Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration. - Test2 - elhamkhani - TriplyDB

Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.

Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.

http://www.wikidata.org/entity/Q34220388

about

The role of estrogen and androgen receptors in bone health and disease Dysapoptosis of osteoblasts and osteocytes increases cancellous bone formation but exaggerates cortical porosity with age.Parathyroid hormone's enhancement of bones' osteogenic response to loading is affected by ageing in a dose- and time-dependent manner.Aging diminishes lamellar and woven bone formation induced by tibial compression in adult C57BL/6 SRT2104 extends survival of male mice on a standard diet and preserves bone and muscle mass Estrogens and Androgens in Skeletal Physiology and Pathophysiology.The roles of lipid oxidation products and receptor activator of nuclear factor-κB signaling in atherosclerotic calcification Unraveling the role of FoxOs in bone--insights from mouse models.Teriparatide Therapy as an Adjuvant for Tissue Engineering and Integration of Biomaterials.Effects of age on parathyroid hormone signaling in human marrow stromal cells.Glucocorticoid dose determines osteocyte cell fate.Age-related decline in osteoblastogenesis and 1α-hydroxylase/CYP27B1 in human mesenchymal stem cells: stimulation by parathyroid hormone.Glucocorticoids and tumor necrosis factor α increase oxidative stress and suppress Wnt protein signaling in osteoblasts Connexins and pannexins in the skeleton: gap junctions, hemichannels and more Bone mechanotransduction may require augmentation in order to strengthen the senescent skeleton.Arterial calcification and bone physiology: role of the bone-vascular axis.Beyond gap junctions: Connexin43 and bone cell signaling Estrogen receptor-α signaling in osteoblast progenitors stimulates cortical bone accrual Role of paraoxonase-1 in bone anabolic effects of parathyroid hormone in hyperlipidemic mice Non-nuclear-initiated actions of the estrogen receptor protect cortical bone mass.Aging mechanisms in bone.Nutrition in Cardioskeletal Health.Suppression of autophagy in osteocytes mimics skeletal aging.Connexin 43 channels protect osteocytes against oxidative stress-induced cell death.Reactive Oxygen Species Differentially Regulate Bone Turnover in an Age-Specific Manner in Catalase Transgenic Female Mice.Basic biology of skeletal aging: role of stress response pathways.Roles of parathyroid hormone (PTH) receptor and reactive oxygen species in hyperlipidemia-induced PTH resistance in preosteoblasts.Bone marrow mesenchymal stem cells and TGF-β signaling in bone remodeling.The Effects of Aging and Sex Steroid Deficiency on the Murine Skeleton Are Independent and Mechanistically Distinct.FoxOs, Wnts and oxidative stress-induced bone loss: new players in the periodontitis arena?The regulation of valvular and vascular sclerosis by osteogenic morphogens Signaling pathways affecting skeletal health.Bone cell senescence: mechanisms and perspectives.Parathyroid Hormone-Related Protein Protects Osteoblastic Cells From Oxidative Stress by Activation of MKP1 Phosphatase.Intermittent parathyroid hormone administration attenuates endothelial dysfunction in old rats.Black bear parathyroid hormone has greater anabolic effects on trabecular bone in dystrophin-deficient mice than in wild type mice Reduced serum IGF-1 associated with hepatic osteodystrophy is a main determinant of low cortical but not trabecular bone mass.Inhibition of apoptosis signal-regulating kinase 1 enhances endochondral bone formation by increasing chondrocyte survival.Old age causes de novo intracortical bone remodeling and porosity in mice.Dalbergioidin (DAL) protects MC3T3-E1 osteoblastic cells against H2O2-induced cell damage through activation of the PI3K/AKT/SMAD1 pathway.

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P2860

Decreased oxidative stress and greater bone anabolism in the aged, when compared to the young, murine skeleton with parathyroid hormone administration.

http://www.wikidata.org/entity/Q34220388

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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Decreased oxidative stress and ...... hyroid hormone administration.

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Decreased oxidative stress and ...... hyroid hormone administration.

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Decreased oxidative stress and ...... hyroid hormone administration.

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Decreased oxidative stress and ...... hyroid hormone administration.

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Decreased oxidative stress and ...... hyroid hormone administration.

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Elena Ambrogini

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Li Han

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Maria Almeida

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Paula K Roberson

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Robert L Jilka

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Robert S Weinstein

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Starros C Manolagas

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P2860

Birth and Death of Bone Cells: Basic Regulatory Mechanisms and Implications for the Pathogenesis and Treatment of Osteoporosis 1

Identification of c-MYC as a target of the APC pathway

FGF-20 and DKK1 are transcriptional targets of beta-catenin and FGF-20 is implicated in cancer and development

Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

Control of bone mass and remodeling by PTH receptor signaling in osteocytes

Chronic elevation of parathyroid hormone in mice reduces expression of sclerostin by osteocytes: a novel mechanism for hormonal control of osteoblastogenesis

Attenuation of the self-renewal of transit-amplifying osteoblast progenitors in the murine bone marrow by 17 beta-estradiol

From estrogen-centric to aging and oxidative stress: a revised perspective of the pathogenesis of osteoporosis

The estrogen receptor-alpha in osteoclasts mediates the protective effects of estrogens on cancellous but not cortical bone

Canonical Wnt signaling in differentiated osteoblasts controls osteoclast differentiation

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