Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages. - Test2 - elhamkhani - TriplyDB

Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

http://www.wikidata.org/entity/Q34185906

about

Antioxidant and Vasodilator Activity of Ugni molinae Turcz. (Murtilla) and Its Modulatory Mechanism in Hypotensive Response Topical Formulation Containing Naringenin: Efficacy against Ultraviolet B Irradiation-Induced Skin Inflammation and Oxidative Stress in Mice.Anti-inflammatory activity of Barleria lupulina: Identification of active compounds that activate the Nrf2 cell defense pathway, organize cortical actin, reduce stress fibers, and improve cell junctions in microvascular endothelial cells.Activation of NRF2 Signaling in HEK293 Cells by a First-in-Class Direct KEAP1-NRF2 Inhibitor.Taurine protects against As2O3-induced autophagy in livers of rat offsprings through PPARγ pathway Potential molecular characteristics in situ in response to repetitive UVB irradiation.Ginsenoside Rg1 attenuates ultraviolet B-induced glucocortisides resistance in keratinocytes via Nrf2/HDAC2 signalling.Antioxidants for Healthy Skin: The Emerging Role of Aryl Hydrocarbon Receptors and Nuclear Factor-Erythroid 2-Related Factor-2.Different roles of ROS and Nrf2 in Cr(VI)-induced inflammatory responses in normal and Cr(VI)-transformed cells Pterostilbene protects against UVB-induced photo-damage through a phosphatidylinositol-3-kinase-dependent Nrf2/ARE pathway in human keratinocytes.The triterpenoid corosolic acid blocks transformation and epigenetically reactivates Nrf2 in TRAMP-C1 prostate cells.Keap1-Nrf2 pathway: A promising target towards lung cancer prevention and therapeutics.Salidroside prevents skin carcinogenesis induced by DMBA/TPA in a mouse model through suppression of inflammation and promotion of apoptosis.Curcumin ameliorates asthmatic airway inflammation by activating nuclear factor-E2-related factor 2/haem oxygenase (HO)-1 signalling pathway.Topical formulation containing hesperidin methyl chalcone inhibits skin oxidative stress and inflammation induced by ultraviolet B irradiation.

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P2860

Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

http://www.wikidata.org/entity/Q34185906

description

2014 nî lūn-bûn

@nan

2014 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի օգոստոսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Cell & Bioscience

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Nrf2 null enhances UVB-induced skin inflammation and extracellular matrix damages.

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Ah-Ng Tony Kong

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Allan H Conney

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Limin Shu

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Mou-Tuan Huang

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Tin Oo Khor

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Yaoping Lu

http://www.w3.org/2001/XMLSchema#string

Yue Liu

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P2860

The in vivo gene expression signature of oxidative stress

Oxidative stress, inflammation, and cancer: how are they linked?

MMP-9 supplied by bone marrow-derived cells contributes to skin carcinogenesis.

Biomarkers of extracellular matrix metabolism (MMP-9 and TIMP-1) and risk of stroke, myocardial infarction, and cause-specific mortality: cohort study.

Nrf2 establishes a glutathione-mediated gradient of UVB cytoprotection in the epidermis.

Disruption of Nrf2 enhances the upregulation of nuclear factor-kappaB activity, tumor necrosis factor-α, and matrix metalloproteinase-9 after spinal cord injury in mice.

Extracellular matrix degradation and remodeling in development and disease

Pathophysiology of premature skin aging induced by ultraviolet light.

Photochemoprevention of ultraviolet B signaling and photocarcinogenesis.

The epidemiology of skin cancer.

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39

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scholarly article

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10.1186/2045-3701-4-39

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4

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Anne Yuqing Yang

Constance Lay-Lay Saw

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2014-08-05T00:00:00Z

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265598856

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1046058499

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25228981

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extracellular matrix

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4164960

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