Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
about
The uncoordinated-5 homolog B (UNC5B) receptor increases myocardial ischemia-reperfusion injuryAn uncoordinated-5 homolog B receptor monoclonal antibody regulates A375 melanoma cell migration.Therapeutic effects of topical netrin-4 in a corneal acute inflammatory model.Therapeutic effects of topical netrin-4 inhibits corneal neovascularization in alkali-burn ratsNetrin-1 promotes cell migration and invasion by down-regulation of BVES expression in human hepatocellular carcinoma.A novel method in preparation of acellularporcine corneal stroma tissue for lamellar keratoplastyActive Pedicle Epithelial Flap Transposition Combined with Amniotic Membrane Transplantation for Treatment of Nonhealing Corneal UlcersEfficacy of the nucleotide-binding oligomerzation domain 1 inhibitor Nodinhibit-1 on corneal alkali burns in rats.Effects of intravitreal injection of netrin-1 in retinal neovascularization of streptozotocin-induced diabetic rats.Netrin-1 is a critical autocrine/paracrine factor for osteoclast differentiationNanotechnology in corneal neovascularization therapy--a reviewPurinergic receptors in ocular inflammation.Current and Upcoming Therapies for Ocular Surface Chemical Injuries.Interactional expression of netrin-1 and its dependence receptor UNC5B in prostate carcinoma.Clinical significance of UNC5B expression in bladder cancer.Recent drug therapies for corneal neovascularization.Partial denervation of sub-basal axons persists following debridement wounds to the mouse cornea.Netrin-1 is highly expressed and required in inflammatory infiltrates in wear particle-induced osteolysis.Laminins: Roles and Utility in Wound Repair.Netrin-1 and its receptor Unc5b are novel targets for the treatment of inflammatory arthritis.Antiangiogenic effects of catalpol on rat corneal neovascularization.Comprehensive Modeling of Corneal Alkali Injury in the Rat Eye.Reduced intraepithelial corneal nerve density and sensitivity accompany desiccating stress and aging in C57BL/6 mice.The artemisinin derivative artesunate inhibits corneal neovascularization by inducing ROS-dependent apoptosis in vascular endothelial cells.Netrin-1 promotes diabetic corneal wound healing through molecular mechanisms mediated via the adenosine 2B receptor.
P2860
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P2860
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
description
2012 nî lūn-bûn
@nan
2012 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի մարտին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@ast
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@en
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@nl
type
label
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@ast
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@en
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@nl
prefLabel
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@ast
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@en
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@nl
P2093
P356
P1476
Netrin-1 simultaneously suppresses corneal inflammation and neovascularization.
@en
P2093
Fung-Rong Hu
Wei-Li Chen
Wensheng Chen
Yang-Luowa Qu
Yongxiong Chen
Yueping Zhou
P304
P356
10.1167/IOVS.11-8722
P407
P577
2012-03-09T00:00:00Z