Human papillomavirus E7 enhances hypoxia-inducible factor 1-mediated transcription by inhibiting binding of histone deacetylases - Test2 - elhamkhani - TriplyDB

Human papillomavirus E7 enhances hypoxia-inducible factor 1-mediated transcription by inhibiting binding of histone deacetylases

Human papillomavirus E7 enhances hypoxia-inducible factor 1-mediated transcription by inhibiting binding of histone deacetylases

http://www.wikidata.org/entity/Q34799489

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Pharmacologic management of advanced cervical cancer: antiangiogenesis therapy and immunotherapeutic considerations Improved survival with bevacizumab in advanced cervical cancer Targeting hexokinase 2 inhibition promotes radiosensitization in HPV16 E7-induced cervical cancer and suppresses tumor growth.Cdc6 contributes to abrogating the G1 checkpoint under hypoxic conditions in HPV E7 expressing cells.Productive replication of human papillomavirus 31 requires DNA repair factor Nbs1.Suppression of STAT-1 expression by human papillomaviruses is necessary for differentiation-dependent genome amplification and plasmid maintenance.The E7 open reading frame acts in cis and in trans to mediate differentiation-dependent activities in the human papillomavirus type 16 life cycle.The interferon-related developmental regulator 1 is used by human papillomavirus to suppress NFκB activation.Systematic analysis of the amino acid residues of human papillomavirus type 16 E7 conserved region 3 involved in dimerization and transformation.Hypoxia inducible factor-1 alpha expression is increased in infected positive HPV16 DNA oral squamous cell carcinoma and positively associated with HPV16 E7 oncoprotein HEXIM1 down-regulates hypoxia-inducible factor-1α protein stability.Opportunities and challenges facing biomarker development for personalized head and neck cancer treatment.Lung Cancer and Human Papilloma Viruses (HPVs): Examining the Molecular Evidence Transcription factor networks as targets for therapeutic intervention of cancer: the breast cancer paradigm.Conserved region 3 of human papillomavirus 16 E7 contributes to deregulation of the retinoblastoma tumor suppressor.Clinical Trials of Antiangiogenesis Therapy in Recurrent/Persistent and Metastatic Cervical Cancer.Breaking down the evidence for bevacizumab in advanced cervical cancer: past, present and future.Cellular transformation by human papillomaviruses: lessons learned by comparing high- and low-risk viruses Overexpressed HDAC4 is associated with poor survival and promotes tumor progression in esophageal carcinoma Towards incorporating epigenetic mechanisms into carcinogen identification and evaluation.The papillomavirus E7 proteins.Association Between HIF-1 Alpha Gene Polymorphisms and Response in Patients Undergoing Neoadjuvant Chemotherapy for Locally Advanced Cervical Cancer.Epigenetics of human papillomaviruses.Validation of an HPV16-mediated carcinogenesis mouse model.Class IIa HDACs: from important roles in differentiation to possible implications in tumourigenesis.Increased expression of RRM2 by human papillomavirus E7 oncoprotein promotes angiogenesis in cervical cancer microRNAs are biomarkers of oncogenic human papillomavirus infections.Epigenetics and cervical cancer: from pathogenesis to therapy.Role of autophagy in head and neck cancer and therapeutic resistance.Exploring the therapeutic rationale for angiogenesis blockade in cervical cancer Metabolic reprogramming: a hallmark of viral oncogenesis.Molecular mechanisms of human papillomavirus-related carcinogenesis in head and neck cancer.High-risk human papillomaviruses repress constitutive kappa interferon transcription via E6 to prevent pathogen recognition receptor and antiviral-gene expression.Overexpression of HPV16 E6/E7 mediated HIF-1α upregulation of GLUT1 expression in lung cancer cells.Human papillomavirus type 16 E7 oncoprotein upregulates the retinoic acid receptor-beta expression in cervical cancer cell lines and K14E7 transgenic mice.The human papillomavirus E7 oncoprotein as a regulator of transcription.A compendium of proteins that interact with HIF-1α.Regulation of the human papillomavirus type 16 late promoter by transcriptional elongation.The Interaction Between Human Papillomaviruses and the Stromal Microenvironment.Modulation of DNA methylation by human papillomavirus E6 and E7 oncoproteins in cervical cancer.

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Human papillomavirus E7 enhances hypoxia-inducible factor 1-mediated transcription by inhibiting binding of histone deacetylases

http://www.wikidata.org/entity/Q34799489

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2010 nî lūn-bûn

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2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

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2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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Human papillomavirus E7 enhanc ...... inding of histone deacetylases

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Human papillomavirus E7 enhanc ...... inding of histone deacetylases

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Human papillomavirus E7 enhanc ...... inding of histone deacetylases

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Human papillomavirus E7 enhanc ...... inding of histone deacetylases

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Human papillomavirus E7 enhanc ...... inding of histone deacetylases

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Cancer Research

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Human papillomavirus E7 enhanc ...... inding of histone deacetylases

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Laimonis A Laimins

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P2860

Metastasis-associated protein 1 enhances stability of hypoxia-inducible factor-1alpha protein by recruiting histone deacetylase 1.

Histone deacetylase inhibitors repress the transactivation potential of hypoxia-inducible factors independently of direct acetylation of HIF-alpha

Transcriptional activation of hypoxia-inducible factor-1alpha by HDAC4 and HDAC5 involves differential recruitment of p300 and FIH-1

Regulation of tumor angiogenesis by p53-induced degradation of hypoxia-inducible factor 1alpha

The human Mi-2/NuRD complex and gene regulation

Histone deacetylase 7 associates with hypoxia-inducible factor 1alpha and increases transcriptional activity

HIF-1 regulation: not so easy come, easy go

The histone deacetylase Sirt6 regulates glucose homeostasis via Hif1alpha

The tumor suppressor p53 down-regulates glucose transporters GLUT1 and GLUT4 gene expression

Patterns and emerging mechanisms of the angiogenic switch during tumorigenesis

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