MicroRNA-486-3p regulates γ-globin expression in human erythroid cells by directly modulating BCL11A.
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MicroRNA Expression in β-Thalassemia and Sickle Cell Disease: A Role in The Induction of Fetal HemoglobinEpigenetic regulation of fetal globin gene expression in adult erythroid cellsAn Improved microRNA Annotation of the Canine GenomeHemoglobin switching's surprise: the versatile transcription factor BCL11A is a master repressor of fetal hemoglobin.Reciprocal regulation of γ-globin expression by exo-miRNAs: Relevance to γ-globin silencing in β-thalassemia major.Molecular mechanism of yisui shengxue granule, a complex chinese medicine, on thalassemia patients suffering from hemolysis and anemia of erythrocytesMicroRNA-486-5p is an erythroid oncomiR of the myeloid leukemias of Down syndromeMicroRNA-486 regulates normal erythropoiesis and enhances growth and modulates drug response in CML progenitorsThe Anti-fibrotic Effects and Mechanisms of MicroRNA-486-5p in Pulmonary FibrosisOriginal Research: Stable expression of miR-34a mediates fetal hemoglobin induction in K562 cells.Recent trends for novel options in experimental biological therapy of β-thalassemia.Concise review: Exploring miRNAs--toward a better understanding of hematopoiesis.Recent trends in the gene therapy of β-thalassemia.Genome-wide analysis of aberrantly expressed lncRNAs and miRNAs with associated co-expression and ceRNA networks in β-thalassemia and hereditary persistence of fetal hemoglobin.2017 Clinical trials update in new treatments of β-thalassemia.Peptide nucleic acids targeting β-globin mRNAs selectively inhibit hemoglobin production in murine erythroleukemia cellsSpecific Depletion of Leukemic Stem Cells: Can MicroRNAs Make the Difference?Featured Article: Modulation of fetal hemoglobin in hereditary persistence of fetal hemoglobin deletion type-2, compared to Sicilian δβ-thalassemia, by BCL11A and SOX6-targeting microRNAs.Fetal haemoglobin induction in sickle cell disease.Pharmacological and molecular approaches for the treatment of β-hemoglobin disorders.BCL11A mRNA Targeting by miR-210: A Possible Network Regulating γ-Globin Gene Expression.Pathophysiology and treatment of patients with beta-thalassemia - an update.Dual Strategies for Argonaute2-Mediated Biogenesis of Erythroid miRNAs Underlie Conserved Requirements for Slicing in Mammals.Comparison of MicroRNAs Mediated in Reactivation of the γ-Globin in β-Thalassemia Patients, Responders and Non-Responders to Hydroxyurea.
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P2860
MicroRNA-486-3p regulates γ-globin expression in human erythroid cells by directly modulating BCL11A.
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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած
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2013 թվականի ապրիլին հրատարակված գիտական հոդված
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2013年の論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年論文
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2013年论文
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name
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@ast
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@en
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
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type
label
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@ast
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@en
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@nl
prefLabel
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@ast
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@en
MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@nl
P2860
P50
P1433
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MicroRNA-486-3p regulates γ-gl ...... by directly modulating BCL11A.
@en
P2093
Ornella Morsilli
Paolo Cianciulli
P2860
P304
P356
10.1371/JOURNAL.PONE.0060436
P407
P577
2013-04-04T00:00:00Z