Osteoclasts are essential for TNF-alpha-mediated joint destruction. - Test2 - elhamkhani - TriplyDB

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

http://www.wikidata.org/entity/Q34826003

about

Identification of the homeobox protein Prx1 (MHox, Prrx-1) as a regulator of osterix expression and mediator of tumor necrosis factor α action in osteoblast differentiation The role of osteoprotegerin in arthritis Collagen type II (CII)-specific antibodies induce arthritis in the absence of T or B cells but the arthritis progression is enhanced by CII-reactive T cells JNK1 is not essential for TNF-mediated joint disease.Animal Models of Bone Loss in Inflammatory Arthritis: from Cytokines in the Bench to Novel Treatments for Bone Loss in the Bedside-a Comprehensive Review Impact of inflammation on the osteoblast in rheumatic diseases Effects of anti-tumor necrosis factor α agents on bone A dynamic real time in vivo and static ex vivo analysis of granulomonocytic cell migration in the collagen-induced arthritis model Biochemical markers of ongoing joint damage in rheumatoid arthritis--current and future applications, limitations and opportunities.Loss of SH3 domain-binding protein 2 function suppresses bone destruction in tumor necrosis factor-driven and collagen-induced arthritis in mice NFATc1 in mice represses osteoprotegerin during osteoclastogenesis and dissociates systemic osteopenia from inflammation in cherubism.An extensive phenotypic characterization of the hTNFalpha transgenic mice.Supplementation of diet with krill oil protects against experimental rheumatoid arthritis.Cytokines regulate complement receptor immunoglobulin expression and phagocytosis of Candida albicans in human macrophages: A control point in anti-microbial immunity.NF-(kappa)B-inducing kinase controls lymphocyte and osteoclast activities in inflammatory arthritis Bone erosion in rheumatoid arthritis: mechanisms, diagnosis and treatment.Postprandial response of bone turnover markers in patients with Crohn's disease.Osteoporosis in inflammatory bowel disease Bone damage in rheumatoid arthritis: mechanistic insights and approaches to prevention Joint damage in rheumatoid arthritis progresses in remission according to the Disease Activity Score in 28 joints and is driven by residual swollen joints.SH3BP2 gain-of-function mutation exacerbates inflammation and bone loss in a murine collagen-induced arthritis model M-CSF mediates TNF-induced inflammatory osteolysis The study of mechanisms of protective effect of Rg1 against arthritis by inhibiting osteoclast differentiation and maturation in CIA mice Wnt4 signaling prevents skeletal aging and inflammation by inhibiting nuclear factor-κB.Genetic deficiency of Syk protects mice from autoantibody-induced arthritis.Ligation of TLR5 promotes myeloid cell infiltration and differentiation into mature osteoclasts in rheumatoid arthritis and experimental arthritis.Staphylococcus aureus protein A plays a critical role in mediating bone destruction and bone loss in osteomyelitis.A combination of methotrexate and zoledronic acid prevents bone erosions and systemic bone mass loss in collagen induced arthritis.Connective tissue growth factor promotes articular damage by increased osteoclastogenesis in patients with rheumatoid arthritis.Abatacept: the evidence for its place in the treatment of rheumatoid arthritis.Inhibition of Inflammation and Bone Erosion by RNA Interference-Mediated Silencing of Heterogeneous Nuclear RNP A2/B1 in Two Experimental Models of Rheumatoid Arthritis.THR0921, a novel peroxisome proliferator-activated receptor gamma agonist, reduces the severity of collagen-induced arthritis The phosphoinositide 3-kinase isoform PI3Kβ regulates osteoclast-mediated bone resorption in humans and mice.Infliximab: 12 years of experience Repair of local bone erosions and reversal of systemic bone loss upon therapy with anti-tumor necrosis factor in combination with osteoprotegerin or parathyroid hormone in tumor necrosis factor-mediated arthritis.Green tea polyphenol epigallocatechin-3-gallate: inflammation and arthritis. [corrected].Long term high intensity exercise and damage of small joints in rheumatoid arthritis Lower limb arterial incompressibility and obstruction in rheumatoid arthritis.Benefit of pregnancy in inflammatory arthritis.Osteoimmunology at the nexus of arthritis, osteoporosis, cancer, and infection.

P2860

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P2860

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

http://www.wikidata.org/entity/Q34826003

description

2002 nî lūn-bûn

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2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2002 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2002年の論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年論文

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2002年论文

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name

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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type

label

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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Osteoclasts are essential for TNF-alpha-mediated joint destruction.

@nl

prefLabel

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

@ast

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

@en

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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Journal of Clinical Investigation

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Osteoclasts are essential for TNF-alpha-mediated joint destruction.

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P2093

George Kollias

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Günter Steiner

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Jean-Pierre David

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Kurt Redlich

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Makiyeh Tohidast-Akrad

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Romeo Ricci

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Silvia Hayer

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P2860

Osteoprotegerin: a novel secreted protein involved in the regulation of bone density

A homologue of the TNF receptor and its ligand enhance T-cell growth and dendritic-cell function

Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis

Osteoclast differentiation factor is a ligand for osteoprotegerin/osteoclastogenesis-inhibitory factor and is identical to TRANCE/RANKL

Protection against cartilage and bone destruction by systemic interleukin-4 treatment in established murine type II collagen-induced arthritis

Modulation of osteoclast differentiation and function by the new members of the tumor necrosis factor receptor and ligand families

The osteoclast differentiation factor osteoprotegerin-ligand is essential for mammary gland development

Bone resorption by osteoclasts

Involvement of receptor activator of NFkappaB ligand and tumor necrosis factor-alpha in bone destruction in rheumatoid arthritis

A new mechanism of bone destruction in rheumatoid arthritis: synovial fibroblasts induce osteoclastogenesis

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10.1172/JCI0215582

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