Enhanced group II mGluR-mediated inhibition of pain-related synaptic plasticity in the amygdala.
about
Pain-related anxiety-like behavior requires CRF1 receptors in the amygdalaGroup III mGluR7 and mGluR8 in the amygdala differentially modulate nocifensive and affective pain behaviorsThe amygdala between sensation and affect: a role in pain.Differential effects of mGluR7 and mGluR8 activation on pain-related synaptic activity in the amygdalaGroup II mGluRs modulate baseline and arthritis pain-related synaptic transmission in the rat medial prefrontal cortex.Amygdala pain mechanisms.Presynaptic GABAB receptors reduce transmission at parabrachial synapses in the lateral central amygdala by inhibiting N-type calcium channels.PKA and ERK, but not PKC, in the amygdala contribute to pain-related synaptic plasticity and behaviorDynamic metabotropic control of intrinsic firing in cerebellar unipolar brush cellsNeuropeptide S: a novel regulator of pain-related amygdala plasticity and behaviorsForebrain pain mechanisms.Group III metabotropic glutamate receptors (mGluRs) modulate transmission of gustatory inputs in the brain stem.Endogenous N-acetylaspartylglutamate (NAAG) inhibits synaptic plasticity/transmission in the amygdala in a mouse inflammatory pain model.Intracerebroventricular administration of N-acetylaspartylglutamate (NAAG) peptidase inhibitors is analgesic in inflammatory pain.Differential mechanisms of CRF1 and CRF2 receptor functions in the amygdala in pain-related synaptic facilitation and behavior.Differential effects of CRF1 and CRF2 receptor antagonists on pain-related sensitization of neurons in the central nucleus of the amygdala.Neurochemical properties of the synapses between the parabrachial nucleus-derived CGRP-positive axonal terminals and the GABAergic neurons in the lateral capsular division of central nucleus of amygdala.Red nucleus glutamate facilitates neuropathic allodynia induced by spared nerve injury through non-NMDA and metabotropic glutamate receptors.Group II Metabotropic Glutamate Receptors: Role in Pain Mechanisms and Pain Modulation
P2860
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P2860
Enhanced group II mGluR-mediated inhibition of pain-related synaptic plasticity in the amygdala.
description
2006 nî lūn-bûn
@nan
2006 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@ast
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@en
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@nl
type
label
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@ast
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@en
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@nl
prefLabel
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@ast
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@en
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@nl
P2093
P2860
P356
P1433
P1476
Enhanced group II mGluR-mediat ...... ic plasticity in the amygdala.
@en
P2093
Gary C Bird
Jeong S Han
Volker Neugebauer
P2860
P2888
P356
10.1186/1744-8069-2-18
P577
2006-05-08T00:00:00Z
P5875
P6179
1016786600