The repressing function of the oncoprotein BCL-3 requires CtBP, while its polyubiquitination and degradation involve the E3 ligase TBLR1. - Test2 - elhamkhani - TriplyDB

The repressing function of the oncoprotein BCL-3 requires CtBP, while its polyubiquitination and degradation involve the E3 ligase TBLR1.

The repressing function of the oncoprotein BCL-3 requires CtBP, while its polyubiquitination and degradation involve the E3 ligase TBLR1.

http://www.wikidata.org/entity/Q34045748

about

Atypical IκB proteins - nuclear modulators of NF-κB signaling BCL-3 expression promotes colorectal tumorigenesis through activation of AKT signalling The tumor promoter and NF-κB modulator Bcl-3 regulates splenic B cell development.Triptolide induces cell-cycle arrest and apoptosis of human multiple myeloma cells in vitro via altering expression of histone demethylase LSD1 and JMJD2B.The transcriptional specificity of NF-κB dimers is coded within the κB DNA response elements NF-κB-induced KIAA1199 promotes survival through EGFR signalling.C-Terminal Binding Protein: A Molecular Link between Metabolic Imbalance and Epigenetic Regulation in Breast Cancer.Transducin (β)-like 1 X-linked receptor 1 promotes proliferation and tumorigenicity in human breast cancer via activation of beta-catenin signaling.BCL-3 attenuation of TNFA expression involves an incoherent feed-forward loop regulated by chromatin structure.TBL1XR1/TP63: a novel recurrent gene fusion in B-cell non-Hodgkin lymphoma.MDM2 restrains estrogen-mediated AKT activation by promoting TBK1-dependent HPIP degradation Role of Bcl-3 in solid tumors Ubiquitination and the Ubiquitin-Proteasome System as regulators of transcription and transcription factors in epithelial mesenchymal transition of cancer.It takes two to tango: IκBs, the multifunctional partners of NF-κB.TBL1XR1 in physiological and pathological states TBLR1 as an androgen receptor (AR) coactivator selectively activates AR target genes to inhibit prostate cancer growth.Expression of Id proteins is regulated by the Bcl-3 proto-oncogene in prostate cancer.Involvement of CtBP2 in LPS-induced microglial activation.Cytoplasmic, full length and novel cleaved variant, TBLR1 reduces apoptosis in prostate cancer under androgen deprivation.

P2860

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P2860

The repressing function of the oncoprotein BCL-3 requires CtBP, while its polyubiquitination and degradation involve the E3 ligase TBLR1.

http://www.wikidata.org/entity/Q34045748

description

2010 nî lūn-bûn

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2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

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2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Alain Chariot

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André Gothot

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Aurore Keutgens

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Benoît Hennuy

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Jean-Paul Chapelle

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Kateryna Shostak

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Marie Aussems

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Patrick Viatour

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Pierre Close

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Xin Zhang

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P2860

CoREST: a functional corepressor required for regulation of neural-specific gene expression

The Bcl-3 oncoprotein acts as a bridging factor between NF-kappaB/Rel and nuclear co-regulators

The N-CoR-HDAC3 nuclear receptor corepressor complex inhibits the JNK pathway through the integral subunit GPS2

Purification and functional characterization of the human N-CoR complex: the roles of HDAC3, TBL1 and TBLR1

Expression of the Bcl-3 proto-oncogene suppresses p53 activation

Inactivation of the CYLD deubiquitinase by HPV E6 mediates hypoxia-induced NF-kappaB activation

Isolation of a novel human cDNA (rhoHP1) homologous to rho genes

TBL1 and TBLR1 phosphorylation on regulated gene promoters overcomes dual CtBP and NCoR/SMRT transcriptional repression checkpoints

Reading and function of a histone code involved in targeting corepressor complexes for repression

Histone demethylation mediated by the nuclear amine oxidase homolog LSD1

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4006-4021

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scholarly article

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10.1128/MCB.01600-09

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16

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30

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Marianne Fillet

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2010-06-14T00:00:00Z

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20547759

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2916444

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