FGFR1 over-expression in primary rhabdomyosarcoma tumors is associated with hypomethylation of a 5' CpG island and abnormal expression of the AKT1, NOG, and BMP4 genes.
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FGFR1 amplification is associated with poor prognosis and smoking in non-small-cell lung cancer.The Regulation and Function of Fibroblast Growth Factor 8 and Its Function during Gonadotropin-Releasing Hormone Neuron DevelopmentBayesian methods for expression-based integration of various types of genomics data.Receptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi.Pediatric Rhabdomyosarcoma.Laryngeal embryonal rhabdomyosarcoma in an adult - a case presentation in the eyes of geneticists and clinicians.DNA methylation dynamics in muscle development and disease.Pedican: an online gene resource for pediatric cancers with literature evidenceGenome-wide DNA methylation studies suggest distinct DNA methylation patterns in pediatric embryonal and alveolar rhabdomyosarcomas.Global DNA methylation profiling uncovers distinct methylation patterns of protocadherin alpha4 in metastatic and non-metastatic rhabdomyosarcoma.Discovery of progenitor cell signatures by time-series synexpression analysis during Drosophila embryonic cell immortalization.Fibroblast Growth Factor 8 Expression in GT1-7 GnRH-Secreting Neurons Is Androgen-Independent, but Can Be Upregulated by the Inhibition of DNA Methyltransferases.Cause and consequences of genetic and epigenetic alterations in human cancer.Molecular and cellular biology of rhabdomyosarcoma.Receptor tyrosine kinases as therapeutic targets in rhabdomyosarcoma.Fibroblast growth factors and their receptors in cancer.Epigenetic and epigenomic mechanisms shape sarcoma and other mesenchymal tumor pathogenesis.Proof-of-concept rare cancers in drug development: the case for rhabdomyosarcoma.Targeted polytherapy in small cell sarcoma and its association with doxorubicin.Downregulation of microRNAs miR-1, -206 and -29 stabilizes PAX3 and CCND2 expression in rhabdomyosarcoma.BMP signalling permits population expansion by preventing premature myogenic differentiation in muscle satellite cells.Genome-wide analysis of histone H3 lysine9 trimethylation by ChIP-seq in peripheral blood mononuclear cells of uremia patients.Integrated time course omics analysis distinguishes immediate therapeutic response from acquired resistance.Single-walled and multi-walled carbon nanotubes induce sequence-specific epigenetic alterations in 16 HBE cells.
P2860
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P2860
FGFR1 over-expression in primary rhabdomyosarcoma tumors is associated with hypomethylation of a 5' CpG island and abnormal expression of the AKT1, NOG, and BMP4 genes.
description
2007 nî lūn-bûn
@nan
2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@ast
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@en
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@nl
type
label
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@ast
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@en
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@nl
prefLabel
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@ast
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@en
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@nl
P356
P1476
FGFR1 over-expression in prima ...... the AKT1, NOG, and BMP4 genes.
@en
P2093
Isaac Meller
Myriam Goldstein
P304
P356
10.1002/GCC.20489
P577
2007-11-01T00:00:00Z