Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damage - Test2 - elhamkhani - TriplyDB

Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damage

Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damage

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Breakdown of Immune Tolerance in Systemic Lupus Erythematosus by Dendritic Cells Dendritic Cells in Systemic Lupus Erythematosus: From Pathogenic Players to Therapeutic Tools Selected Aspects in the Pathogenesis of Autoimmune Diseases Dendritic cells: arbiters of immunity and immunological tolerance Dendritic cells and macrophages in the kidney: a spectrum of good and evil TLRs and interferons: a central paradigm in autoimmunity Targeting dendritic cell function during systemic autoimmunity to restore tolerance Contribution of dendritic cells to the autoimmune pathology of systemic lupus erythematosus Mutation of POLB causes lupus in mice Mechanisms of necroptosis in T cells The pathogenesis of systemic lupus erythematosus—an update The role of dendritic cells in autoimmunity Caspase-8 acts as a molecular rheostat to limit RIPK1- and MyD88-mediated dendritic cell activation Molecular control of steady-state dendritic cell maturation and immune homeostasis.Pivotal Functions of Plasmacytoid Dendritic Cells in Systemic Autoimmune Pathogenesis.Dysregulated cytokine production by dendritic cells modulates B cell responses in the NZM2410 mouse model of lupus The nanomaterial-dependent modulation of dendritic cells and its potential influence on therapeutic immunosuppression in lupus.Normalization of the lymph node T cell stromal microenvironment in lpr/lpr mice is associated with SU5416-induced reduction in autoantibodies.Early, transient depletion of plasmacytoid dendritic cells ameliorates autoimmunity in a lupus model.The role of dendritic cells in tissue-specific autoimmunity.Vitamin D in Autoimmunity: Molecular Mechanisms and Therapeutic Potential.Requirements for innate immune pathways in environmentally induced autoimmunity.A CD8 T cell/indoleamine 2,3-dioxygenase axis is required for mesenchymal stem cell suppression of human systemic lupus erythematosus.Differential pathways regulating innate and adaptive antitumor immune responses by particulate and soluble yeast-derived β-glucans.Interferon regulatory factor 1 marks activated genes and can induce target gene expression in systemic lupus erythematosus.Paradoxical effects of all-trans-retinoic acid on lupus-like disease in the MRL/lpr mouse model.Mechanisms of antigen presentation to T cells in murine graft-versus-host disease: cross-presentation and the appearance of cross-presentation.Local triggering of the ICOS coreceptor by CD11c(+) myeloid cells drives organ inflammation in lupus.The Role of Dendritic Cell Subsets and Innate Immunity in the Pathogenesis of Type 1 Diabetes and Other Autoimmune Diseases.Transforming growth factor beta-activated kinase 1 (TAK1)-dependent checkpoint in the survival of dendritic cells promotes immune homeostasis and function.Requirement of myeloid cell-specific Fas expression for prevention of systemic autoimmunity in mice.Mechanisms of B cell autoimmunity in SLE.Profound depletion of host conventional dendritic cells, plasmacytoid dendritic cells, and B cells does not prevent graft-versus-host disease induction.Megakaryocyte progenitors are the main APCs inducing Th17 response to lupus autoantigens and foreign antigens B Cell-Specific MHC Class II Deletion Reveals Multiple Nonredundant Roles for B Cell Antigen Presentation in Murine Lupus.Alpha 1 Antitrypsin Inhibits Dendritic Cell Activation and Attenuates Nephritis in a Mouse Model of Lupus.Langerhans Cells Maintain Local Tissue Tolerance in a Model of Systemic Autoimmune Disease.Serum from patients with SLE instructs monocytes to promote IgG and IgA plasmablast differentiation.IRF4-Dependent and IRF4-Independent Pathways Contribute to DC Dysfunction in Lupus.RNA sensing by conventional dendritic cells is central to the development of lupus nephritis

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P2860

Dendritic cells in lupus are not required for activation of T and B cells but promote their expansion, resulting in tissue damage

http://www.wikidata.org/entity/Q34443260

description

2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Dendritic cells in lupus are n ...... on, resulting in tissue damage

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J.IMMUNI.2010.11.025

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Dendritic cells in lupus are n ...... on, resulting in tissue damage

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Daniel H Kaplan

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Lino L Teichmann

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Mark J Shlomchik

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Michael Kashgarian

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Michelle L Ols

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P2860

Constitutive ablation of dendritic cells breaks self-tolerance of CD4 T cells and results in spontaneous fatal autoimmunity

iNOS-producing inflammatory dendritic cells constitute the major infected cell type during the chronic Leishmania major infection phase of C57BL/6 resistant mice

Development of monocytes, macrophages, and dendritic cells

Dendritic cells: specialized and regulated antigen processing machines

Dendritic cells induce peripheral T cell unresponsiveness under steady state conditions in vivo

B cell-targeted therapies for systemic lupus erythematosus: an update on clinical trial data.

T cell hyperactivity in lupus as a consequence of hyperstimulatory antigen-presenting cells.

TLR9 regulates TLR7- and MyD88-dependent autoantibody production and disease in a murine model of lupus

Deficiency of the type I interferon receptor protects mice from experimental lupus.

Skin immune sentinels in health and disease.

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