Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
about
Detrimental or beneficial: the role of TRPM2 in ischemia/reperfusion injuryFunctional implications of axon initial segment cytoskeletal disruption in strokeA molecular signature in the pannexin1 intracellular loop confers channel activation by the α1 adrenoreceptor in smooth muscle cells.Regulation of the pannexin-1 promoter in the rat epididymis.Conantokin-G attenuates detrimental effects of NMDAR hyperactivity in an ischemic rat model of stroke.24S-hydroxycholesterol and 25-hydroxycholesterol differentially impact hippocampal neuronal survival following oxygen-glucose deprivation.Endogenous 24S-hydroxycholesterol modulates NMDAR-mediated function in hippocampal slices.Molecular pathways of pannexin1-mediated neurotoxicity.Pannexin1 as a novel cerebral target in pathogenesis of hepatic encephalopathy.Pannexin channels and ischaemia.Danger- and pathogen-associated molecular patterns recognition by pattern-recognition receptors and ion channels of the transient receptor potential family triggers the inflammasome activation in immune cells and sensory neurons.Non-NMDAR neuronal Ca(2+)-permeable channels in delayed neuronal death and as potential therapeutic targets for ischemic brain damage.Non-specific inhibition of ischemia- and acidosis-induced intracellular calcium elevations and membrane currents by α-phenyl-N-tert-butylnitrone, butylated hydroxytoluene and trolox.Dysfunction of the neurovascular unit in ischemic stroke and neurodegenerative diseases: An aging effect.Interactions of Pannexin1 channels with purinergic and NMDA receptor channels.Oxygen and Glucose Deprivation Induces Bergmann Glia Membrane Depolarization and Ca2+ Rises Mainly Mediated by K+ and ATP Increases in the Extracellular Space.Tinnitus: Prospects for Pharmacological Interventions With a Seesaw Model.Protecting Oligodendrocytes by Targeting Non-Glutamate Receptors as a New Therapeutic Strategy for Ischemic Stroke.Treatment with Isorhamnetin Protects the Brain Against Ischemic Injury in Mice.Differential gene expression profiles between two subtypes of ischemic stroke with blood stasis syndromes.Pannexin 1 sustains the electrophysiological responsiveness of retinal ganglion cells.Glutamate protects against Ca(2+) paradox-induced injury and inhibits calpain activity in isolated rat hearts.The two faces of pannexins: new roles in inflammation and repair.
P2860
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P2860
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
description
2012 nî lūn-bûn
@nan
2012 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@ast
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@en
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@nl
type
label
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@ast
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@en
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@nl
prefLabel
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@ast
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@en
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@nl
P2093
P2860
P356
P1476
Ionotropic receptors and ion channels in ischemic neuronal death and dysfunction.
@en
P2093
Jennifer Bialecki
Nicholas L Weilinger
Peter L Tang
Roger J Thompson
Sarup S Sridharan
Valentyna Maslieieva
P2860
P2888
P356
10.1038/APS.2012.95
P577
2012-08-06T00:00:00Z
P5875
P6179
1005487478