Loss of Miro1-directed mitochondrial movement results in a novel murine model for neuron disease. - Test2 - elhamkhani - TriplyDB

Loss of Miro1-directed mitochondrial movement results in a novel murine model for neuron disease.

Loss of Miro1-directed mitochondrial movement results in a novel murine model for neuron disease.

http://www.wikidata.org/entity/Q34144429

about

Transporting mitochondria in neurons Impaired Autophagy and Defective Mitochondrial Function: Converging Paths on the Road to Motor Neuron Degeneration Emerging Roles of Filopodia and Dendritic Spines in Motoneuron Plasticity during Development and Disease Mitochondria-associated membranes as hubs for neurodegeneration MIRO GTPases in Mitochondrial Transport, Homeostasis and Pathology Mitotic redistribution of the mitochondrial network by Miro and Cenp-F.Structural insights into Parkin substrate lysine targeting from minimal Miro substrates Abnormalities of Mitochondrial Dynamics in Neurodegenerative Diseases.MSTO1 is a cytoplasmic pro-mitochondrial fusion protein, whose mutation induces myopathy and ataxia in humans.Tau reduction prevents Aβ-induced axonal transport deficits by blocking activation of GSK3β.Regulation of mitochondrial transport in neurons.Miro1 deficiency in amyotrophic lateral sclerosis.Role of RHOT1 on migration and proliferation of pancreatic cancer.Mitochondrial dynamic abnormalities in amyotrophic lateral sclerosis.ROS regulation of axonal mitochondrial transport is mediated by Ca2+ and JNK in Drosophila Disabling mitochondrial reprogramming in cancer C. elegans miro-1 Mutation Reduces the Amount of Mitochondria and Extends Life Span.MICU1 Serves as a Molecular Gatekeeper to Prevent In Vivo Mitochondrial Calcium Overload.Disturbed mitochondrial dynamics and neurodegenerative disorders.A PGAM5-KEAP1-Nrf2 complex is required for stress-induced mitochondrial retrograde trafficking.Miro1-mediated mitochondrial positioning shapes intracellular energy gradients required for cell migration.Functional Impairment in Miro Degradation and Mitophagy Is a Shared Feature in Familial and Sporadic Parkinson's Disease.The Interplay of Axonal Energy Homeostasis and Mitochondrial Trafficking and Anchoring.Axonal transport deficits in multiple sclerosis: spiraling into the abyss.Defining the momiome: Promiscuous information transfer by mobile mitochondria and the mitochondrial genome.An emerging role for mitochondrial dynamics in schizophrenia.Regulation of long-distance transport of mitochondria along microtubules.Polo Kinase Phosphorylates Miro to Control ER-Mitochondria Contact Sites and Mitochondrial Ca(2+) Homeostasis in Neural Stem Cell Development.Analysis of mitochondrial organization and function in the Drosophila blastoderm embryo.Loss of Dendritic Complexity Precedes Neurodegeneration in a Mouse Model with Disrupted Mitochondrial Distribution in Mature Dendrites.Developmental changes in trak-mediated mitochondrial transport in neurons.ROS Control Mitochondrial Motility through p38 and the Motor Adaptor Miro/Trak.Miro proteins coordinate microtubule- and actin-dependent mitochondrial transport and distribution.New splicing variants of mitochondrial Rho GTPase-1 (Miro1) transport peroxisomes.A role for Mitochondrial Rho GTPase 1 (MIRO1) in motility and membrane dynamics of peroxisomes.Regulation of mitochondrial dynamics in astrocytes: Mechanisms, consequences, and unknowns.Understanding Miro GTPases: Implications in the Treatment of Neurodegenerative Disorders.Dealing with Stress: Defective Metabolic Adaptation in Chronic Obstructive Pulmonary Disease Pathogenesis.MIRO-1 Determines Mitochondrial Shape Transition upon GPCR Activation and Ca2+ Stress.Miro-Working beyond Mitochondria and Microtubules.

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Loss of Miro1-directed mitochondrial movement results in a novel murine model for neuron disease.

http://www.wikidata.org/entity/Q34144429

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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Loss of Miro1-directed mitocho ...... rine model for neuron disease.

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PNAS.1402449111

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Proceedings of the National Academy of Sciences of the United States of America

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Loss of Miro1-directed mitocho ...... urine model for neuron disease

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Dane Maxfield

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David Weaver

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György Hajnóczky

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Jane Macfarlane

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Janet M Shaw

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Nathan K Smith

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Sang S Oh

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Stefan Pulst

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Tammy T Nguyen

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P2860

Syntaphilin: a syntaxin-1 clamp that controls SNARE assembly

PINK1 and Parkin target Miro for phosphorylation and degradation to arrest mitochondrial motility

A direct interaction between cytoplasmic dynein and kinesin I may coordinate motor activity

The mechanism of Ca2+ -dependent regulation of kinesin-mediated mitochondrial motility

The atypical Rho GTPases Miro-1 and Miro-2 have essential roles in mitochondrial trafficking

Atypical Rho GTPases have roles in mitochondrial homeostasis and apoptosis

GRIF-1 and OIP106, members of a novel gene family of coiled-coil domain proteins: association in vivo and in vitro with kinesin

HUMMR, a hypoxia- and HIF-1alpha-inducible protein, alters mitochondrial distribution and transport

The axonal transport of mitochondria

Mitochondrial transport in neurons: impact on synaptic homeostasis and neurodegeneration.

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111

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Cheryl Ann Palmer

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264903373

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25136135

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