Corin mutation R539C from hypertensive patients impairs zymogen activation and generates an inactive alternative ectodomain fragment.
about
Atrial natriuretic peptide in cardiovascular biology and disease (NPPA)Epithelial sodium transport and its control by aldosterone: the story of our internal environment revisitedPCSK6-mediated corin activation is essential for normal blood pressureThe Hypertension Pandemic: An Evolutionary Perspective.Corin mutations K317E and S472G from preeclamptic patients alter zymogen activation and cell surface targeting. [Corrected]N-glycosylation is required for matriptase-2 autoactivation and ectodomain sheddingCorin in natriuretic peptide processing and hypertensionDistinct roles of N-glycosylation at different sites of corin in cell membrane targeting and ectodomain shedding.A corin variant identified in hypertensive patients that alters cytoplasmic tail and reduces cell surface expression and activityANP-induced signaling cascade and its implications in renal pathophysiologyA novel cytoplasmic tail motif regulates mouse corin expression on the cell surface.The Transmembrane Serine Protease HAT-like 4 Is Important for Epidermal Barrier Function to Prevent Body Fluid Loss.Role of corin in the regulation of blood pressure.Localization of corin and atrial natriuretic peptide expression in human renal segments.Short-term High-salt Diet Increases Corin Level to Regulate the Salt-water Balance in Humans and Rodents.Functional analysis of corin protein domains required for PCSK6-mediated activation.Pharmacogenomics of the Natriuretic Peptide System in Heart Failure.Role of the protease corin in chondrogenic differentiation of human bone marrow-derived mesenchymal stem cells.Identification and functional analysis of CORIN variants in hypertensive patients.Serum Corin Level Is Associated With Subsequent Decline in Renal Function in Patients With Suspected Coronary Artery Disease.N-glycosylation in the protease domain of trypsin-like serine proteases mediates calnexin-assisted protein folding.
P2860
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P2860
Corin mutation R539C from hypertensive patients impairs zymogen activation and generates an inactive alternative ectodomain fragment.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@ast
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@en
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@nl
type
label
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@ast
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@en
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@nl
prefLabel
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@ast
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@en
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@nl
P2093
P2860
P356
P1476
Corin mutation R539C from hype ...... ternative ectodomain fragment.
@en
P2093
Chaodong Fang
Jianping Zhou
Jianzhong Shen
Koichi Fukuda
Ningzheng Dong
Tiantian Zhou
Yizhi Jiang
P2860
P304
P356
10.1074/JBC.M112.411512
P407
P577
2013-01-31T00:00:00Z