Defective repair of oxidative base lesions by the DNA glycosylase Nth1 associates with multiple telomere defects.
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APE1 incision activity at abasic sites in tandem repeat sequencesThe origin of oxidized guanine resolves the puzzle of oxidation-induced telomere-length alterations.Senescence induced by RECQL4 dysfunction contributes to Rothmund-Thomson syndrome features in mice.Rare missense variants in POT1 predispose to familial cutaneous malignant melanomaInteractive Roles of DNA Helicases and Translocases with the Single-Stranded DNA Binding Protein RPA in Nucleic Acid Metabolism.No cancer predisposition or increased spontaneous mutation frequencies in NEIL DNA glycosylases-deficient miceBase excision repair: a critical player in many gamesMediterranean diet and telomere length in Nurses' Health Study: population based cohort study.The replicometer is broken: telomeres activate cellular senescence in response to genotoxic stresses.Defective repair of uracil causes telomere defects in mouse hematopoietic cells.The NEIL glycosylases remove oxidized guanine lesions from telomeric and promoter quadruplex DNA structuresFlap Endonuclease 1 Limits Telomere Fragility on the Leading StrandNeil2-null Mice Accumulate Oxidized DNA Bases in the Transcriptionally Active Sequences of the Genome and Are Susceptible to Innate InflammationDNA excision repair at telomeres.Polymerase η suppresses telomere defects induced by DNA damaging agentsMechanistic and biological considerations of oxidatively damaged DNA for helicase-dependent pathways of nucleic acid metabolism.Identifying novel protein phenotype annotations by hybridizing protein-protein interactions and protein sequence similarities.NEIL3 Repairs Telomere Damage during S Phase to Secure Chromosome Segregation at Mitosis.Molecular mechanisms by which oxidative DNA damage promotes telomerase activity.Age-Dependent Protective Effect of Selenium against UVA Irradiation in Primary Human Keratinocytes and the Associated DNA Repair Signature.The Good, the Bad, and the Ugly of ROS: New Insights on Aging and Aging-Related Diseases from Eukaryotic and Prokaryotic Model Organisms.
P2860
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P2860
Defective repair of oxidative base lesions by the DNA glycosylase Nth1 associates with multiple telomere defects.
description
2013 nî lūn-bûn
@nan
2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Defective repair of oxidative ...... ith multiple telomere defects.
@ast
Defective repair of oxidative ...... ith multiple telomere defects.
@en
Defective repair of oxidative ...... ith multiple telomere defects.
@nl
type
label
Defective repair of oxidative ...... ith multiple telomere defects.
@ast
Defective repair of oxidative ...... ith multiple telomere defects.
@en
Defective repair of oxidative ...... ith multiple telomere defects.
@nl
prefLabel
Defective repair of oxidative ...... ith multiple telomere defects.
@ast
Defective repair of oxidative ...... ith multiple telomere defects.
@en
Defective repair of oxidative ...... ith multiple telomere defects.
@nl
P2093
P2860
P1433
P1476
Defective repair of oxidative ...... ith multiple telomere defects.
@en
P2093
Haritha Vallabhaneni
Nathan O'Callaghan
P2860
P304
P356
10.1371/JOURNAL.PGEN.1003639
P577
2013-07-18T00:00:00Z