Chemokines, inflammation and Trypanosoma cruzi infection.
about
Activation of transcription factors AP-1 and NF-kappa B in murine Chagasic myocarditisImmune Evasion Strategies of Trypanosoma cruziCurrent understanding of immunity to Trypanosoma cruzi infection and pathogenesis of Chagas diseaseIL-17 produced during Trypanosoma cruzi infection plays a central role in regulating parasite-induced myocarditisMexican Trypanosoma cruzi T. cruzi I strains with different degrees of virulence induce diverse humoral and cellular immune responses in a murine experimental infection model.Cardiac involvement with parasitic infections.Inflammasomes in host response to protozoan parasites.Myocardial gene expression of T-bet, GATA-3, Ror-γt, FoxP3, and hallmark cytokines in chronic Chagas disease cardiomyopathy: an essentially unopposed TH1-type response.Immunoregulatory networks in human Chagas disease.Chagas disease cardiomyopathy: immunopathology and genetics.Type 1 chemokine receptor expression in Chagas' disease correlates with morbidity in cardiac patients.Deficient regulatory T cell activity and low frequency of IL-17-producing T cells correlate with the extent of cardiomyopathy in human Chagas' disease.Parasite-derived neurotrophic factor/trans-sialidase of Trypanosoma cruzi links neurotrophic signaling to cardiac innate immune responseThe receptor Slamf1 on the surface of myeloid lineage cells controls susceptibility to infection by Trypanosoma cruziMannose-binding lectin regulates host resistance and pathology during experimental infection with Trypanosoma cruziMyocardial chemokine expression and intensity of myocarditis in Chagas cardiomyopathy are controlled by polymorphisms in CXCL9 and CXCL10.Macrophage migration inhibitory factor contributes to host defense against acute Trypanosoma cruzi infection.High levels of soluble CD40 ligand and matrix metalloproteinase-9 in serum are associated with favorable clinical evolution in human visceral leishmaniasis.Microarray analysis of the mammalian thromboxane receptor-Trypanosoma cruzi interaction.Human leucocyte antigen-G (HLA-G) and its murine functional homolog Qa2 in the Trypanosoma cruzi Infection.Polymorphism in the alpha cardiac muscle actin 1 gene is associated to susceptibility to chronic inflammatory cardiomyopathy.Cardiac gene expression profiling provides evidence for cytokinopathy as a molecular mechanism in Chagas' disease cardiomyopathy.Interferon-gamma promotes infection of astrocytes by Trypanosoma cruziMast cell function and death in Trypanosoma cruzi infection.The role of co-infections in mother-to-child transmission of HIV.MicroRNA Transcriptome Profiling in Heart of Trypanosoma cruzi-Infected Mice: Parasitological and Cardiological OutcomesGenetic Susceptibility to Cardiac and Digestive Clinical Forms of Chronic Chagas Disease: Involvement of the CCR5 59029 A/G Polymorphism.Role of the Toll/interleukin-1 receptor signaling pathway in host resistance and pathogenesis during infection with protozoan parasites.Innate and acquired immunity in the pathogenesis of Chagas disease.Thromboxane A2 is a key regulator of pathogenesis during Trypanosoma cruzi infection.Enalapril in Combination with Benznidazole Reduces Cardiac Inflammation and Creatine Kinases in Mice Chronically Infected with Trypanosoma cruzi.Low-dose benznidazole treatment results in parasite clearance and attenuates heart inflammatory reaction in an experimental model of infection with a highly virulent Trypanosoma cruzi strain.Developments in the management of Chagas cardiomyopathy.Trypanosoma cruzi (Chagas' disease agent) reduces HIV-1 replication in human placenta.Tissue-specific oxidative imbalance and mitochondrial dysfunction during Trypanosoma cruzi infection in micePathology affects different organs in two mouse strains chronically infected by a Trypanosoma cruzi clone: a model for genetic studies of Chagas' diseaseCurrent concepts in immunoregulation and pathology of human Chagas disease.A magnetic resonance imaging study of intestinal dilation in Trypanosoma cruzi-infected mice deficient in nitric oxide synthaseNLR proteins and parasitic disease.Trypanosoma cruzi infection activates extracellular signal-regulated kinase in cultured endothelial and smooth muscle cells.
P2860
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P2860
Chemokines, inflammation and Trypanosoma cruzi infection.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Chemokines, inflammation and Trypanosoma cruzi infection.
@ast
Chemokines, inflammation and Trypanosoma cruzi infection.
@en
Chemokines, inflammation and Trypanosoma cruzi infection.
@nl
type
label
Chemokines, inflammation and Trypanosoma cruzi infection.
@ast
Chemokines, inflammation and Trypanosoma cruzi infection.
@en
Chemokines, inflammation and Trypanosoma cruzi infection.
@nl
prefLabel
Chemokines, inflammation and Trypanosoma cruzi infection.
@ast
Chemokines, inflammation and Trypanosoma cruzi infection.
@en
Chemokines, inflammation and Trypanosoma cruzi infection.
@nl
P1476
Chemokines, inflammation and Trypanosoma cruzi infection
@en
P2093
Mauro M Teixeira
P304
P356
10.1016/S1471-4922(02)02283-3
P577
2002-06-01T00:00:00Z