Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A "central hub" in schizophrenia pathophysiology?
about
Early interventions in risk groups for schizophrenia: what are we waiting for?In Sickness and in Health: Perineuronal Nets and Synaptic Plasticity in Psychiatric DisordersNMDA Receptor Function During Senescence: Implication on Cognitive PerformanceTargeting Oxidative Stress and Aberrant Critical Period Plasticity in the Developmental Trajectory to Schizophrenia.A Novel Bio-Psychosocial-Behavioral Treatment Model in Schizophrenia.Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia.Plastic and Neuroprotective Mechanisms Involved in the Therapeutic Effects of Cannabidiol in Psychiatric Disorders.Altered gene expression in schizophrenia: findings from transcriptional signatures in fibroblasts and blood.Cortical fast-spiking parvalbumin interneurons enwrapped in the perineuronal net express the metallopeptidases Adamts8, Adamts15 and NeprilysinAlternative kynurenic acid synthesis routes studied in the rat cerebellum.Dietary Intake of Sulforaphane-Rich Broccoli Sprout Extracts during Juvenile and Adolescence Can Prevent Phencyclidine-Induced Cognitive Deficits at AdulthoodNew Targets for Prevention of Schizophrenia: Is It Time for Interventions in the Premorbid Phase?Interneuron epigenomes during the critical period of cortical plasticity: Implications for schizophrenia.Neuronal copper homeostasis susceptibility by genetic defects in dysbindin, a schizophrenia susceptibility factor.Progress and Future Directions in Research on the Psychosis Prodrome: A Review for Clinicians.Long-Term Effects of Maternal Deprivation on Redox Regulation in Rat Brain: Involvement of NADPH Oxidase.Weaving a Net of Neurobiological Mechanisms in Schizophrenia and Unraveling the Underlying Pathophysiology.Dysregulation of the dopamine system in the pathophysiology of schizophrenia and depression.Neuroinflammation and Oxidative Stress in Psychosis and Psychosis RiskNew drug developments in psychosis: Challenges, opportunities and strategies.Potential drug targets and treatment of schizophrenia.Clozapine as a Model for Antipsychotic Development.Translational evaluation of translocator protein as a marker of neuroinflammation in schizophrenia.Supplementation with D-serine prevents the onset of cognitive deficits in adult offspring after maternal immune activation.Oxidative stress, prefrontal cortex hypomyelination and cognitive symptoms in schizophreniaImpaired fornix-hippocampus integrity is linked to peripheral glutathione peroxidase in early psychosisDifferences in omega-3 and omega-6 polyunsaturated fatty acid consumption in people at ultra-high risk of psychosis, first-episode schizophrenia, and in healthy controls.Reduced Slc1a1 expression is associated with neuroinflammation and impaired sensorimotor gating and cognitive performance in mice: Implications for schizophrenia.New Targets for Schizophrenia Treatment beyond the Dopamine HypothesisDecreased NOX2 expression in the brain of patients with bipolar disorder: association with valproic acid prescription and substance abuse.Implication of the glutamate-cystine antiporter xCT in schizophrenia cases linked to impaired GSH synthesis.Genetic Polymorphism Associated Prefrontal Glutathione and Its Coupling With Brain Glutamate and Peripheral Redox Status in Early PsychosisCharacterization of a Novel Mutation in SLC1A1 Associated with Schizophrenia.Integrative Analysis of Disease Signatures Shows Inflammation Disrupts Juvenile Experience-Dependent Cortical Plasticity.Enhanced storage capacity with errors in scale-free Hopfield neural networks: An analytical study.Reconceptualization of translocator protein as a biomarker of neuroinflammation in psychiatry.Dietary glucoraphanin prevents the onset of psychosis in the adult offspring after maternal immune activation.Social isolation stress and chronic glutathione deficiency have a common effect on the glutamine-to-glutamate ratio and myo-inositol concentration in the mouse frontal cortex.White Matter Disruptions in Schizophrenia Are Spatially Widespread and Topologically Converge on Brain Network Hubs.N-acetylcysteine in a Double-Blind Randomized Placebo-Controlled Trial: Toward Biomarker-Guided Treatment in Early Psychosis.
