High-mobility group box-1 induces proinflammatory cytokines production of Kupffer cells through TLRs-dependent signaling pathway after burn injury
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Immunity in arterial hypertension: associations or causalities?Inhibitor of Tec kinase, LFM-A13, decreases pro-inflammatory mediators production in LPS-stimulated RAW264.7 macrophages via NF-κB pathway.HMGB1 promotes a p38MAPK associated non-infectious inflammatory response pathway in human fetal membranes.High mobility group box-1 promotes the proliferation and migration of hepatic stellate cells via TLR4-dependent signal pathways of PI3K/Akt and JNKThe sterile inflammation in the exacerbation of HBV-associated liver injury.Effects of hydrogen-rich saline on early acute kidney injury in severely burned rats by suppressing oxidative stress induced apoptosis and inflammation.Modulation of Human Macrophage Responses to Mycobacterium tuberculosis by Silver Nanoparticles of Different Size and Surface Modification.TSG-6 secreted by human umbilical cord-MSCs attenuates severe burn-induced excessive inflammation via inhibiting activations of P38 and JNK signalingCurrent understanding of tyrosine kinase BMX in inflammation and its inhibitors.HMGB1 translocation and release mediate cigarette smoke-induced pulmonary inflammation in mice through a TLR4/MyD88-dependent signaling pathway.High-mobility group box 1 promotes extracellular matrix synthesis and wound repair in human bronchial epithelial cells.Age-dependent alterations to paraventricular nucleus insulin-like growth factor 1 receptor as a possible link between sympathoexcitation and inflammation.The liver is a metabolic and immunologic organ: A reconsideration of metabolic decompensation due to infection in inborn errors of metabolism (IEM).Toll-like receptor responses are suppressed in trauma ICU patients.The adaptation model of immunity.Toll-Like Receptor Signaling in Burn Wound Healing and Scarring.Damage-associated molecular patterns (DAMPs) released after burn are associated with inflammation and monocyte activation.HMGB1 Increases IL-1β Production in Vascular Smooth Muscle Cells via NLRP3 Inflammasome.High-Mobility Group Box-1 and Liver DiseaseInhibitory effects of miR-25 targeting HMGB1 on macrophage secretion of inflammatory cytokines in sepsis
P2860
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P2860
High-mobility group box-1 induces proinflammatory cytokines production of Kupffer cells through TLRs-dependent signaling pathway after burn injury
description
2012 nî lūn-bûn
@nan
2012 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
High-mobility group box-1 indu ...... ling pathway after burn injury
@ast
High-mobility group box-1 indu ...... ling pathway after burn injury
@en
High-mobility group box-1 indu ...... ling pathway after burn injury
@nl
type
label
High-mobility group box-1 indu ...... ling pathway after burn injury
@ast
High-mobility group box-1 indu ...... ling pathway after burn injury
@en
High-mobility group box-1 indu ...... ling pathway after burn injury
@nl
prefLabel
High-mobility group box-1 indu ...... ling pathway after burn injury
@ast
High-mobility group box-1 indu ...... ling pathway after burn injury
@en
High-mobility group box-1 indu ...... ling pathway after burn injury
@nl
P2093
P2860
P1433
P1476
High-mobility group box-1 indu ...... ling pathway after burn injury
@en
P2093
Ren-Su Wang
Xu-Lin Chen
Ye-Xiang Sun
Yong-Jie Wang
P2860
P304
P356
10.1371/JOURNAL.PONE.0050668
P407
P577
2012-11-27T00:00:00Z