Innate immune genes synergize to predict increased risk of chronic disease in hepatitis C virus infection. - Test2 - elhamkhani - TriplyDB

Innate immune genes synergize to predict increased risk of chronic disease in hepatitis C virus infection.

Innate immune genes synergize to predict increased risk of chronic disease in hepatitis C virus infection.

http://www.wikidata.org/entity/Q34804539

about

Natural killer cells in hepatitis C: Current progress NK cell function and receptor diversity in the context of HCV infection Natural killer cell responses during viral infections: flexibility and conditioning of innate immunity by experience Influence of host resistance on viral adaptation: hepatitis C virus as a case study IL28B, HLA-C, and KIR variants additively predict response to therapy in chronic hepatitis C virus infection in a European Cohort: a cross-sectional study Peg-Interferon Lambda Treatment Induces Robust Innate and Adaptive Immunity in Chronic Hepatitis B Patients.Protective immunity against hepatitis C: many shades of gray.Meta-analysis: implications of interleukin-28B polymorphisms in spontaneous and treatment-related clearance for patients with hepatitis C.Variation in both IL28B and KIR2DS3 genes influence pegylated interferon and ribavirin hepatitis C treatment outcome in HIV-1 co-infection.KIR, HLA, and IL28B variant predict response to antiviral therapy in genotype 1 chronic hepatitis C patients in Japan.Genetic diversity of the KIR/HLA system and susceptibility to hepatitis C virus-related diseases.PEG-IFN alpha but not ribavirin alters NK cell phenotype and function in patients with chronic hepatitis C Diversity of killer cell immunoglobulin-like receptor (KIR) genotypes and KIR2DL2/3 variants in HCV treatment outcome Additive effects of HLA alleles and innate immune genes determine viral outcome in HCV infection Natural killer inhibitory receptor expression associated with treatment failure and interleukin-28B genotype in patients with chronic hepatitis C NK cells require IL-28R for optimal in vivo activity Gene expression profiling of HCV genotype 3a initial liver fibrosis and cirrhosis patients using microarray Early changes in natural killer cell function indicate virologic response to interferon therapy for hepatitis C.Association of IL28B SNP With Progression of Egyptian HCV Genotype 4 Patients to End Stage Liver Disease.The impact of interferon lambda 3 gene polymorphism on natural course and treatment of hepatitis C Influence of HLA class I, HLA class II and KIRs on vertical transmission and chronicity of hepatitis C virus in children.IL28B SNP screening and distribution in the French Canadian population using a rapid PCR-based test The interaction of genetic determinants in the outcome of HCV infection: evidence for discrete immunological pathways.Killer Cell Immunoglobulin-like Receptor Genotype and Haplotype Investigation of Natural Killer Cells from an Australian Population of Chronic Fatigue Syndrome/Myalgic Encephalomyelitis Patients.A method for killer-cell immunoglobulin-like receptor (KIR) 3DL1/3DS1 genotyping using DNA recovered from frozen plasma.IFN-λ3 polymorphism indirectly influences NK cell phenotype and function during acute HCV infection IL-28B Genetic Variants Determine the Extent of Monocyte-Induced Activation of NK Cells in Hepatitis C.The natural killer cell response to HCV infection.Between Scylla and Charybdis: the role of the human immune system in the pathogenesis of hepatitis C.Type III Interferons in Hepatitis C Virus Infection.Variable NK cell receptors and their MHC class I ligands in immunity, reproduction and human evolution.Activating killer cell Ig-like receptors in health and disease.IL28B polymorphism and genetic biomarkers of viral clearance in hepatitis C virus infection.Maternal KIR and fetal HLA-C: a fine balance.Hepatitis C virus--T-cell responses and viral escape mutations.IL28B polymorphisms as a pretreatment predictor of response to HCV treatment.Innate and adaptive genetic pathways in HCV infection.NK cells: tuned by peptide?An effective interferon-gamma-mediated inhibition of hepatitis C virus replication by natural killer cells is associated with spontaneous clearance of acute hepatitis C in human immunodeficiency virus-positive patients.Host and viral determinants of the outcome of exposure to HCV infection genotype 4: a large longitudinal study.

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Innate immune genes synergize to predict increased risk of chronic disease in hepatitis C virus infection.

http://www.wikidata.org/entity/Q34804539

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Proceedings of the National Academy of Sciences of the United States of America

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Innate immune genes synergize ...... n hepatitis C virus infection.

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Brian P McSharry

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Clair M Gardiner

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Cliona O'Farrelly

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Irish HCV Research Consortium

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Kieran J Guinan

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Maria H Morrison

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Megan M Dring

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Richard Hagan

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P2860

Genetic variation in IL28B and spontaneous clearance of hepatitis C virus

Polymorphisms of killer cell immunoglobulin-like receptor gene: possible association with susceptibility to or clearance of hepatitis B virus infection in Chinese Han population

Lambda interferon inhibits hepatitis B and C virus replication.

Viral and immunological determinants of hepatitis C virus clearance, persistence, and disease.

Hepatitis C virus versus innate and adaptive immune responses: a tale of coevolution and coexistence

A New Statistical Method for Haplotype Reconstruction from Population Data

Analysis of successful immune responses in persons infected with hepatitis C virus

Global epidemiology of hepatitis C virus infection

Determinants of viral clearance and persistence during acute hepatitis C virus infection

Genetic variation in IL28B predicts hepatitis C treatment-induced viral clearance

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