Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton. - Test2 - elhamkhani - TriplyDB

Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton.

Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton.

http://www.wikidata.org/entity/Q34165677

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Protein kinase C-delta regulates HIV-1 replication at an early post-entry step in macrophages Gelsolin activity controls efficient early HIV-1 infection SAMHD1 restricts HIV-1 reverse transcription in quiescent CD4(+) T-cells HIV restriction in quiescent CD4⁺ T cells The immunological synapse: the gateway to the HIV reservoir Impact of antiretroviral therapy (ART) timing on chronic immune activation/inflammation and end-organ damage How HIV takes advantage of the cytoskeleton in entry and replication Human immunodeficiency virus (HIV)-1 integration sites in viral latency Large Variations in HIV-1 Viral Load Explained by Shifting-Mosaic Metapopulation Dynamics Bioinformatic analysis of HIV-1 entry and pathogenesis.Understanding HIV latency: the road to an HIV cure The M. tuberculosis HAD phosphatase (Rv3042c) interacts with host proteins and is inhibited by Clofazimine Phosphorylation of CDK9 at Ser175 enhances HIV transcription and is a marker of activated P-TEFb in CD4(+) T lymphocytes An in-depth comparison of latent HIV-1 reactivation in multiple cell model systems and resting CD4+ T cells from aviremic patients Directly infected resting CD4+T cells can produce HIV Gag without spreading infection in a model of HIV latency Quantification of HIV-1 latency reversal in resting CD4+ T cells from patients on suppressive antiretroviral therapy Plasma IP-10 Concentrations Correlate Positively with Viraemia and Inversely with CD4 Counts in Untreated HIV Infection The CCR5-antagonist Maraviroc reverses HIV-1 latency in vitro alone or in combination with the PKC-agonist Bryostatin-1 HIV persistence: chemokines and their signalling pathways.Discovery of Novel Small-Molecule Inhibitors of LIM Domain Kinase for Inhibiting HIV-1 Entinostat is a histone deacetylase inhibitor selective for class 1 histone deacetylases and activates HIV production from latently infected primary T cells.Quantitative phosphoproteomics of CXCL12 (SDF-1) signaling.Challenges in HIV Vaccine Research for Treatment and Prevention.Hyperthermia stimulates HIV-1 replication Immune activation and HIV persistence: implications for curative approaches to HIV infection.Cell-to-cell transmission can overcome multiple donor and target cell barriers imposed on cell-free HIV.Finding a cure for HIV: will it ever be achievable?Towards an HIV cure: a global scientific strategy Dendritic cell-induced activation of latent HIV-1 provirus in actively proliferating primary T lymphocytes Myeloid dendritic cells induce HIV-1 latency in non-proliferating CD4+ T cells.Distinct requirements for HIV-cell fusion and HIV-mediated cell-cell fusion.Viral exploitation of actin: force-generation and scaffolding functions in viral infection.Quantitative analysis of the processes and signaling events involved in early HIV-1 infection of T cells.Eradicating HIV-1 infection: seeking to clear a persistent pathogen.Sulfonation pathway inhibitors block reactivation of latent HIV-1.Spinoculation triggers dynamic actin and cofilin activity that facilitates HIV-1 infection of transformed and resting CD4 T cells.Expression and reactivation of HIV in a chemokine induced model of HIV latency in primary resting CD4+ T cells.TFH cells accumulate in mucosal tissues of humanized-DRAG mice and are highly permissive to HIV-1 Direct evidence of nuclear Argonaute distribution during transcriptional silencing links the actin cytoskeleton to nuclear RNAi machinery in human cells Cannabinoid receptor 2-mediated attenuation of CXCR4-tropic HIV infection in primary CD4+ T cells

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Establishment of HIV-1 latency in resting CD4+ T cells depends on chemokine-induced changes in the actin cytoskeleton.

http://www.wikidata.org/entity/Q34165677

description

2010 nî lūn-bûn

@nan

2010 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի սեպտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

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name

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@ast

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

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Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@nl

type

label

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

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Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@en

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@nl

prefLabel

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@ast

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@en

Establishment of HIV-1 latency ...... ges in the actin cytoskeleton.

@nl

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PNAS.1002894107

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Proceedings of the National Academy of Sciences of the United States of America

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Establishment of HIV-1 latency ...... nges in the actin cytoskeleton

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Ajantha Solomon

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Andrew N Harman

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Anthony Jaworowski

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Elias K Haddad

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Georgina Sallmann

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Kate L Jones

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Rafick-Pierre Sekaly

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Suha Saleh

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Suberoylanilide hydroxamic acid reactivates HIV from latently infected cells

The intersectin 2 adaptor links Wiskott Aldrich Syndrome protein (WASp)-mediated actin polymerization to T cell antigen receptor endocytosis

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10.1073/PNAS.1002894107

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39

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107

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Sharon R. Lewin

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Vanessa A Evans

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2010-09-13T00:00:00Z

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