Knockout models are useful tools to dissect the pathophysiology and genetics of insulin resistance.
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Does inflammation determine whether obesity is metabolically healthy or unhealthy? The aging perspectiveBeta-cell-specific ablation of the hepatocyte growth factor receptor results in reduced islet size, impaired insulin secretion, and glucose intoleranceCurrent views on type 2 diabetes.Muscle-specific Pten deletion protects against insulin resistance and diabetesIncreased insulin sensitivity and reduced adiposity in phosphatidylinositol 5-phosphate 4-kinase beta-/- miceControl of body weight: a physiologic and transgenic perspective.When the usual insulin is just not enoughThe role of estrogens in control of energy balance and glucose homeostasis.Oxidized LDL impair adipocyte response to insulin by activating serine/threonine kinases.Metreleptin improves blood glucose in patients with insulin receptor mutations.Separation of insulin signaling into distinct GLUT4 translocation and activation steps.An essential role of the JIP1 scaffold protein for JNK activation in adipose tissue.Insulin receptor substrate 2 plays a crucial role in beta cells and the hypothalamus.Akt isoforms and glucose homeostasis - the leptin connection.Menopausal Hormone Therapy and Type 2 Diabetes Prevention: Evidence, Mechanisms, and Clinical Implications.The Genetic Basis of Type 2 Diabetes.A stress signaling pathway in adipose tissue regulates hepatic insulin resistance.Small interfering peptide (siP) for in vivo examination of the developing lung interactonome.Leptin deficiency and beta-cell dysfunction underlie type 2 diabetes in compound Akt knockout mice.Mechanism for the synergistic effect of rapamycin and resveratrol on hyperinsulinemia may involve the activation of protein kinase B.Characterization of the abnormal pancreatic development, reduced growth and infertility in Cdk4 mutant mice.Both insulin signaling defects in the liver and obesity contribute to insulin resistance and cause diabetes in Irs2(-/-) mice.Low sympathetic tone and obese phenotype in oxytocin-deficient mice.In vivo inhibition of focal adhesion kinase causes insulin resistance.Negative Impact of Testosterone Deficiency and 5α-Reductase Inhibitors Therapy on Metabolic and Sexual Function in Men.Menopause, Estrogens, and Glucose Homeostasis in Women.
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P2860
Knockout models are useful tools to dissect the pathophysiology and genetics of insulin resistance.
description
2002 nî lūn-bûn
@nan
2002 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2002年の論文
@ja
2002年論文
@yue
2002年論文
@zh-hant
2002年論文
@zh-hk
2002年論文
@zh-mo
2002年論文
@zh-tw
2002年论文
@wuu
name
Knockout models are useful too ...... enetics of insulin resistance.
@ast
Knockout models are useful too ...... enetics of insulin resistance.
@en
Knockout models are useful too ...... enetics of insulin resistance.
@nl
type
label
Knockout models are useful too ...... enetics of insulin resistance.
@ast
Knockout models are useful too ...... enetics of insulin resistance.
@en
Knockout models are useful too ...... enetics of insulin resistance.
@nl
prefLabel
Knockout models are useful too ...... enetics of insulin resistance.
@ast
Knockout models are useful too ...... enetics of insulin resistance.
@en
Knockout models are useful too ...... enetics of insulin resistance.
@nl
P2860
P1476
Knockout models are useful too ...... enetics of insulin resistance.
@en
P2093
Franck Mauvais-Jarvis
Rohit N Kulkarni
P2860
P356
10.1046/J.1365-2265.2002.01563.X
P577
2002-07-01T00:00:00Z