Progestin suppression of miR-29 potentiates dedifferentiation of breast cancer cells via KLF4.
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Steroid hormones, steroid receptors, and breast cancer stem cellsThe role of miR-29b in cancer: regulation, function, and signalingMicroRNA regulation of progesterone receptor in breast cancerEpithelial-to-mesenchymal transition and the cancer stem cell phenotype: insights from cancer biology with therapeutic implications for colorectal cancer.Suppressive role exerted by microRNA-29b-1-5p in triple negative breast cancer through SPIN1 regulation.Natural and synthetic progestins enrich cancer stem cell-like cells in hormone-responsive human breast cancer cell populations in vitro.A novel role of Krüppel-like factor 4 in Zhikong scallop Chlamys farreri during spermatogenesis.The inhibitory role of Mir-29 in growth of breast cancer cellsOverexpression of microRNA-200c predicts poor outcome in patients with PR-negative breast cancer.Ovarian steroids, stem cells and uterine leiomyoma: therapeutic implications.Generation of breast cancer stem cells by steroid hormones in irradiated human mammary cell linesProgesterone action in breast, uterine, and ovarian cancers.ADAM12-L is a direct target of the miR-29 and miR-200 families in breast cancer.Progesterone downregulation of miR-141 contributes to expansion of stem-like breast cancer cells through maintenance of progesterone receptor and Stat5a.A systems biology approach identifies a regulatory network in parotid acinar cell terminal differentiation.Disruption of miR-29 Leads to Aberrant Differentiation of Smooth Muscle Cells Selectively Associated with Distal Lung Vasculature.Krüppel-like Factor 4 Promotes Esophageal Squamous Cell Carcinoma Differentiation by Up-regulating Keratin 13 ExpressionCorrelative Analysis of miRNA Expression and Oncotype Dx Recurrence Score in Estrogen Receptor Positive Breast CarcinomasLuteolin inhibits progestin-dependent angiogenesis, stem cell-like characteristics, and growth of human breast cancer xenografts.Fucoidan Elevates MicroRNA-29b to Regulate DNMT3B-MTSS1 Axis and Inhibit EMT in Human Hepatocellular Carcinoma CellsMicroRNA-29b/Tet1 regulatory axis epigenetically modulates mesendoderm differentiation in mouse embryonic stem cells.Cross disease analysis of co-functional microRNA pairs on a reconstructed network of disease-gene-microRNA tripartite.Progesterone-inducible cytokeratin 5-positive cells in luminal breast cancer exhibit progenitor properties.Steroid induction of therapy-resistant cytokeratin-5-positive cells in estrogen receptor-positive breast cancer through a BCL6-dependent mechanism.Toll-like receptor 3 (TLR3) activation induces microRNA-dependent reexpression of functional RARβ and tumor regression.Exploring and exploiting the fundamental role of microRNAs in tumor pathogenesis.microRNA-mediated regulation of the tumor microenvironment.Brown Seaweed Fucoidan Inhibits Cancer Progression by Dual Regulation of mir-29c/ADAM12 and miR-17-5p/PTEN Axes in Human Breast Cancer Cells.Lysine methylation of progesterone receptor at activation function 1 regulates both ligand-independent activity and ligand sensitivity of the receptor.Role of 3'-untranslated region translational control in cancer development, diagnostics and treatment.Induced cancer stem cells generated by radiochemotherapy and their therapeutic implications.MicroRNAs and the cancer phenotype: profiling, signatures and clinical implications.MicroRNAs in human lung cancer.Recurrence and metastasis of breast cancer is influenced by ovarian hormone's effect on breast cancer stem cells.Impact of progesterone on stem/progenitor cells in the human breast.Progesterone stimulates progenitor cells in normal human breast and breast cancer cells.Downregulation of miR-29 contributes to cisplatin resistance of ovarian cancer cells.Evaluation of miR-29c inhibits endotheliocyte migration and angiogenesis of human endothelial cells by suppressing the insulin like growth factor 1The MicroRNA 29 Family Promotes Type II Cell Differentiation in Developing Lung.MicroRNAs, a subpopulation of regulators, are involved in breast cancer progression through regulating breast cancer stem cells.
P2860
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P2860
Progestin suppression of miR-29 potentiates dedifferentiation of breast cancer cells via KLF4.
description
2012 nî lūn-bûn
@nan
2012 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@ast
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@en
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@nl
type
label
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@ast
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@en
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@nl
prefLabel
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@ast
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@en
Progestin suppression of miR-2 ...... breast cancer cells via KLF4.
@nl
P2093
P2860
P356
P1433
P1476
Progestin suppression of miR-2 ...... f breast cancer cells via KLF4
@en
P2093
C A Sartorius
J Finlay-Schultz
J K Richer
N S Spoelstra
P Hendricks
S D Axlund
P2860
P2888
P304
P356
10.1038/ONC.2012.275
P407
P577
2012-07-02T00:00:00Z
P6179
1012220098