Matrix metalloproteinase expression correlates with virulence following neurotropic mouse hepatitis virus infection - Test2 - elhamkhani - TriplyDB

Matrix metalloproteinase expression correlates with virulence following neurotropic mouse hepatitis virus infection

Matrix metalloproteinase expression correlates with virulence following neurotropic mouse hepatitis virus infection

http://www.wikidata.org/entity/Q34343357

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Differential effects of Tat proteins derived from HIV-1 subtypes B and recombinant CRF02_AG on human brain microvascular endothelial cells: implications for blood-brain barrier dysfunction A protective role for ELR+ chemokines during acute viral encephalomyelitis.Expression of matrix metalloproteinases and their tissue inhibitor during viral encephalitis.The pathogenesis of murine coronavirus infection of the central nervous system.Excess neutrophil infiltration during cytomegalovirus brain infection of interleukin-10-deficient mice T-cell production of matrix metalloproteinases and inhibition of parasite clearance by TIMP-1 during chronic Toxoplasma infection in the brain.Promoting remyelination: utilizing a viral model of demyelination to assess cell-based therapies Infection of human endothelial cells by Japanese encephalitis virus: increased expression and release of soluble HLA-E.Expression of the mouse hepatitis virus receptor by central nervous system microglia The role of the blood-brain barrier during Venezuelan equine encephalitis virus infection.CXCR2 signaling and host defense following coronavirus-induced encephalomyelitis.The chemokine receptor CXCR2 and coronavirus-induced neurologic disease.Chemokines in and out of the central nervous system: much more than chemotaxis and inflammation.Mouse hepatitis virus infection of the CNS: a model for defense, disease, and repair.The Biology of Persistent Infection: Inflammation and Demyelination following Murine Coronavirus Infection of the Central Nervous System Murine coronavirus neuropathogenesis: determinants of virulence.Interaction between endometrial epithelial cells and blood leucocytes promotes cytokine release and epithelial barrier function in response to Chlamydia trachomatis lipopolysaccharide stimulation.Immune responses to non-tumor antigens in the central nervous system.The contribution of neutrophils to CNS autoimmunity.Proangiogenic role of neutrophil-like inflammatory heterophils during neovascularization induced by growth factors and human tumor cells.

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P2860

Matrix metalloproteinase expression correlates with virulence following neurotropic mouse hepatitis virus infection

http://www.wikidata.org/entity/Q34343357

description

2002 nî lūn-bûn

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2002 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2002 թվականի օգոստոսին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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name

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@ast

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@en

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@nl

type

label

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@ast

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@en

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@nl

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Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@ast

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

@en

Matrix metalloproteinase expre ...... ouse hepatitis virus infection

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JVI.76.15.7374-7384.2002

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Journal of Virology

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Matrix metalloproteinase expre ...... ouse hepatitis virus infection

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P2093

David R Hinton

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Jiehao Zhou

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Norman W Marten

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Roscoe Atkinson

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Stephen A Stohlman

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P2860

A matrix metalloproteinase expressed on the surface of invasive tumour cells

Gelatinase B functions as regulator and effector in leukocyte biology

The cell biology of leukocyte-mediated proteolysis.

Leukocyte traffic in the central nervous system: the participants and their roles.

Mononuclear phagocyte differentiation, activation, and viral infection regulate matrix metalloproteinase expression: implications for human immunodeficiency virus type 1-associated dementia

Inflammatory mediators and modulation of blood-brain barrier permeability.

Morbillivirus infection of the mouse central nervous system induces region-specific upregulation of MMPs and TIMPs correlated to inflammatory cytokine expression

The role of matrix metalloproteinases in autoimmune damage to the central and peripheral nervous system.

Activation of matrix-metalloproteinase-2 and membrane-type-1-matrix-metalloproteinase in endothelial cells and induction of vascular permeability in vivo by human immunodeficiency virus-1 Tat protein and basic fibroblast growth factor

MHV infection of the CNS: mechanisms of immune-mediated control.

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7374-7384

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scholarly article

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10.1128/JVI.76.15.7374-7384.2002

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136378

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