Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
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Redox mechanisms in age-related lung fibrosisCancer: An Oxidative Crosstalk between Solid Tumor Cells and Cancer Associated FibroblastsThe sexual dimorphism associated with pulmonary hypertension corresponds to a fibrotic phenotypeEnhanced caveolin-1 expression in smooth muscle cells: Possible prelude to neointima formation.TGF-β and Hypoxia/Reoxygenation Promote Radioresistance of A549 Lung Cancer Cells through Activation of Nrf2 and EGFR.Nox and Inflammation in the Vascular Adventitia.Enzymatic regulation and functional relevance of NOX5Curcumin attenuates high glucose-induced podocyte apoptosis by regulating functional connections between caveolin-1 phosphorylation and ROS.Cloning, Characteristics, and Functional Analysis of Rabbit NADPH Oxidase 5.Nox5 stability and superoxide production is regulated by C-terminal binding of Hsp90 and CO-chaperones.Endothelial Nox1 oxidase assembly in human pulmonary arterial hypertension; driver of Gremlin1-mediated proliferation.Reactive Oxygen and Nitrogen Species in the Development of Pulmonary Hypertension.Caveolin 1-related autophagy initiated by aldosterone-induced oxidation promotes liver sinusoidal endothelial cells defenestration.Inhibition of histone deacetylase reduces transcription of NADPH oxidases and ROS production and ameliorates pulmonary arterial hypertensionMechanism of melanoma cells selective apoptosis induced by a photoactive NADPH analogueCaveolin-1 serves as a negative effector in senescent human gingival fibroblasts during Fusobacterium nucleatum infection.Expression and significance of caveolin-1 in hepatitis B virus-associated hepatocellular carcinoma.From Physiological Redox Signalling to Oxidant Stress.The ROS derived mitochondrial respirstion not from NADPH oxidase plays key role in Celastrol against angiotensin II-mediated HepG2 cell proliferation.Endothelial deletion of protein tyrosine phosphatase-1B protects against pressure overload-induced heart failure in mice.RhoA S-nitrosylation as a regulatory mechanism influencing endothelial barrier function in response to G+-bacterial toxins.Caveolae are involved in mechanotransduction during pulmonary hypertension.
P2860
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P2860
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
description
2014 nî lūn-bûn
@nan
2014 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2014 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2014年の論文
@ja
2014年論文
@yue
2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
@zh-mo
2014年論文
@zh-tw
2014年论文
@wuu
name
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@ast
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@en
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@nl
type
label
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@ast
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@en
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@nl
prefLabel
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@ast
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@en
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@nl
P2093
P2860
P1476
Caveolin-1 is a negative regulator of NADPH oxidase-derived reactive oxygen species.
@en
P2093
David J R Fulton
Huijuan Dou
Ruslan Rafikov
Scott Barman
Stephen M Black
Steven Haigh
Weihong Han
Yanfang Yu
Yunchao Su
P2860
P304
P356
10.1016/J.FREERADBIOMED.2014.04.029
P577
2014-05-14T00:00:00Z