Chromosome instability in human lung cancers: possible underlying mechanisms and potential consequences in the pathogenesis. - Test2 - elhamkhani - TriplyDB

Chromosome instability in human lung cancers: possible underlying mechanisms and potential consequences in the pathogenesis.

Chromosome instability in human lung cancers: possible underlying mechanisms and potential consequences in the pathogenesis.

http://www.wikidata.org/entity/Q34152941

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Chronic exposure to zinc chromate induces centrosome amplification and spindle assembly checkpoint bypass in human lung fibroblasts Cigarette smoke induces genetic instability in airway epithelial cells by suppressing FANCD2 expression Multipolar spindle pole coalescence is a major source of kinetochore mis-attachment and chromosome mis-segregation in cancer cells Hepatitis C Virus Causes Uncoupling of Mitotic Checkpoint and Chromosomal Polyploidy through the Rb Pathway gammaH2AX: A potential DNA damage response biomarker for assessing toxicological risk of tobacco products DNA damage response induced by tobacco smoke in normal human bronchial epithelial and A549 pulmonary adenocarcinoma cells assessed by laser scanning cytometry Fusion transcripts and transcribed retrotransposed loci discovered through comprehensive transcriptome analysis using Paired-End diTags (PETs)Cigarette smoke induces endoplasmic reticulum stress and the unfolded protein response in normal and malignant human lung cells Interphase cytogenetics of sputum cells for the early detection of lung carcinogenesis.The relationship between the presence of chromosomal instability and prognosis of squamous cell carcinoma of the lung: fluorescence in situ hybridization analysis of paraffin-embedded tissue from 47 Korean patients.Ladder-like amplification of the type I interferon gene cluster in the human osteosarcoma cell line MG63 MicroRNA-450a-3p represses cell proliferation and regulates embryo development by regulating Bub1 expression in mouse.Expulsion of micronuclei containing amplified genes contributes to a decrease in double minute chromosomes from malignant tumor cells.Ionizing radiation-induced γ-H2AX activity in whole blood culture and the risk of lung cancer Geminin overexpression prevents the completion of topoisomerase IIα chromosome decatenation, leading to aneuploidy in human mammary epithelial cells Elevated level of spindle checkprotein MAD2 correlates with cellular mitotic arrest, but not with aneuploidy and clinicopathological characteristics in gastric cancer.Neoplastic transformation of human bronchial cells by lead chromate particles.Chronic Exposure to Particulate Chromate Induces Premature Centrosome Separation and Centriole Disengagement in Human Lung Cells.Deregulation of cyclin E in human cells interferes with prereplication complex assembly.Homologous recombination repair protects against particulate chromate-induced chromosome instability in Chinese hamster cells.v-Src Causes Chromosome Bridges in a Caffeine-Sensitive Manner by Generating DNA Damage Hexavalent chromium-induced DNA damage and repair mechanisms.Decreased Dp71 expression is associated with gastric adenocarcinoma prognosis.Shorter telomeres and high telomerase activity correlate with a highly aggressive phenotype in breast cancer cell lines.PLK4 overexpression and its effect on centrosome regulation and chromosome stability in human gastric cancer.DNA copy number alterations in endobronchial squamous metaplastic lesions predict lung cancer.Clinical Utility of Chromosomal Aneusomy in Individuals at High Risk of Lung Cancer.pRB2/p130 target genes in non-small lung cancer cells identified by microarray analysis.Association between mitotic spindle checkpoint impairment and susceptibility to the induction of apoptosis by anti-microtubule agents in human lung cancers.Increased cell apoptosis in human lung adenocarcinoma and in vivo tumor growth inhibition by RBM10, a tumor suppressor gene.Ring chromosomes: from formation to clinical potential.UBASH3B-mediated silencing of the mitotic checkpoint: Therapeutic perspectives in cancer.MYC-containing double minutes in hematologic malignancies: evidence in favor of the episome model and exclusion of MYC as the target gene

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P2860

Chromosome instability in human lung cancers: possible underlying mechanisms and potential consequences in the pathogenesis.

http://www.wikidata.org/entity/Q34152941

description

2002 nî lūn-bûn

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2002 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2002年の論文

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Chromosome instability in huma ...... sequences in the pathogenesis.

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Akira Masuda

http://www.w3.org/2001/XMLSchema#string

Takashi Takahashi

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P2860

Stable interaction between the products of the BRCA1 and BRCA2 tumor suppressor genes in mitotic and meiotic cells

Mutations of a novel human RAD54 homologue, RAD54B, in primary cancer

Identification of Mad as a repressor of the human telomerase (hTERT) gene

Chfr defines a mitotic stress checkpoint that delays entry into metaphase

p53AIP1, a potential mediator of p53-dependent apoptosis, and its regulation by Ser-46-phosphorylated p53

Nucleophosmin/B23 is a target of CDK2/cyclin E in centrosome duplication

A role for the Adenomatous Polyposis Coli protein in chromosome segregation

The ATM-Chk2-Cdc25A checkpoint pathway guards against radioresistant DNA synthesis

The p21 Cdk-interacting protein Cip1 is a potent inhibitor of G1 cyclin-dependent kinases

Mutations of mitotic checkpoint genes in human cancers

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