High-resolution mapping of the 11q13 amplicon and identification of a gene, TAOS1, that is amplified and overexpressed in oral cancer cells.
about
Epigenetic inactivation and aberrant transcription of CSMD1 in squamous cell carcinoma cell lines.Calcium-activated chloride channel ANO1 promotes breast cancer progression by activating EGFR and CAMK signalingAnoctamin/TMEM16 family members are Ca2+-activated Cl- channelsCellular functions of TMEM16/anoctaminInverted duplication pattern in anaphase bridges confirms the breakage-fusion-bridge (BFB) cycle model for 11q13 amplification.Oral cancer overexpressed 1 (ORAOV1) regulates cell cycle and apoptosis in cervical cancer HeLa cells.Genomic profiling of advanced-stage oral cancers reveals chromosome 11q alterations as markers of poor clinical outcome.Frequent amplification of ORAOV1 gene in esophageal squamous cell cancer promotes an aggressive phenotype via proline metabolism and ROS production.Overexpression of KLF13 and FGFR3 in oral cancer cells.Algorithms to model single gene, single chromosome, and whole genome copy number changes jointly in tumor phylogenetics.The TMEM16 protein family: a new class of chloride channels?Integrative molecular characterization of head and neck cancer cell model genomesComparative analysis of algorithms for integration of copy number and expression data.Regulation of apoptosis by fau revealed by functional expression cloning and antisense expression.Characterization of genetic rearrangements in esophageal squamous carcinoma cell lines by a combination of M-FISH and array-CGH: further confirmation of some split genomic regions in primary tumors.Chromosomal imbalances in oral squamous cell carcinoma: examination of 31 cell lines and review of the literature.Identification of TRPC6 as a possible candidate target gene within an amplicon at 11q21-q22.2 for migratory capacity in head and neck squamous cell carcinomasChaperone Hsp47 Drives Malignant Growth and Invasion by Modulating an ECM Gene NetworkInhibition of Calcium-Activated Chloride Channel ANO1/TMEM16A Suppresses Tumor Growth and Invasion in Human Lung Cancer.Malignant and nonmalignant gene signatures in squamous head and neck cancerThe deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.ANO1 plays a critical role in prostatic hyperplasia.Anoctamin 1 (TMEM16A) is essential for testosterone-induced prostate hyperplasia.Chromosomal instability in oral cancer cells.The influence of clinical and demographic risk factors on the establishment of head and neck squamous cell carcinoma cell lines.TMEM16A/ANO1 is differentially expressed in HPV-negative versus HPV-positive head and neck squamous cell carcinoma through promoter methylationGenomic amplification upregulates estrogen-related receptor alpha and its depletion inhibits oral squamous cell carcinoma tumors in vivoPrognostic significance of cortactin levels in head and neck squamous cell carcinoma: comparison with epidermal growth factor receptor status11q13 amplification status and human papillomavirus in relation to p16 expression defines two distinct etiologies of head and neck tumoursSuppression of 14-3-3γ-mediated surface expression of ANO1 inhibits cancer progression of glioblastoma cells.Physiological roles and diseases of Tmem16/Anoctamin proteins: are they all chloride channels?Shikonin Inhibits Intestinal Calcium-Activated Chloride Channels and Prevents Rotaviral Diarrhea.Association between CCND1 G/A870 polymorphism, allele-specific amplification, cyclin D1 expression, and survival in esophageal and lung carcinoma.Decreased expression of miR-125b and miR-100 in oral cancer cells contributes to malignancyCytogenetic alterations and their molecular genetic correlates in head and neck squamous cell carcinoma: a next generation window to the biology of disease.Frequent chromosomal aberrations and candidate genes in head and neck squamous cell carcinoma.TMEM16A/ANO1 suppression improves response to antibody-mediated targeted therapy of EGFR and HER2/ERBB2.Inhibition of calcium-activated chloride channel ANO1 suppresses proliferation and induces apoptosis of epithelium originated cancer cells.An update on the implications of cyclin D1 in oral carcinogenesis.RNAi-mediated downregulation of oral cancer overexpressed 1 (ORAOV1) inhibits vascular endothelial cell proliferation, migration, invasion, and tube formation.
P2860
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P2860
High-resolution mapping of the 11q13 amplicon and identification of a gene, TAOS1, that is amplified and overexpressed in oral cancer cells.
description
2002 nî lūn-bûn
@nan
2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
High-resolution mapping of the ...... xpressed in oral cancer cells.
@ast
High-resolution mapping of the ...... xpressed in oral cancer cells.
@en
High-resolution mapping of the ...... xpressed in oral cancer cells.
@nl
type
label
High-resolution mapping of the ...... xpressed in oral cancer cells.
@ast
High-resolution mapping of the ...... xpressed in oral cancer cells.
@en
High-resolution mapping of the ...... xpressed in oral cancer cells.
@nl
prefLabel
High-resolution mapping of the ...... xpressed in oral cancer cells.
@ast
High-resolution mapping of the ...... xpressed in oral cancer cells.
@en
High-resolution mapping of the ...... xpressed in oral cancer cells.
@nl
P2860
P356
P1476
High-resolution mapping of the ...... expressed in oral cancer cells
@en
P2093
P2860
P304
11369-11374
P356
10.1073/PNAS.172285799
P407
P577
2002-08-09T00:00:00Z