The myocardial contractile response to physiological stress improves with high saturated fat feeding in heart failure.
about
Mitochondrial fatty acid oxidation alterations in heart failure, ischaemic heart disease and diabetic cardiomyopathyCardioprotective effects of dietary lipids evident in the time-dependent alterations of cardiac function and gene expression following myocardial infarction.Acute consumption of a high-fat diet prior to ischemia-reperfusion results in cardioprotection through NF-κB-dependent regulation of autophagic pathways.Chronic inhibition of pyruvate dehydrogenase in heart triggers an adaptive metabolic response.Caloric excess or restriction mediated modulation of metabolic enzyme acetylation-proposed effects on cardiac growth and function.Changes in myofilament proteins, but not Ca²⁺ regulation, are associated with a high-fat diet-induced improvement in contractile function in heart failure.Dietary fat and heart failure: moving from lipotoxicity to lipoprotection.Normalizing the metabolic phenotype after myocardial infarction: impact of subchronic high fat feedingChronic ingestion of a Western diet increases O-linked-β-N-acetylglucosamine (O-GlcNAc) protein modification in the rat heart.Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heartInhibition of G-protein-coupled Receptor Kinase 2 Prevents the Dysfunctional Cardiac Substrate Metabolism in Fatty Acid Synthase Transgenic Mice.Dietary saturated fat and docosahexaenoic acid differentially effect cardiac mitochondrial phospholipid fatty acyl composition and Ca(2+) uptake, without altering permeability transition or left ventricular function.Aldehyde stress and up-regulation of Nrf2-mediated antioxidant systems accompany functional adaptations in cardiac mitochondria from mice fed n-3 polyunsaturated fatty acids.A modified approach to induce predictable congestive heart failure by volume overload in rats.Effects of continuous triiodothyronine infusion on the tricarboxylic acid cycle in the normal immature swine heart under extracorporeal membrane oxygenation in vivo.Dietary management of heart failure: room for improvement?Methylene blue decreases mitochondrial lysine acetylation in the diabetic heartDietary Fat is a Key Determinant in Balancing Mitochondrial Dynamics in Heart Failure: A Novel Mechanism Underlying the Obesity Paradox.Pathological hypertrophy and cardiac dysfunction are linked to aberrant endogenous unsaturated fatty acid metabolism.
P2860
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P2860
The myocardial contractile response to physiological stress improves with high saturated fat feeding in heart failure.
description
2010 nî lūn-bûn
@nan
2010 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
The myocardial contractile res ...... fat feeding in heart failure.
@ast
The myocardial contractile res ...... fat feeding in heart failure.
@en
The myocardial contractile res ...... fat feeding in heart failure.
@nl
type
label
The myocardial contractile res ...... fat feeding in heart failure.
@ast
The myocardial contractile res ...... fat feeding in heart failure.
@en
The myocardial contractile res ...... fat feeding in heart failure.
@nl
prefLabel
The myocardial contractile res ...... fat feeding in heart failure.
@ast
The myocardial contractile res ...... fat feeding in heart failure.
@en
The myocardial contractile res ...... fat feeding in heart failure.
@nl
P2093
P2860
P1476
The myocardial contractile res ...... fat feeding in heart failure.
@en
P2093
Brian D Hoit
Jessica M Berthiaume
Margaret P Chandler
Martin E Young
Molly S Bray
Salman Azam
Tracy A McElfresh
Xiaoqin Chen
P2860
P304
P356
10.1152/AJPHEART.00270.2010
P577
2010-05-28T00:00:00Z