SMAD2 is essential for TGF beta-mediated Th17 cell generation. - Test2 - elhamkhani - TriplyDB

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

http://www.wikidata.org/entity/Q34121444

about

MicroRNA-21 promotes Th17 differentiation and mediates experimental autoimmune encephalomyelitis TGF-β activation and function in immunity.Leucine-rich alpha 2 glycoprotein promotes Th17 differentiation and collagen-induced arthritis in mice through enhancement of TGF-β-Smad2 signaling in naïve helper T cells.The transcription factor IRF8 activates integrin-mediated TGF-β signaling and promotes neuroinflammation.FK506 binding protein 12 deficiency in endothelial and hematopoietic cells decreases regulatory T cells and causes hypertension.Signal transduction pathways and transcriptional regulation in Th17 cell differentiation Two sides of the story? Smad4 loss in pancreatic cancer versus head-and-neck cancer Phosphorylation status determines the opposing functions of Smad2/Smad3 as STAT3 cofactors in TH17 differentiation MicroRNA-144 dysregulates the transforming growth factor-β signaling cascade and contributes to the development of bronchiolitis obliterans syndrome after human lung transplantation Kaempferol Suppresses Transforming Growth Factor-β1-Induced Epithelial-to-Mesenchymal Transition and Migration of A549 Lung Cancer Cells by Inhibiting Akt1-Mediated Phosphorylation of Smad3 at Threonine-179.Cholera Toxin Promotes Th17 Cell Differentiation by Modulating Expression of Polarizing Cytokines and the Antigen-Presenting Potential of Dendritic Cells.Inhibition of TGF-β1 signaling promotes central memory T cell differentiation.Genetic controls of Th17 cell differentiation and plasticity.SMAD regulatory networks construct a balanced immune system.The balance of intestinal Foxp3+ regulatory T cells and Th17 cells and its biological significance.Yin-Yang Regulation of RORγt Protein Complex in Th17 Differentiation.A network of high-mobility group box transcription factors programs innate interleukin-17 production.IL10, TGF beta1, and IFN gamma modulate intracellular signaling pathways and cytokine production to control Toxoplasma gondii infection in BeWo trophoblast cells.MicroRNA-21 and -146b are involved in the pathogenesis of murine viral myocarditis by regulating TH-17 differentiation.Suppression of Th17 cell differentiation by misshapen/NIK-related kinase MINK1.Activation of TGF-β-induced non-Smad signaling pathways during Th17 differentiation.The kinases MEKK2 and MEKK3 regulate transforming growth factor-β-mediated helper T cell differentiation.Clinical effects of soluble interleukin-6 receptor detection on autoimmune rheumatic disease patients.The kinase PKCα selectively upregulates interleukin-17A during Th17 cell immune responses.MINK1: The missing link between ROS and its inhibition of Th17 cells.RGC-32 Promotes Th17 Cell Differentiation and Enhances Experimental Autoimmune Encephalomyelitis.Fatty acid amide hydrolase (FAAH) blockade ameliorates experimental colitis by altering microRNA expression and suppressing inflammation.Transforming growth factor-β1 promotes Treg commitment in nasal polyposis after intranasal steroid treatment.Regulatory T-Cell Augmentation or Interleukin-17 Inhibition Prevents Calcineurin Inhibitor-Induced Hypertension in Mice.Functions of Smad Transcription Factors in TGF-β1-Induced Selectin Ligand Expression on Murine CD4 Th Cells.The role of TGF-β in Th17 differentiation.MEK inhibition prevents tumour-shed transforming growth factor-β-induced T-regulatory cell augmentation in tumour milieu.Role of IL-17A in the development of colitis-associated cancer.Effector T Helper Cell Subsets in Inflammatory Bowel Diseases.

P2860

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P2860

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

http://www.wikidata.org/entity/Q34121444

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

@wuu

name

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@ast

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@en

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@nl

type

label

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@ast

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@en

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@nl

prefLabel

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@ast

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@en

SMAD2 is essential for TGF beta-mediated Th17 cell generation.

@nl

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P356

JBC.C110.156745

P1433

Journal of Biological Chemistry

P1476

SMAD2 is essential for TGF beta-mediated Th17 cell generation

@en

P2093

Elizabeth Robertson

http://www.w3.org/2001/XMLSchema#string

Nidhi Malhotra

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P2860

TGF-beta-induced Foxp3 inhibits T(H)17 cell differentiation by antagonizing RORgammat function

The biological functions of T helper 17 cell effector cytokines in inflammation

A critical role for transforming growth factor-beta but not interleukin 4 in the suppression of T helper type 1-mediated colitis by CD45RB(low) CD4+ T cells

Reciprocal developmental pathways for the generation of pathogenic effector TH17 and regulatory T cells

Transforming growth factor-beta induces development of the T(H)17 lineage

Hematopoiesis controlled by distinct TIF1gamma and Smad4 branches of the TGFbeta pathway

TGFbeta in the context of an inflammatory cytokine milieu supports de novo differentiation of IL-17-producing T cells

Cell fate decisions within the mouse organizer are governed by graded Nodal signals

Interleukin-2 signaling via STAT5 constrains T helper 17 cell generation

Molecular antagonism and plasticity of regulatory and inflammatory T cell programs

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29044-29048

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38

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285

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2010-07-23T00:00:00Z

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