An 86-probe-set gene-expression signature predicts survival in cytogenetically normal acute myeloid leukemia
about
A scalable method for molecular network reconstruction identifies properties of targets and mutations in acute myeloid leukemiaMusashi-2 regulates normal hematopoiesis and promotes aggressive myeloid leukemiaAcute myeloid leukemia: 2012 update on diagnosis, risk stratification, and management.A framework for regularized non-negative matrix factorization, with application to the analysis of gene expression dataPre-clinical drug prioritization via prognosis-guided genetic interaction networksAssociation of a cytarabine chemosensitivity related gene expression signature with survival in cytogenetically normal acute myeloid leukemia.Lis1 regulates asymmetric division in hematopoietic stem cells and in leukemiaInvestigating the prediction ability of survival models based on both clinical and omics data: two case studies.Deviance residuals-based sparse PLS and sparse kernel PLS regression for censored data.Data-Driven Phenotypic Dissection of AML Reveals Progenitor-like Cells that Correlate with Prognosis.PrognoScan: a new database for meta-analysis of the prognostic value of genes.Quantitative comparison of microarray experiments with published leukemia related gene expression signatures.The use of knockout mice reveals a synergistic role of the Vav1 and Rasgrf2 gene deficiencies in lymphomagenesis and metastasisGenetic tests to evaluate prognosis and predict therapeutic response in acute myeloid leukemia.A differentiation-based phylogeny of cancer subtypes.Higher HOPX expression is associated with distinct clinical and biological features and predicts poor prognosis in de novo acute myeloid leukemiaFatty acid-binding protein FABP4 mechanistically links obesity with aggressive AML by enhancing aberrant DNA methylation in AML cells.Association of a leukemic stem cell gene expression signature with clinical outcomes in acute myeloid leukemia.Spectral clustering using Nyström approximation for the accurate identification of cancer molecular subtypesIdentifying the gene signatures from gene-pathway bipartite network guarantees the robust model performance on predicting the cancer prognosis.A nucleolin-DNMT1 regulatory axis in acute myeloid leukemogenesis.Transformation resistance in a premature aging disorder identifies a tumor-protective function of BRD4.Molecular signatures in acute myeloid leukemia.Identification of therapeutic targets for quiescent, chemotherapy-resistant human leukemia stem cells.Engagement of SIRPα inhibits growth and induces programmed cell death in acute myeloid leukemia cells.Integrative genomic analyses of a novel cytokine, interleukin-34 and its potential role in cancer prediction.Added predictive value of omics data: specific issues related to validation illustrated by two case studies.Sparse expression bases in cancer reveal tumor drivers.Prognostic and biologic significance of DNMT3B expression in older patients with cytogenetically normal primary acute myeloid leukemiaHierarchy in gene expression is predictive of risk, progression, and outcome in adult acute myeloid leukemiaSystematic computation with functional gene-sets among leukemic and hematopoietic stem cells reveals a favorable prognostic signature for acute myeloid leukemiaAn integrated approach to dissecting oncogene addiction implicates a Myb-coordinated self-renewal program as essential for leukemia maintenance.Sox4 cooperates with PU.1 haploinsufficiency in murine myeloid leukemia.PU.1 is essential for MLL leukemia partially via crosstalk with the MEIS/HOX pathway.Identification of the Adapter Molecule MTSS1 as a Potential Oncogene-Specific Tumor Suppressor in Acute Myeloid LeukemiaFHL2 interacts with CALM and is highly expressed in acute erythroid leukemia.High expression of inositol 1,4,5-trisphosphate receptor, type 2 (ITPR2) as a novel biomarker for worse prognosis in cytogenetically normal acute myeloid leukemia.Mitogen-activated protein kinase binding protein 1 (MAPKBP1) is an unfavorable prognostic biomarker in cytogenetically normal acute myeloid leukemia.Functional analysis of the NUP98-CCDC28A fusion protein.The aryl hydrocarbon receptor pathway: a key component of the microRNA-mediated AML signalisome.
P2860
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P2860
An 86-probe-set gene-expression signature predicts survival in cytogenetically normal acute myeloid leukemia
description
2008 nî lūn-bûn
@nan
2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@ast
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@en
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@nl
type
label
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@ast
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@en
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@nl
prefLabel
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@ast
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@en
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@nl
P2093
P2860
P50
P1433
P1476
An 86-probe-set gene-expressio ...... normal acute myeloid leukemia
@en
P2093
Achim Heinecke
Bernhard Wörmann
Christian Buske
German AML Cooperative Group
Jan Braess
Karsten Spiekermann
Manuela Hummel
Maria-Cristina Sauerland
Michael Radmacher
Peter Paschka
P2860
P304
P356
10.1182/BLOOD-2008-02-134411
P407
P50
P577
2008-08-20T00:00:00Z