fail to ...... sociated Heterochromatin Foci.

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Senescent mouse cells fail to ...... sociated Heterochromatin Foci.

@en

P2093

Alyssa L Kennedy

http://www.w3.org/2001/XMLSchema#string

F Brad Johnson

http://www.w3.org/2001/XMLSchema#string

Greg H Enders

http://www.w3.org/2001/XMLSchema#string

Peter D Adams

http://www.w3.org/2001/XMLSchema#string

Rugang Zhang

http://www.w3.org/2001/XMLSchema#string

Tony McBryan

http://www.w3.org/2001/XMLSchema#string

P2860

HIRA, the human homologue of yeast Hir1p and Hir2p, is a novel cyclin-cdk2 substrate whose expression blocks S-phase progression

A biomarker that identifies senescent human cells in culture and in aging skin in vivo

Remodeling of chromatin structure in senescent cells and its potential impact on tumor suppression and aging

Molecular dissection of formation of senescence-associated heterochromatin foci

Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase B

CDK inhibitors: positive and negative regulators of G1-phase progression

The Wnt signaling pathway in development and disease

Creation of human tumour cells with defined genetic elements

Tumorigenic conversion of primary embryo fibroblasts requires at least two cooperating oncogenes

Histone macroH2A1 is concentrated in the inactive X chromosome of female mammals

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16

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10.1186/1747-1028-5-16

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5

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2010-06-22T00:00:00Z

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44693906

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1003550434

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20569479

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molecular chaperones

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2904742

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