Angiotensin II-induced mitochondrial Nox4 is a major endogenous source of oxidative stress in kidney tubular cells
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Reciprocal regulation of TGF-β and reactive oxygen species: A perverse cycle for fibrosisThe balance of powers: Redox regulation of fibrogenic pathways in kidney injuryNADPH oxidases in lung health and diseaseTreating the host response to emerging virus diseases: lessons learned from sepsis, pneumonia, influenza and Ebola.The protective effects of beta-casomorphin-7 against glucose -induced renal oxidative stress in vivo and vitroNuclear expression of renin-angiotensin system components in NRK-52E renal epithelial cells.Stress reaction in outer segments of photoreceptors after blue light irradiationNoninvasive in vivo imaging of diabetes-induced renal oxidative stress and response to therapy using hyperpolarized 13C dehydroascorbate magnetic resonance.Impaired endocytosis in proximal tubule from subchronic exposure to cadmium involves angiotensin II type 1 and cubilin receptors.Reactive oxygen species as important determinants of medullary flow, sodium excretion, and hypertensionGhrelin protects against renal damages induced by angiotensin-II via an antioxidative stress mechanism in mice.Upstream regulators and downstream effectors of NADPH oxidases as novel therapeutic targets for diabetic kidney disease.The ACE2/Ang-(1-7)/Mas Axis Regulates the Development of Pancreatic Endocrine Cells in Mouse Embryos.Reactive Oxygen Species and Nuclear Factor Erythroid 2-Related Factor 2 Activation in Diabetic Nephropathy: A Hidden TargetDual Activation of TRIF and MyD88 Adaptor Proteins by Angiotensin II Evokes Opposing Effects on Pressure, Cardiac Hypertrophy, and Inflammatory Gene Expression.Evidence of the Importance of Nox4 in Production of Hypertension in Dahl Salt-Sensitive Rats.The brain Renin-Angiotensin system and mitochondrial function: influence on blood pressure and baroreflex in transgenic rat strains.Evidence for a mitochondrial angiotensin-(1-7) system in the kidney.Mitochondrial-localized NADPH oxidase 4 is a source of superoxide in angiotensin II-stimulated neurons.Nox4 NADPH oxidase mediates peroxynitrite-dependent uncoupling of endothelial nitric-oxide synthase and fibronectin expression in response to angiotensin II: role of mitochondrial reactive oxygen species.Diabetes and Kidney Disease: Role of Oxidative Stress.The Angiotensin-(1-7)/Mas Axis Counteracts Angiotensin II-Dependent and -Independent Pro-inflammatory Signaling in Human Vascular Smooth Muscle CellsNox-derived ROS are acutely activated in pressure overload pulmonary hypertension: indications for a seminal role for mitochondrial Nox4NLRP3 inflammasome activation is involved in Ang II-induced kidney damage via mitochondrial dysfunction.From form to function: the role of Nox4 in the cardiovascular system.Nox4 and diabetic nephropathy: with a friend like this, who needs enemies?Renal ischemia/reperfusion injury; from pathophysiology to treatment.Angiotensinogen import in isolated proximal tubules: evidence for mitochondrial trafficking and uptake.Roles of oxidative stress, apoptosis, and heme oxygenase-1 in ethylbenzene-induced renal toxicity in NRK-52E cells.ERK5/HDAC5-mediated, resveratrol-, and pterostilbene-induced expression of MnSOD in human endothelial cells.Mycophenolate Mofetil Ameliorates Diabetic Nephropathy in db/db Mice.NOX4 functions as a mitochondrial energetic sensor coupling cancer metabolic reprogramming to drug resistance.Calcitriol Ameliorates AngiotensinII-Induced Renal Injury Partly via Upregulating A20.Genetic depletion of glutathione peroxidase-1 potentiates nephrotoxicity induced by multiple doses of cocaine via activation of angiotensin II AT1 receptor.Angiotensin (1-7) facilitates cardioprotection of ischemic preconditioning on ischemia-reperfusion-challenged rat heart.Assessment of the Renin-Angiotensin System in Cellular Organelle: New Arenas for Study in the Mitochondria.Angiotensin-(1-7) attenuated long-term hypoxia-stimulated cardiomyocyte apoptosis by inhibiting HIF-1α nuclear translocation via Mas receptor regulation.Upregulation of mitochondrial Nox4 mediates TGF-β-induced apoptosis in cultured mouse podocytes.
P2860
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P2860
Angiotensin II-induced mitochondrial Nox4 is a major endogenous source of oxidative stress in kidney tubular cells
description
2012 nî lūn-bûn
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2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2012年の論文
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2012年学术文章
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2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
name
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@ast
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@en
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@nl
type
label
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@ast
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@en
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@nl
prefLabel
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@ast
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@en
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@nl
P2093
P2860
P1433
P1476
Angiotensin II-induced mitocho ...... stress in kidney tubular cells
@en
P2093
Chun-Gyoo Ihm
Ju-Young Moon
Kyung-Hwan Jeong
Sang-Ho Lee
Tae-Won Lee
Yang-Gyun Kim
P2860
P304
P356
10.1371/JOURNAL.PONE.0039739
P407
P577
2012-07-09T00:00:00Z