Indirect recognition of allopeptides promotes the development of cardiac allograft vasculopathy - Test2 - elhamkhani - TriplyDB

Indirect recognition of allopeptides promotes the development of cardiac allograft vasculopathy

Indirect recognition of allopeptides promotes the development of cardiac allograft vasculopathy

http://www.wikidata.org/entity/Q34721121

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Cell therapy to induce allograft tolerance: time to switch to plan B?Allorecognition by T Lymphocytes and Allograft Rejection Alloantibody production is regulated by CD4+ T cells' alloreactive pathway, rather than precursor frequency or Th1/Th2 differentiation.In situ-targeting of dendritic cells with donor-derived apoptotic cells restrains indirect allorecognition and ameliorates allograft vasculopathy.Measurement of T Cell Alloreactivity Using Imaging Flow Cytometry The effects of tolerance on allograft damage caused by the innate immune system Role of persistence of antigen and indirect recognition in the maintenance of tolerance to renal allografts.Mechanisms of chronic rejection in cardiothoracic transplantation.Immunosuppression for lung transplantation.Lymphoid neogenesis in chronic rejection: evidence for a local humoral alloimmune response.Genetic or pharmaceutical blockade of phosphoinositide 3-kinase p110δ prevents chronic rejection of heart allografts Induction of transplantation tolerance to fully mismatched cardiac allografts by T cell mediated delivery of alloantigen.The role of CD8+ T cells during allograft rejection.Pathogenesis of chronic allograft rejection.Antigen location contributes to the pathological features of a transplanted heart graft.T-cell allorecognition and transplant rejection: a summary and update.The indirect alloresponse impairs the induction but not maintenance of tolerance to MHC class I-disparate allografts.Administration of the stress protein gp96 prolongs rat cardiac allograft survival, modifies rejection-associated inflammatory events, and induces a state of peripheral T-cell hyporesponsiveness T-cell depletion eliminates the development of cardiac allograft vasculopathy in mice rendered tolerant by the induction of mixed chimerism.Inhibition of arterial allograft intimal hyperplasia using recipient dendritic cells pretreated with B7 antisense peptide.The role of indirect recognition of MHC class I and II allopeptides in a fully mismatched miniature swine model of lung transplantation.From current immunosuppressive strategies to clinical tolerance of allografts.Quantification of CD4(+) T Cell Alloreactivity and Its Control by Regulatory T Cells Using Time-Lapse Microscopy and Immune Synapse Detection.Direct and indirect antigen presentation lead to deletion of donor-specific T cells after in utero hematopoietic cell transplantation in mice Linked suppression across an MHC-mismatched barrier in a miniature swine kidney transplantation model.Repetitive gastric aspiration leads to augmented indirect allorecognition after lung transplantation in miniature swine.Donor exosomes rather than passenger leukocytes initiate alloreactive T cell responses after transplantation Natural killer cells in rejection and tolerance of solid organ allografts Indirect recognition of MHC class I allopeptides accelerates lung allograft rejection in miniature swine.The exciting "bench to bedside" journey of cell therapies for acute kidney injury and renal transplantation.Contributions of direct and indirect alloresponses to chronic rejection of kidney allografts in nonhuman primates.Tolerance of Lung Allografts Achieved in Nonhuman Primates via Mixed Hematopoietic Chimerism Primary vascularization of allografts governs their immunogenicity and susceptibility to tolerogenesis.Dual effects of the alloresponse by Th1 and Th2 cells on acute and chronic rejection of allotransplants.Innate immunity and resistance to tolerogenesis in allotransplantation Both rejection and tolerance of allografts can occur in the absence of secondary lymphoid tissues.Tolerogenicity of donor major histocompatibility complex-matched skin grafts in previously tolerant Massachusetts general hospital miniature swine.A rendezvous before rejection: where do T cells meet transplant antigens?Direct versus indirect allorecognition pathways: on the right track.Prolongation of cardiac allograft survival by systemic administration of immature recipient dendritic cells deficient in NF-kappaB activity.

P2860

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P2860

Indirect recognition of allopeptides promotes the development of cardiac allograft vasculopathy

http://www.wikidata.org/entity/Q34721121

description

2001 nî lūn-bûn

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2001 թվականի մարտին հրատարակված գիտական հոդված

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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Proceedings of the National Academy of Sciences of the United States of America

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Indirect recognition of allope ...... cardiac allograft vasculopathy

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H S Rose

http://www.w3.org/2001/XMLSchema#string

J C Madsen

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K L Womer

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K Yamada

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M E Maloney

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M H Sayegh

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R S Lee

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S L Houser

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P2860

Atherosclerosis — An Inflammatory Disease

Arteriosclerosis in normal and von Willebrand pigs: long-term prospective study and aortic transplantation study.

Coronary arteriosclerosis after T-cell-mediated injury in transplanted mouse hearts: role of interferon-gamma.

The role of "indirect" recognition in initiating rejection of skin grafts from major histocompatibility complex class II-deficient mice

Contribution of direct and indirect recognition pathways to T cell alloreactivity.

Experimental models of coronary artery restenosis.

Indirect recognition of donor HLA-DR peptides in organ allograft rejection.

Persistent allopeptide reactivity and epitope spreading in chronic rejection of organ allografts.

Late blockade of T cell costimulation interrupts progression of experimental chronic allograft rejection.

The molecular basis of alloreactivity.

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3276-3281

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10.1073/PNAS.051584498

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6

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98

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2001-03-01T00:00:00Z

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12084448

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11248069

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30644

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