Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
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Cytokine responses during chronic denervation.Regulating Axonal Responses to Injury: The Intersection between Signaling Pathways Involved in Axon Myelination and The Inhibition of Axon RegenerationSpatiotemporal expression of SKIP after rat sciatic nerve crushRegulation of expression of fibronectin and its receptor, alpha 5 beta 1, during development and regeneration of peripheral nerve.Platelet-derived growth factors and fibroblast growth factors are mitogens for rat Schwann cells.Combinatorial actions of Tgfβ and Activin ligands promote oligodendrocyte development and CNS myelinationMalignant peripheral nerve sheath tumour arising within neurofibroma. An immunohistochemical analysis in the comparison between benign and malignant components.Transforming growth factor-beta 1 in experimental autoimmune neuritis. Cellular localization and time courseBasement membrane and repair of injury to peripheral nerve: defining a potential role for macrophages, matrix metalloproteinases, and tissue inhibitor of metalloproteinases-1.Transforming growth factor-beta 1 regulates axon/Schwann cell interactions.Stromelysin generates a fibronectin fragment that inhibits Schwann cell proliferation.Expression and localization of Ski determine cell type-specific TGFbeta signaling effects on the cell cycleGrowth factor and stem cell enhanced conduits in peripheral nerve regeneration and repair.Pre-degenerated peripheral nerves co-cultured with bone marrow-derived cells: a new technique for harvesting high-purity Schwann cells.Early mutation of the neu (erbB-2) gene during ethylnitrosourea-induced oncogenesis in the rat Schwann cell lineage.Clinical strategies to enhance nerve regeneration in composite tissue allotransplantation.Schwann Cells: Development and Role in Nerve RepairPolyurethane/Gelatin Nanofibrils Neural Guidance Conduit Containing Platelet-Rich Plasma and Melatonin for Transplantation of Schwann Cells.Non-antagonistic relationship between mitogenic factors and cAMP in adult Schwann cell re-differentiation.Cytokines and their role as growth factors and in regulation of immune responses.Glial growth factors I-III are specific mitogens for glial cells.Effects of transforming growth factor beta 1 on the adenylyl cyclase-cAMP pathway in prostate cancer.Transforming growth factor-beta 1 enhances serum-induced dephosphorylation of the P53 protein in cell lines growth-inhibited by this factor.In vitro studies of glial cells: what can we learn about demyelinating diseases?The effects of cAMP on differentiation of cultured Schwann cells: progression from an early phenotype (04+) to a myelin phenotype (P0+, GFAP-, N-CAM-, NGF-receptor-) depends on growth inhibition.Calcitonin gene-related peptide promotes Schwann cell proliferation.Interferon-gamma, tumor necrosis factor-alpha, and transforming growth factor-beta inhibit cyclic AMP-induced Schwann cell differentiation.Effects of ethanol and basic fibroblast growth factor on the transforming growth factor beta1 regulated proliferation of cortical astrocytes and C6 astrocytoma cells.Transforming growth factor-beta and forskolin attenuate the adverse effects of long-term Schwann cell denervation on peripheral nerve regeneration in vivo.Nerve injury and inflammatory cytokines modulate gap junctions in the peripheral nervous system.Phenotypic and Functional Characteristics of Human Schwann Cells as Revealed by Cell-Based Assays and RNA-SEQ.Effect of local administration of platelet-rich plasma (PRP) on peripheral nerve regeneration: An experimental study in the rabbit model.Multiple isoforms of neuregulin are expressed in developing rat dorsal root ganglia.Epigenetic induction of the Ink4a/Arf locus prevents Schwann cell overproliferation during nerve regeneration and after tumorigenic challenge.Schwann Cells Secrete a PDGF-like Factor: Evidence for an Autocrine Growth Mechanism involving PDGF.MicroRNA Mediated Regulation of Schwann Cell Migration and Proliferation in Peripheral Nerve Injury.
P2860
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P2860
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
description
1989 nî lūn-bûn
@nan
1989 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1989 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
1989年の論文
@ja
1989年論文
@yue
1989年論文
@zh-hant
1989年論文
@zh-hk
1989年論文
@zh-mo
1989年論文
@zh-tw
1989年论文
@wuu
name
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@ast
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@en
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@nl
type
label
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@ast
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@en
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@nl
prefLabel
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@ast
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@en
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@nl
P2093
P2860
P356
P1476
Transforming growth factors-beta 1 and beta 2 are mitogens for rat Schwann cells.
@en
P2093
P2860
P304
P356
10.1083/JCB.109.6.3419
P407
P433
P50
P577
1989-12-01T00:00:00Z