IL-33 is a crucial amplifier of innate rather than acquired immunity - Test2 - elhamkhani - TriplyDB

IL-33 is a crucial amplifier of innate rather than acquired immunity

IL-33 is a crucial amplifier of innate rather than acquired immunity

http://www.wikidata.org/entity/Q34276672

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F-box protein FBXL19-mediated ubiquitination and degradation of the receptor for IL-33 limits pulmonary inflammation IL-33 is processed into mature bioactive forms by neutrophil elastase and cathepsin G A Player and Coordinator: The Versatile Roles of Eosinophils in the Immune System Cytokine-Induced Modulation of Colorectal Cancer Interleukin-33 and Mast Cells Bridge Innate and Adaptive Immunity: From the Allergologist's Perspective Re-defining the unique roles for eosinophils in allergic respiratory inflammation Innate immunity in the central nervous system Insights into the initiation of type 2 immune responses Phospholipase cε, an effector of ras and rap small GTPases, is required for airway inflammatory response in a mouse model of bronchial asthma.Insights into Group 2 Innate Lymphoid Cells in Human Airway Disease Glyphosate-rich air samples induce IL-33, TSLP and generate IL-13 dependent airway inflammation Paracrine IL-33 stimulation enhances lipopolysaccharide-mediated macrophage activation IL-33 induces nuocytes and modulates liver injury in viral hepatitis.ST2 and IL-33 in pregnancy and pre-eclampsia Dealing with Danger in the CNS: The Response of the Immune System to Injury.T cell factor 1 is required for group 2 innate lymphoid cell generation.Interleukin-33 regulates intestinal inflammation by modulating macrophages in inflammatory bowel disease.Replicating viral vector platform exploits alarmin signals for potent CD8+ T cell-mediated tumour immunotherapy.Experimental atopic dermatitis depends on IL-33R signaling via MyD88 in dendritic cells.IL-33 targeting attenuates intestinal mucositis and enhances effective tumor chemotherapy in mice.IL-33 mediates multi-walled carbon nanotube (MWCNT)-induced airway hyper-reactivity via the mobilization of innate helper cells in the lung.Unique temporal and spatial expression patterns of IL-33 in ovaries during ovulation and estrous cycle are associated with ovarian tissue homeostasis Whole-genome microarray analysis and functional characterization reveal distinct gene expression profiles and patterns in two mouse models of ileal inflammation.The interleukin-1 family: back to the future Deletion of IL-33R (ST2) abrogates resistance to EAE in BALB/C mice by enhancing polarization of APC to inflammatory phenotype Central domain of IL-33 is cleaved by mast cell proteases for potent activation of group-2 innate lymphoid cells.IL-33 promotes ST2-dependent lung fibrosis by the induction of alternatively activated macrophages and innate lymphoid cells in mice Associations between genetic polymorphisms in IL-33, IL1R1 and risk for inflammatory bowel disease.IL-33 and Airway Inflammation.IL-33, but not IL-25, is crucial for the development of house dust mite antigen-induced allergic rhinitis.Transcriptome analysis of the hormone-sensing cells in mammary epithelial reveals dynamic changes in early pregnancy.Global secretome characterization of A549 human alveolar epithelial carcinoma cells during Mycoplasma pneumoniae infection.Lactobacillus paracasei CBA L74 metabolic products and fermented milk for infant formula have anti-inflammatory activity on dendritic cells in vitro and protective effects against colitis and an enteric pathogen in vivo The alarmin concept applied to human renal transplantation: evidence for a differential implication of HMGB1 and IL-33 Dusp5 negatively regulates IL-33-mediated eosinophil survival and function The alarmin IL-33 promotes regulatory T-cell function in the intestine IL-33 is required for disposal of unnecessary cells during ovarian atresia through regulation of autophagy and macrophage migration Novel cytokine signaling pathways in inflammatory bowel disease: insight into the dichotomous functions of IL-33 during chronic intestinal inflammation IL-33 expands suppressive CD11b+ Gr-1(int) and regulatory T cells, including ST2L+ Foxp3+ cells, and mediates regulatory T cell-dependent promotion of cardiac allograft survival.An IL-9 fate reporter demonstrates the induction of an innate IL-9 response in lung inflammation

P2860

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P2860

IL-33 is a crucial amplifier of innate rather than acquired immunity

http://www.wikidata.org/entity/Q34276672

description

2010 nî lūn-bûn

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2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

@zh-hk

2010年論文

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2010年論文

@zh-tw

2010年论文

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name

IL-33 is a crucial amplifier of innate rather than acquired immunity

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IL-33 is a crucial amplifier of innate rather than acquired immunity

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IL-33 is a crucial amplifier of innate rather than acquired immunity

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type

label

IL-33 is a crucial amplifier of innate rather than acquired immunity

@ast

IL-33 is a crucial amplifier of innate rather than acquired immunity

@en

IL-33 is a crucial amplifier of innate rather than acquired immunity

@nl

prefLabel

IL-33 is a crucial amplifier of innate rather than acquired immunity

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IL-33 is a crucial amplifier of innate rather than acquired immunity

@en

IL-33 is a crucial amplifier of innate rather than acquired immunity

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PNAS.1003059107

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Proceedings of the National Academy of Sciences of the United States of America

P1476

IL-33 is a crucial amplifier of innate rather than acquired immunity

@en

P2093

Akina Ishii

http://www.w3.org/2001/XMLSchema#string

Aya Nambu

http://www.w3.org/2001/XMLSchema#string

Hideaki Morita

http://www.w3.org/2001/XMLSchema#string

Hiroshi Kiyonari

http://www.w3.org/2001/XMLSchema#string

Katsuko Sudo

http://www.w3.org/2001/XMLSchema#string

Keisuke Oboki

http://www.w3.org/2001/XMLSchema#string

Ken Arae

http://www.w3.org/2001/XMLSchema#string

Kenji Matsumoto

http://www.w3.org/2001/XMLSchema#string

Ko Okumura

http://www.w3.org/2001/XMLSchema#string

Naoki Kajiwara

http://www.w3.org/2001/XMLSchema#string

P2860

IL-33, an interleukin-1-like cytokine that signals via the IL-1 receptor-related protein ST2 and induces T helper type 2-associated cytokines

Allergenicity resulting from functional mimicry of a Toll-like receptor complex protein

The IL-1-like cytokine IL-33 is inactivated after maturation by caspase-1

Interleukin-33 enhances adhesion, CD11b expression and survival in human eosinophils

IL-33 reduces the development of atherosclerosis

Immunological and inflammatory functions of the interleukin-1 family

House dust mite allergen induces asthma via Toll-like receptor 4 triggering of airway structural cells.

Shift towards pro-inflammatory intestinal bacteria aggravates acute murine colitis via Toll-like receptors 2 and 4.

Occupational asthma after inhalation of dust of the proteolytic enzyme, papain

Isolation of a novel interleukin-1-inducible nuclear protein bearing ankyrin-repeat motifs.

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18581-18586

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10.1073/PNAS.1003059107

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43

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107

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47395668

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20937871

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2972966

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