Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages. - Test2 - elhamkhani - TriplyDB

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

http://www.wikidata.org/entity/Q34451870

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Gene function in early mouse embryonic stem cell differentiation.Down-regulation of the forkhead transcription factor Foxp1 is required for monocyte differentiation and macrophage function Mammalian Krüppel-like factors in health and diseases The Kruppel-like factor KLF15 inhibits connective tissue growth factor (CTGF) expression in cardiac fibroblasts Kruppel-like factor 5 is an important mediator for lipopolysaccharide-induced proinflammatory response in intestinal epithelial cells Alert cell strategy in SIRS-induced vasculitis: sepsis and endothelial cells The Role of Krüppel-like Factor 4 in Renal Fibrosis Regulation of an inflammatory disease: Krüppel-like factors and atherosclerosis The Krüppel-like factors in female reproductive system pathologies Gene silencing of TACE enhances plaque stability and improves vascular remodeling in a rabbit model of atherosclerosis.Integrative assessment of chlorine-induced acute lung injury in mice Deficiency of the Kruppel-like factor KLF4 correlates with increased cell proliferation and enhanced skin tumorigenesis Transcriptional regulation of adipogenesis by KLF4 Epimorphin-induced MET sensitizes ovarian cancer cells to platinum Krüppel-like factor 4 inhibits proliferation by platelet-derived growth factor receptor beta-mediated, not by retinoic acid receptor alpha-mediated, phosphatidylinositol 3-kinase and ERK signaling in vascular smooth muscle cells Maternal deprivation alters epithelial secretory cell lineages in rat duodenum: role of CRF-related peptides Splenomegaly and modified erythropoiesis in KLF13-/- mice KLF4 is a key determinant in the development and progression of cerebral cavernous malformations.TNF-α induces phenotypic modulation in cerebral vascular smooth muscle cells: implications for cerebral aneurysm pathology.KLF4 regulates abdominal aortic aneurysm morphology and deletion attenuates aneurysm formation Cooperative binding of KLF4, pELK-1, and HDAC2 to a G/C repressor element in the SM22α promoter mediates transcriptional silencing during SMC phenotypic switching in vivo.Cigarette smoke modulates vascular smooth muscle phenotype: implications for carotid and cerebrovascular disease Identification of 15 new psoriasis susceptibility loci highlights the role of innate immunity.Smooth and cardiac muscle-selective knock-out of Kruppel-like factor 4 causes postnatal death and growth retardation.Sp1-dependent activation of KLF4 is required for PDGF-BB-induced phenotypic modulation of smooth muscle.Conditional deletion of Krüppel-like factor 4 delays downregulation of smooth muscle cell differentiation markers but accelerates neointimal formation following vascular injury Gene expression profiling in a mouse model of infantile neuronal ceroid lipofuscinosis reveals upregulation of immediate early genes and mediators of the inflammatory response Identification of novel transcriptional regulators involved in macrophage differentiation and activation in U937 cells.The Krüppel-like factor 9 cistrome in mouse hippocampal neurons reveals predominant transcriptional repression via proximal promoter binding Platelet factor 4 regulation of monocyte KLF4 in experimental cerebral malaria.The Transcription Regulator Krüppel-Like Factor 4 and Its Dual Roles of Oncogene in Glioblastoma and Tumor Suppressor in Neuroblastoma.Distinct role of Kruppel-like factor 11 in the regulation of prostaglandin E2 biosynthesis.KLF4 is a novel regulator of the constitutively expressed HSP90.Genetic deletion of Klf4 in the mouse intestinal epithelium ameliorates dextran sodium sulfate-induced colitis by modulating the NF-κB pathway inflammatory response Kruppel-like factor 4 inhibits epithelial-to-mesenchymal transition through regulation of E-cadherin gene expression Physiological stress-induced corticosterone increases heme uptake via KLF4-HCP1 signaling pathway in hippocampus neurons.High KLF4 level in normal tissue predicts poor survival in colorectal cancer patients Kruppel-like factor-4 transcriptionally regulates VE-cadherin expression and endothelial barrier function Klf4 overexpression activates epithelial cytokines and inflammation-mediated esophageal squamous cell cancer in mice.Expression of the tumor suppressor Krüppel-like factor 4 as a prognostic predictor for colon cancer.

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P2860

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

http://www.wikidata.org/entity/Q34451870

description

2005 nî lūn-bûn

@nan

2005 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@ast

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@en

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@nl

type

label

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@ast

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@en

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@nl

prefLabel

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@ast

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@en

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@nl

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P1433

Journal of Biological Chemistry

P1476

Kruppel-like factor 4 is a mediator of proinflammatory signaling in macrophages.

@en

P2093

Akm Khyrul Wara

http://www.w3.org/2001/XMLSchema#string

Maria A Lebedeva

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Mark W Feinberg

http://www.w3.org/2001/XMLSchema#string

Mukesh K Jain

http://www.w3.org/2001/XMLSchema#string

Sucharita Senbanerjee

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Zhuoxiao Cao

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P2860

RFLAT-1: a new zinc finger transcription factor that activates RANTES gene expression in T lymphocytes

Identification and characterization of a gene encoding a gut-enriched Krüppel-like factor expressed during growth arrest

Isolation, genomic structure, and expression of human erythroid Krüppel-like factor (EKLF)

Transactivation and growth suppression by the gut-enriched Krüppel-like factor (Krüppel-like factor 4) are dependent on acidic amino acid residues and protein-protein interaction

CREB-binding protein/p300 are transcriptional coactivators of p65

Functional interaction between coactivators CBP/p300, PCAF, and transcription factor FKLF2

Atherosclerosis — An Inflammatory Disease

A CBP integrator complex mediates transcriptional activation and AP-1 inhibition by nuclear receptors

Mechanisms of TGF-beta signaling from cell membrane to the nucleus

Jak-STAT pathways and transcriptional activation in response to IFNs and other extracellular signaling proteins

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38247-38258

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scholarly article

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10.1074/JBC.M509378200

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P433

46

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280

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2005-09-16T00:00:00Z

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16169848

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