Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans. - Test2 - elhamkhani - TriplyDB

Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans.

Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans.

http://www.wikidata.org/entity/Q34028910

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Non-steroid agents for idiopathic pulmonary fibrosis Bone morphogenetic protein-4 inhibitor gremlin is overexpressed in idiopathic pulmonary fibrosis WNT5A is a regulator of fibroblast proliferation and resistance to apoptosis Inhibition and role of let-7d in idiopathic pulmonary fibrosis Gene expression profiles of acute exacerbations of idiopathic pulmonary fibrosis WNT1-inducible signaling protein-1 mediates pulmonary fibrosis in mice and is upregulated in humans with idiopathic pulmonary fibrosis Application of microarray technology in pulmonary diseases The absence of reactive oxygen species production protects mice against bleomycin-induced pulmonary fibrosis CD8+ T lymphocytes in lung tissue from patients with idiopathic pulmonary fibrosis Overexpression of cathepsin K during silica-induced lung fibrosis and control by TGF-beta.Up-regulation and profibrotic role of osteopontin in human idiopathic pulmonary fibrosis Wnt signalling in lung development and diseases Macrophage metalloelastase (MMP-12) deficiency does not alter bleomycin-induced pulmonary fibrosis in mice.Matrix Metalloproteinases in Non-Neoplastic Disorders.Roles of transcriptional factor 7 in production of inflammatory factors for lung diseases Diverse functions of matrix metalloproteinases during fibrosis Always cleave up your mess: targeting collagen degradation to treat tissue fibrosis Epigenomics of idiopathic pulmonary fibrosis Molecular classification of idiopathic pulmonary fibrosis: personalized medicine, genetics and biomarkers Personalized medicine: applying 'omics' to lung fibrosis Peripheral blood biomarkers in idiopathic pulmonary fibrosis Molecular biomarkers in idiopathic pulmonary fibrosis Evolving genomic approaches to idiopathic pulmonary fibrosis: moving beyond genes Heterogeneous gene expression signatures correspond to distinct lung pathologies and biomarkers of disease severity in idiopathic pulmonary fibrosis Role of matrix metalloproteinases in the pathogenesis of idiopathic pulmonary fibrosis Update on therapeutic management of idiopathic pulmonary fibrosis Matrix metalloproteinase-9 activates TGF-β and stimulates fibroblast contraction of collagen gels Matrilysin (MMP-7) is a major matrix metalloproteinase upregulated in biliary atresia-associated liver fibrosis An official ATS/ERS/JRS/ALAT statement: idiopathic pulmonary fibrosis: evidence-based guidelines for diagnosis and management Matrix metalloproteinases as therapeutic targets for idiopathic pulmonary fibrosis Genetically manipulated mouse models of lung disease: potential and pitfalls Potential role of the inflammasome-derived inflammatory cytokines in pulmonary fibrosis Matrix metalloproteinase-10: a novel biomarker for idiopathic pulmonary fibrosis Associations of MMP1, MMP2 and MMP3 Genes Polymorphism with Coal Workers' Pneumoconiosis in Chinese Han Population MMP1 and MMP7 as potential peripheral blood biomarkers in idiopathic pulmonary fibrosis Gene expression profiling of pulmonary fibrosis identifies Twist1 as an antiapoptotic molecular "rectifier" of growth factor signaling The peripheral blood transcriptome identifies the presence and extent of disease in idiopathic pulmonary fibrosis Novel rank-based statistical methods reveal microRNAs with differential expression in multiple cancer types New mechanisms of pulmonary fibrosis.What differentiates normal lung repair and fibrosis? Inflammation, resolution of repair, and fibrosis

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Gene expression analysis reveals matrilysin as a key regulator of pulmonary fibrosis in mice and humans.

http://www.wikidata.org/entity/Q34028910

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2002 nî lūn-bûn

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Gene expression analysis revea ...... y fibrosis in mice and humans.

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Proceedings of the National Academy of Sciences of the United States of America

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Gene expression analysis revea ...... y fibrosis in mice and humans.

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Amir Ben-Dor

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Annie Pardo

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Barbara DeLustro

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David Morris

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Dean Sheppard

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Elsie Eugui

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Fengrong Zuo

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John Allard

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Lance Lollini

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Moises Selman

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P2860

Cluster analysis and display of genome-wide expression patterns

Matrix metalloproteinases cleave tissue factor pathway inhibitor. Effects on coagulation

Regulation of intestinal alpha-defensin activation by the metalloproteinase matrilysin in innate host defense

Idiopathic pulmonary fibrosis: prevailing and evolving hypotheses about its pathogenesis and implications for therapy

DNA microarrays: raising the profile.

The use of microarrays in medicine.

Differential expression of extracellular matrix remodeling genes in a murine model of bleomycin-induced pulmonary fibrosis.

Bacterial exposure induces and activates matrilysin in mucosal epithelial cells.

Global analysis of gene expression in pulmonary fibrosis reveals distinct programs regulating lung inflammation and fibrosis.

Matrilysin expression and function in airway epithelium.

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10.1073/PNAS.092134099

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9

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99

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Naftali Kaminski

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11385672

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pulmonary fibrosis

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