Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. - Test2 - elhamkhani - TriplyDB

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

http://www.wikidata.org/entity/Q34193811

about

Cancer Metabolism: A Modeling Perspective Dietary and pharmacological modification of the insulin/IGF-1 system: exploiting the full repertoire against cancer Tumor Metabolism, the Ketogenic Diet and β-Hydroxybutyrate: Novel Approaches to Adjuvant Brain Tumor Therapy MUC1 facilitates metabolomic reprogramming in triple-negative breast cancer.Tumor Cells Growth and Survival Time with the Ketogenic Diet in Animal Models: A Systematic Review Reprogramming induced by isoliquiritigenin diminishes melanoma cachexia through mTORC2-AKT-GSK3β signaling Beneficial effects of ketogenic diets for cancer patients: a realist review with focus on evidence and confirmation.Famine versus feast: understanding the metabolism of tumors in vivo.Non-Toxic Metabolic Management of Metastatic Cancer in VM Mice: Novel Combination of Ketogenic Diet, Ketone Supplementation, and Hyperbaric Oxygen Therapy.The Ketogenic Diet Does Not Affect Growth of Hedgehog Pathway Medulloblastoma in Mice.Anti-Tumor Effects of Ketogenic Diets in Mice: A Meta-Analysis.The Plasma Membrane Calcium Pump in Pancreatic Cancer Cells Exhibiting the Warburg Effect Relies on Glycolytic ATP.Leucine-rich diet alters the (1)H-NMR based metabolomic profile without changing the Walker-256 tumour mass in rats MUC16-mediated activation of mTOR and c-Myc reprograms pancreatic cancer metabolism.Silibinin-mediated metabolic reprogramming attenuates pancreatic cancer-induced cachexia and tumor growth Pancreatic cancer cell-derived IGFBP-3 contributes to muscle wasting The 2-oxoglutarate analog 3-oxoglutarate decreases normoxic hypoxia-inducible factor-1α in cancer cells, induces cell death, and reduces tumor xenograft growth.Meta-analysis of clinical metabolic profiling studies in cancer: challenges and opportunities.Nuclear Magnetic Resonance metabolomics reveals an excretory metabolic signature of renal cell carcinoma.Premorbid obesity, but not nutrition, prevents critical illness-induced muscle wasting and weakness.The action of β-hydroxybutyrate on the growth, metabolism and global histone H3 acetylation of spontaneous mouse mammary tumours: evidence of a β-hydroxybutyrate paradox T cell metabolic reprogramming and plasticity.GPR55 receptor antagonist decreases glycolytic activity in PANC-1 pancreatic cancer cell line and tumor xenografts.Detection of Pancreatic Cancer-Induced Cachexia Using a Fluorescent Myoblast Reporter System and Analysis of Metabolite Abundance.Multi-dimensional Roles of Ketone Bodies in Fuel Metabolism, Signaling, and Therapeutics.Regulation of hepatic stellate cell proliferation and activation by glutamine metabolism Inactivation of HMGCL promotes proliferation and metastasis of nasopharyngeal carcinoma by suppressing oxidative stress.Fenofibrate Induces Ketone Body Production in Melanoma and Glioblastoma Cells.Press-pulse: a novel therapeutic strategy for the metabolic management of cancer.Introduction: What to Eat When You Can't Eat.Metabolomic profile in pancreatic cancer patients: a consensus-based approach to identify highly discriminating metabolites.A ketogenic diet supplemented with medium-chain triglycerides enhances the anti-tumor and anti-angiogenic efficacy of chemotherapy on neuroblastoma xenografts in a CD1-nu mouse model.Correction: Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.The influence of ketogenic therapy on the 5 R's of radiobiology.Glucose Limitation Alters Glutamine Metabolism in MUC1-Overexpressing Pancreatic Cancer Cells.Hypoxia-Mediated In Vivo Tumor Glucose Uptake Measurement and Analysis.Low ketolytic enzyme levels in tumors predict ketogenic diet responses in cancer cell lines in vitro and in vivo.MUC1 and HIF-1alpha Signaling Crosstalk Induces Anabolic Glucose Metabolism to Impart Gemcitabine Resistance to Pancreatic Cancer.Glutaminolysis and carcinogenesis of oral squamous cell carcinoma.De Novo Lipid Synthesis Facilitates Gemcitabine Resistance through Endoplasmic Reticulum Stress in Pancreatic Cancer.

P2860

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Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

http://www.wikidata.org/entity/Q34193811

description

2014 nî lūn-bûn

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2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@ast

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@en

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@nl

type

label

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@ast

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@en

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@nl

prefLabel

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@ast

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@en

Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

@nl

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Cancer & Metabolism

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Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia.

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Fang Yu

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Iraklis I Pipinos

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Kamiya Mehla

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Nina V Chaika

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Pankaj K Singh

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Prakash Radhakrishnan

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Robert Powers

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Surendra K Shukla

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Teklab Gebregiworgis

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Venugopal Gunda

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P2860

Is there a role for carbohydrate restriction in the treatment and prevention of cancer?

Hallmarks of Cancer: The Next Generation

The Ketogenic Diet: One Decade Later

Identification of c-MYC as a target of the APC pathway

Cytoscape: a software environment for integrated models of biomolecular interaction networks

HMDB 3.0--The Human Metabolome Database in 2013

Understanding the Warburg effect: the metabolic requirements of cell proliferation

Metscape: a Cytoscape plug-in for visualizing and interpreting metabolomic data in the context of human metabolic networks

Brick by brick: metabolism and tumor cell growth

Cancer cachexia: mediators, signaling, and metabolic pathways

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