P2860
Q26747545-FDFCFFF1-D52D-44AF-90E0-885DA912BA0CQ26769948-AFF8E377-4C60-4918-A5DC-0AE2EEC6068CQ26770902-E25BCF77-A082-4DB9-A4C1-008C0698017FQ30380519-20982EF0-83D1-4F68-B107-159E060D2B12Q33624417-CCDA0309-501F-448A-9206-0CC9D21183D0Q33854995-F350E8C0-986F-4BA7-9D3E-87E6DD47EA55Q34557613-F3AC072B-D337-4A83-963F-8754D98E75AEQ35058307-0207C5B2-7EB9-4A7F-803C-E306A1C5C680Q35168850-0BE9ABDA-6AD0-4E27-AA2F-813DF95E5A15Q35618064-2BB81DFB-9EDC-469D-B099-DF2F6DD05219Q35673126-F4440394-2ECE-4C97-A956-78FD0A1226ECQ35739029-83DA18B4-3DC8-47C2-A785-C9EB6B8C11D2Q36055182-EB1CDCE6-8A29-48B2-BB5A-8EFE3439FC40Q36065482-3692AAF2-FAB7-419E-A92A-644DBED33B83Q36910682-C38CBB6E-E589-4A68-8D17-38D90C576CAEQ37734352-418B6EF6-2733-40CF-9B38-2CCE0624AE25Q38817438-A1AF478F-9D5E-4AE5-81DB-95DBE21F6002Q38852249-BB3F78E2-AE6B-478E-8E90-AA960D53A451Q38901529-3EF32C00-1614-4D02-96C7-6F919906B220Q38926246-99782A5A-9DA6-4699-9C4C-610FF4CF512AQ39206300-A048D91D-E5E6-4C3B-A66F-5693CBD8AE89Q39400094-9E276D3A-C79A-4BEC-87A5-6E4DA884C600Q40369760-3EC3CB13-F763-45F8-84C3-D63A30C1F880Q40448883-ACC709BF-C5C2-4F54-A7D7-444A1B183B85Q41197010-D2A29CCA-76AF-45A2-A6C7-80A22293F2F1Q41271064-2E3F5908-62C0-421A-9C93-5B14C23124E7Q41477685-078958EF-1A98-4380-9EFC-285505F1B05CQ41495551-4832DDE2-C396-4758-95EC-B3D0FAFAFEC0Q41573540-FF1C8F83-9F33-444C-8B95-CA2883B6AB1FQ41687504-84AB65F0-7DB8-46E8-8FA0-9628C0BF0821Q41701597-E07C716A-D401-4529-B0B6-135396628CCEQ41703320-3CAB1F30-59C8-4C84-8C5B-78E541431D61Q42154902-190F44FE-5D7F-413F-9CBB-118B7B31BF0FQ42156189-F97E26F1-3997-4FA8-982A-E132F9A68D8BQ45383729-7CC70A2F-EDD2-4DFE-9902-CFDC8D2DBA92Q47331971-6916C2C8-EE50-4397-B838-05055D3ED586Q47549654-14BB8AC2-8C9F-4797-BAF7-A45C84F54CFCQ47800687-4E4EA785-9D8A-4FBA-B36E-F03272EBA429Q47958113-5088FB50-0C09-48C8-B7C6-36D3E43A9560Q50281040-401A7734-2FB4-421B-9C85-90ACFF0E8DFF
P2860
Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A "central hub" in schizophrenia pathophysiology?
description
2014 nî lūn-bûn
@nan
2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@ast
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@en
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@nl
type
label
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@ast
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@en
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@nl
prefLabel
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@ast
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@en
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@nl
P2093
P2860
P921
P1476
Redox dysregulation, neuroinfl ...... schizophrenia pathophysiology?
@en
P2093
P2860
P356
10.1016/J.SCHRES.2014.06.021
P407
P50
P577
2014-07-04T00:00:00Z