Somatic chromosome abnormalities in the lungs of patients with pulmonary arterial hypertension. - Test2 - elhamkhani - TriplyDB

Somatic chromosome abnormalities in the lungs of patients with pulmonary arterial hypertension.

Somatic chromosome abnormalities in the lungs of patients with pulmonary arterial hypertension.

http://www.wikidata.org/entity/Q34402401

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Molecular pathogenesis of pulmonary arterial hypertension DNA Damage and Pulmonary Hypertension The genetics of pulmonary arterial hypertension Molecular Mechanisms of Pulmonary Vascular Remodeling in Pulmonary Arterial Hypertension Cellular and molecular biology of aging endothelial cells Whole-exome sequencing reveals TopBP1 as a novel gene in idiopathic pulmonary arterial hypertension Human primary lung endothelial cells in culture Dynamin-related protein 1-mediated mitochondrial mitotic fission permits hyperproliferation of vascular smooth muscle cells and offers a novel therapeutic target in pulmonary hypertension MicroRNA-140-5p and SMURF1 regulate pulmonary arterial hypertension.Genomic stability of pulmonary artery endothelial colony-forming cells in culture.Heritable forms of pulmonary arterial hypertension.Complement C3 deficiency attenuates chronic hypoxia-induced pulmonary hypertension in mice.The crossroads of iron with hypoxia and cellular metabolism. Implications in the pathobiology of pulmonary hypertension.DNA microarray and signal transduction analysis in pulmonary artery smooth muscle cells from heritable and idiopathic pulmonary arterial hypertension subjects Hyperplastic Growth of Pulmonary Artery Smooth Muscle Cells from Subjects with Pulmonary Arterial Hypertension Is Activated through JNK and p38 MAPK.The genetics of pulmonary arterial hypertension in the post-BMPR2 era.Altered MicroRNA processing in heritable pulmonary arterial hypertension: an important role for Smad-8 Pulmonary artery endothelium resident endothelial colony-forming cells in pulmonary arterial hypertension.Increased Mutagen Sensitivity and DNA Damage in Pulmonary Arterial Hypertension.RNA Sequencing Analysis Detection of a Novel Pathway of Endothelial Dysfunction in Pulmonary Arterial Hypertension.Altered expression and signal transduction of endothelin-1 receptors in heritable and idiopathic pulmonary arterial hypertension Human immunodeficiency virus, herpes virus infections, and pulmonary vascular disease.Integrating molecular genetics and systems approaches to pulmonary vascular diseases.Modulating the dysregulated migration of pulmonary arterial hypertensive smooth muscle cells with motif mimicking cell permeable peptides FK506 activates BMPR2, rescues endothelial dysfunction, and reverses pulmonary hypertension.A potential role for insulin resistance in experimental pulmonary hypertension Schistosomiasis causes remodeling of pulmonary vessels in the lung in a heterogeneous localized manner: Detailed study.The 6-Minute-Walk Distance Test as a Chronic Obstructive Pulmonary Disease Stratification Tool. Insights from the COPD Biomarker Qualification Consortium.Amphetamines promote mitochondrial dysfunction and DNA damage in pulmonary hypertension Relevant issues in the pathology and pathobiology of pulmonary hypertension.Update on pulmonary vascular diseases 2010.Anticipated classes of new medications and molecular targets for pulmonary arterial hypertension Clinical and molecular genetic features of hereditary pulmonary arterial hypertension.Genomics and proteomics of pulmonary vascular disease.Genetics and the molecular pathogenesis of pulmonary arterial hypertension.The genetic basis of pulmonary arterial hypertension.Molecular pathogenesis and current pathology of pulmonary hypertension.Acquired Mitochondrial Abnormalities, Including Epigenetic Inhibition of Superoxide Dismutase 2, in Pulmonary Hypertension and Cancer: Therapeutic Implications.The role of genetics in pulmonary arterial hypertension.Protein Expression by Human Pulmonary Artery Smooth Muscle Cells Containing a BMPR2 Mutation and the Action of ET-1 as Determined by Proteomic Mass Spectrometry.

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Somatic chromosome abnormalities in the lungs of patients with pulmonary arterial hypertension.

http://www.wikidata.org/entity/Q34402401

description

2010 nî lūn-bûn

@nan

2010 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հունիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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type

label

Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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prefLabel

Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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RCCM.201003-0491OC

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American Journal of Respiratory and Critical Care Medicine

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Somatic chromosome abnormaliti ...... lmonary arterial hypertension.

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Christopher D Coldren

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Kewal Asosingh

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Marileila Varella-Garcia

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Mark W Geraci

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Micheala A Aldred

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Patricia A Thistlethwaite

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Rubin M Tuder

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Serpil C Erzurum

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Suzy A Comhair

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Weiling Xu

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P2860

Noninvasive diagnosis of fetal aneuploidy by shotgun sequencing DNA from maternal blood

Genome instability: a mechanistic view of its causes and consequences

An abnormal mitochondrial-hypoxia inducible factor-1alpha-Kv channel pathway disrupts oxygen sensing and triggers pulmonary arterial hypertension in fawn hooded rats: similarities to human pulmonary arterial hypertension

Updated clinical classification of pulmonary hypertension.

Comparison of X-chromosome inactivation patterns in multiple tissues from human females.

The pathology of the early and late stages of primary pulmonary hypertension

Germline mutations in BMPR1A/ALK3 cause a subset of cases of juvenile polyposis syndrome and of Cowden and Bannayan-Riley-Ruvalcaba syndromes.

The bone morphogenetic protein pathway is inactivated in the majority of sporadic colorectal cancers.

ACCF/AHA 2009 expert consensus document on pulmonary hypertension a report of the American College of Cardiology Foundation Task Force on Expert Consensus Documents and the American Heart Association developed in collaboration with the American Coll

X chromosome-inactivation patterns of 1,005 phenotypically unaffected females.

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10.1164/RCCM.201003-0491OC

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