Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytes - Test2 - elhamkhani - TriplyDB

Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytes

Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytes

http://www.wikidata.org/entity/Q34099073

about

Alterations in T-tubule and dyad structure in heart disease: challenges and opportunities for computational analyses Testosterone modulates cardiac contraction and calcium homeostasis: cellular and molecular mechanisms A critical role for Telethonin in regulating t-tubule structure and function in the mammalian heart Methods in cardiomyocyte isolation, culture, and gene transfer.Modeling effects of L-type ca(2+) current and na(+)-ca(2+) exchanger on ca(2+) trigger flux in rabbit myocytes with realistic T-tubule geometries Regulation of Cardiomyocyte T-Tubular Structure: Opportunities for Therapy.Analysis of Cav1.2 and ryanodine receptor clusters in rat ventricular myocytes Targeting cardiomyocyte Ca2+ homeostasis in heart failure.Mechanical unloading reverses transverse tubule remodelling and normalizes local Ca(2+)-induced Ca(2+)release in a rodent model of heart failure Synchrony of cardiomyocyte Ca(2+) release is controlled by T-tubule organization, SR Ca(2+) content, and ryanodine receptor Ca(2+) sensitivity.No rest for the weary: diastolic calcium homeostasis in the normal and failing myocardium.Manipulation of sarcoplasmic reticulum Ca(2+) release in heart failure through mechanical intervention.Cardiomyocyte Ca2+ dynamics: clinical perspectives.Studying dyadic structure-function relationships: a review of current modeling approaches and new insights into Ca2+ (mis)handling.Gene Transfer in Isolated Adult Cardiomyocytes.Bursting dynamics in the normal and failing hearts.Sodium accumulation in SERCA knockout-induced heart failure.Modelling cardiac calcium sparks in a three-dimensional reconstruction of a calcium release unit.Induced NCX1 overexpression attenuates pressure overload-induced pathological cardiac remodelling.Increased passive stiffness promotes diastolic dysfunction despite improved Ca2+ handling during left ventricular concentric hypertrophy.

P2860

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P2860

Control of Ca2+ release by action potential configuration in normal and failing murine cardiomyocytes

http://www.wikidata.org/entity/Q34099073

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Control of Ca2+ release by act ...... failing murine cardiomyocytes

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Control of Ca2+ release by act ...... failing murine cardiomyocytes

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Control of Ca2+ release by act ...... failing murine cardiomyocytes

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type

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Control of Ca2+ release by act ...... failing murine cardiomyocytes

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Control of Ca2+ release by act ...... failing murine cardiomyocytes

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Control of Ca2+ release by act ...... failing murine cardiomyocytes

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prefLabel

Control of Ca2+ release by act ...... failing murine cardiomyocytes

@ast

Control of Ca2+ release by act ...... failing murine cardiomyocytes

@en

Control of Ca2+ release by act ...... failing murine cardiomyocytes

@nl

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J.BPJ.2010.06.055

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Biophysical Journal

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Control of Ca2+ release by act ...... failing murine cardiomyocytes

@en

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Glenn T Lines

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Guro Five Jølle

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Halvor K Mørk

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Johan Hake

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William E Louch

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P2860

Calcium sparks: elementary events underlying excitation-contraction coupling in heart muscle

Three-dimensional electron microscopy reveals new details of membrane systems for Ca2+ signaling in the heart.

Reduction in density of transverse tubules and L-type Ca(2+) channels in canine tachycardia-induced heart failure.

Loss of T-tubules and other changes to surface topography in ventricular myocytes from failing human and rat heart.

Optical single-channel resolution imaging of the ryanodine receptor distribution in rat cardiac myocytes

There goes the neighborhood: pathological alterations in T-tubule morphology and consequences for cardiomyocyte Ca2+ handling.

An integrative model of the cardiac ventricular myocyte incorporating local control of Ca2+ release.

Cardiac Ca2+ dynamics: the roles of ryanodine receptor adaptation and sarcoplasmic reticulum load

Shape, size, and distribution of Ca(2+) release units and couplons in skeletal and cardiac muscles

Ion concentration-dependence of rat cardiac unitary L-type calcium channel conductance.

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1377-1386

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5

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Ole M. Sejersted

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46147412

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20816049

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2931738

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