Hydrogen ions kill brain at concentrations reached in ischemia. - Test2 - elhamkhani - TriplyDB

Hydrogen ions kill brain at concentrations reached in ischemia.

Hydrogen ions kill brain at concentrations reached in ischemia.

http://www.wikidata.org/entity/Q34608924

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Proton modulation of recombinant GABA(A) receptors: influence of GABA concentration and the beta subunit TM2-TM3 domain Astrocytic clasmatodendrosis in hippocampal organ culture Effects of 2-deoxy-d-glucose on focal cerebral ischemia in hyperglycemic rats.Effect of dichloroacetate on recovery of brain lactate, phosphorus energy metabolites, and glutamate during reperfusion after complete cerebral ischemia in rats.Bicarbonate and ammonia changes in brain during spreading depression.What do stroke and brain trauma have in common?Acid-induced injury in elasmobranch brain.Phenylbutyrate therapy for pyruvate dehydrogenase complex deficiency and lactic acidosis.Lactate storm marks cerebral metabolism following brain trauma Neurotoxicity of advanced glycation endproducts during focal stroke and neuroprotective effects of aminoguanidine Light and electron microscopic evaluation of hydrogen ion-induced brain necrosis.The effects of extracellular acidosis on neurons and glia in vitro Neocortical spreading depression provokes the expression of c-fos protein-like immunoreactivity within trigeminal nucleus caudalis via trigeminovascular mechanisms.NADPH oxidase-2: linking glucose, acidosis, and excitotoxicity in stroke.Relationship between injury severity, random blood glucose and management outcome in a cohort of Nigerian patients with head injury Calcium-permeable ion channels involved in glutamate receptor-independent ischemic brain injury Failure and function of intracellular pH regulation in acute hypoxic-ischemic injury of astrocytes.Ca2+ -permeable acid-sensing ion channels and ischemic brain injury.Pyruvate Dehydrogenase Kinase-mediated Glycolytic Metabolic Shift in the Dorsal Root Ganglion Drives Painful Diabetic Neuropathy.Effects of deferoxamine on brain injury after transient focal cerebral ischemia in rats with hyperglycemia.In vivo proton-electron double-resonance imaging of extracellular tumor pH using an advanced nitroxide probe.Age or ischemia uncouples the blood flow response, tissue acidosis, and direct current potential signature of spreading depolarization in the rat brain.Lactacidosis modulates glutathione metabolism and oxidative glutamate toxicity.Reperfusion accelerates acute neuronal death induced by simulated ischemia.Contribution of anion transporters to the acidosis-induced swelling and intracellular acidification of glial cells.Astrocytes: form, functions, and roles in disease.Evolving concepts about the role of acidosis in ischemic neuropathology.Pathophysiological mechanisms of brain damage from status epilepticus.Characterization of the rat Na+/H+ exchanger isoform NHE4 and localization in rat hippocampus.Newer insights to pathogenesis of traumatic brain injury.Neuropathology and pathogenesis of mitochondrial diseases.Biological basis for PCO2 as a detector of ischemia.Effects of lactacidosis on glial cell volume and viability.Oxidative stress and apoptosis after acute respiratory hypoxia and reoxygenation in rat brain.Effects of extracellular pH reductions on [(3)H]D-aspartate and [(3)H]noradrenaline release by presynaptic nerve terminals isolated from rat cerebral cortex.Neuronal damage following intraspinal injection of a nitric oxide synthase inhibitor in the rat.Rapid astrocyte death induced by transient hypoxia, acidosis, and extracellular ion shifts.Neurotensin analog NT69L induces rapid and prolonged hypothermia after hypoxic ischemia.The influence of hyperglycemia on neurological outcome in patients with severe head injury.An intravascular technique to occlude the middle cerebral artery in baboons.

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P2860

Hydrogen ions kill brain at concentrations reached in ischemia.

http://www.wikidata.org/entity/Q34608924

description

1987 nî lūn-bûn

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1987 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

1987 թվականի օգոստոսին հրատարակված գիտական հոդված

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1987年の論文

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1987年論文

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1987年論文

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1987年論文

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1987年論文

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1987年論文

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1987年论文

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name

Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Hydrogen ions kill brain at concentrations reached in ischemia.

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Journal of Cerebral Blood Flow & Metabolism

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Hydrogen ions kill brain at concentrations reached in ischemia.

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C K Petito

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F Plum

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R P Kraig

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W A Pulsinelli

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P2860

Carbonic acid buffer changes during complete brain ischemia.

Bicarbonate and ammonia changes in brain during spreading depression.

Acid-induced changes of brain protein buffering.

Intracellular pH.

Light and electron microscopic evaluation of hydrogen ion-induced brain necrosis.

The anion transport system of the red blood cell. The role of membrane protein evaluated by the use of 'probes'.

A new model of bilateral hemispheric ischemia in the unanesthetized rat.

pH regulation in barnacle muscle fibers: dependence on intracellular and extracellular pH.

Experimental displacement of intracellular pH and the mechanism of its subsequent recovery.

Hydrogen ion buffering during complete brain ischemia

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379-386

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10.1038/JCBFM.1987.80

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19551133

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3611202

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